Wyniki wyszukiwania - AGRO

1

Recent concepts of cell death

100%

Mandys V., Jirsova K.

Folia Histochemica et Cytobiologica. Supplement

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1997

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tom 35

|

nr 2

2

Activation induced cell death [AICD] in Jurkat line cells

80%

Filip A., Koczkodaj D., Kwiatkowska-Drabik B., Rozynkowa D.

Folia Histochemica et Cytobiologica. Supplement

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1997

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tom 35

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nr 2

3

Induction of apoptosis in cells of GH3 line by bromocryptine

80%

Wasko R., Wolun M., Warchol J. B.

Folia Histochemica et Cytobiologica

|

1999

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tom 37

|

nr 2

4

Disorders of programmed cell death in differentiating tracheids and anatomical abnormalities of Pinus sylvestris wood in polluted environment

80%

Rusin A., Tulik M.

Polish Journal of Natural Sciences. Supplement

|

2003

|

tom 01

5

Regulatory role of cathepsin D in apoptosis

80%

Minarowska A., Miarowski L., Karwowska A., Gacko M.

Folia Histochemica et Cytobiologica

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2007

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tom 45

|

nr 3

6

Apoptosis and proliferation in peripheral blood lymphocytes on different maturation stages

80%

Halasa M.

Folia Histochemica et Cytobiologica. Supplement

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1997

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tom 35

|

nr 2

7

Changes in thymocytes undergoing programmed death.

80%

Gozdzicka-Jozefiak A., Warchol J. B., Zagorski L., Izycki D., Stasiak A., Jaroszewski J., Augustyniak J.

Acta Biochimica Polonica

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1992

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tom 39

|

nr 1

8

Cytogenetic assay in apoptosis investigations

80%

Akopyan H., Sirenko A., Sedneva I., Jakimovich M., Stojka R., Hnatejko O.

Folia Histochemica et Cytobiologica. Supplement

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2001

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tom 39

|

nr 1

9

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Molecular aspects of effect of Shiga toxin in humans - a Review

71%

Pal P.

International Letters of Natural Sciences

|

2015

|

tom 07

Shiga toxins belonging to a family of structurally and functionally related protein toxins serve as the key virulence factors for pathogenecity of the virulent enteric bacterial strains namely Shigella dysenteriae serotype 1 including Shiga toxin-producing Escherichia coli (STEC). S. dysenteriae type 1 isolates remain major public health concerns due to their widespread outbreaks and the severity of extra-intestinal diseases, including acute renal failure and also affects the central nervous system. Despite practicing improved hygienic conditions and regulating food and drinking water safety, the enteric pathogens are imposing a major threat in the well being of the human society. Shiga toxin on entry into host cells’ endoplasmic reticulum (ER) activates the stress response in ER and inhibits protein synthesis by catalytic inactivation of eukaryotic ribosomes. In many cell types shiga toxins trigger apoptosis. Recent studies have shown that shiga toxins induce autophagy which activates different signaling pathways in toxin-sensitive and toxin-resistant human cells. In this review the molecular basis of Shiga toxins’ effect on host cell leading to manifestation of the infection in affected individuals are discussed with an emphasis on recent findings.

10

Restoring TRAIL mediated signaling in ovarian cancer cells

71%

Farooqi A. A., Yaylim I., Ozkan N. E., Zaman F., Halim T. A., Chang H.-W.

Archivum Immunologiae et Therapiae Experimentalis

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2014

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tom 62

|

nr 6

11

Expression of IL-3 in WEHI3B cells during apoptosis

71%

Kawiak J., Glowacka E., Hoser G.

Folia Histochemica et Cytobiologica. Supplement

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1997

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tom 35

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nr 2

12

The role of endogenous nitric oxide in inhibition of ischemia-reperfusion-induced cardiomyocyte apoptosis

71%

Czarnowska E., Kurzelewski M., Beresewicz A., Karczmarewicz E.

Folia Histochemica et Cytobiologica

|

2001

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tom 39

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nr 2

13

Modulation of ERK1-2 activity is crucial for sphingosine-induced death of glioma C6 cells

71%

Krzeminski P.

