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The study was conducted on 36 female Pharaoh quails (3 groups, 12 birds per group). The experiment covered the 7th to the 20th weeks of birds’ lives. The control group (I) received standard feed formulated for adult quails. Groups II and III received standard feed with 4% and 7% of amaranth seeds, respectively. All feeds were isoproteinous and isocaloric. At the age of weeks 20, 12 females were randomly selected from each group and slaughtered. In the isolated breast and leg muscles water-holding capacity, thermal drip, colour, basic chemical composition, fatty acids profile were determined and sensory evaluation performed. Amaranth seed supplementation showed no effect on the basic chemical composition of breast and legs muscles and fatty acids profile. Deterioration in the flavor of cooked quail breast muscle was observed with the higher dosage of the seeds in the diet.Improved muscle tenderness was observed in birds receiving 4% of amaranth seeds