Acta Biochimica Polonica

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2005

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tom 52

|

nr 4

In this study the contribution of the ERK1/2 pathway to sphingosine-induced death and morphological changes of the actin cytoskeleton in glioma C6 cells was investigated. Surprisingly, the level of ERK1/2 phosphorylation does not change after incubation of cells with sphingosine. Despite this, sphingosine induces rounding and detachment of cells without formation of apoptotic bodies. To shed light on this process, a specific inhibitor of ERK1/2 phosphorylation, U0126, was used. Cells incubated simultaneously with sphingosine and U0126 not only detached, but also exhibited formation of apoptotic-like blebs. These data suggest that during sphingosine-induced glioma C6 cell death apoptotic blebbing is dependent on ERK1/2 signalling and occurs only when ERK1/2 activity is decreased or abolished.

14

The mechanism of preconditioning involves mitoKATP mediated prevention of free radical generation and apoptosis in the post-ischemic heart

71%

Czarnowska E., Duda M., Beresewicz A.

Cellular and Molecular Biology Letters

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2002

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tom 07

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nr Suppl.

15

The impact of neuraminidase on apoptosis in cultures of blood lymphocytes isolated from rats bearing Morris hepatoma

71%

Gasiorowski K., Steciwko A., Grata-Borkowska U., Drobnik J.

Cellular and Molecular Biology Letters

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2004

|

tom 09

|

nr 2

Lymphocytes were obtained by heart-punction from rats bearing Morris hepatoma. In the short term, 18-hour cultures of these lymphocytes exhibited a significantly higher amount of apoptotic cells than lymphocyte cultures from the healthy, control animals. Neuraminidase, injected into the caudal vein of the rats with Morris hepatoma, caused a marked lowering in the amount of apoptotic blood-lymphocytes and an elevation of the amount of viable cells. The possible mechanism of neuraminidase preventing the apoptosis of blood-circulating lymphocytes in tumour hosts is discussed herein.

16

The molecular basis of resistance to apoptosis in calcitriol differentiated cells

71%

Chrobak A.

Cellular and Molecular Biology Letters

|

2002

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tom 07

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nr Suppl.

17

Non-lymphoid cultured cells possess a system controlling cellular compatibility

71%

Sashchenko L. P., Dukhanin E. A., Ioudinkova E. S., Iarovaia O. V., Lukianova T. I., Razin S. V.

Cellular and Molecular Biology Letters

|

2000

|

tom 05

|

nr 2

18

Influence of culture medium pH on cytotoxicity of CD95L in vitro

71%

Grzela T., Lazarczyk M., Oldak M., Korczak-Kowalska G.

Folia Histochemica et Cytobiologica

|

2001

|

tom 39

|

nr 1

19

Monocytes which ingested bacteria first express cytokines and then enter apoptosis: A direct demonstration using single-cell RT-PCR

71%

Guzik K., Pryjma J.

Folia Histochemica et Cytobiologica. Supplement

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1997

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tom 35

|

nr 2

20

Site-specific excision or protection of an alpha a globin gene genomic site in apoptotic transformed chicken erythroblasts

71%

Sjakste N.

Cellular and Molecular Biology Letters

|

2004

|

tom 09

|

nr 3

There is still no clarity on whether the endonuclease incisions in apoptotic cells are induced randomly in the genome or induced in some preferable sites. In order to evaluate the intensity of DNA fragmentation in the chicken alpha-globin domain, AEV-virus transformed chicken erythroblasts (HD3) were incubated in a serum free medium, and their DNA was Southern blotted and hybridised with probes representing different fragments of the domain. Probes corresponding to the upstream areas of the domain mostly hybridised with high molecular weight DNA. Unlike these, the probe corresponding to the 2 Kb BamHI-BamHI fragment, containing the alphaA globin gene (B18), revealed a 5 Kb band on the hybridisation autoradiographs. The probe to the neighbouring upstream fragment did not reveal this band, but it was clearly seen on hybridisations with a downstream 1 Kb BamHI-BamHI fragment. The intensity of the band increased with overall apoptotic DNA degradation, hence its appearance should be coupled to apoptosis. Hybridisation of BamHI-digested DNA with B18 probe revealed a shortening of the 2 Kb band in preparations of DNA from apoptotic cells. The presumable positions of the cuts correspond to the formerly described DNase hypersensitive sites in the domain. Slot-blot and Northern hybridisation of RNA extracted from apoptotic HD3 cells revealed that the excision of the area of the B18 gene is coupled to a decrease in the intensity of alphaA globin gene transcription. Transcription of the non-erythroid NIK gene, transcribed in the upstream part of the domain, did not depend on the level of apoptotic DNA fragmentation.

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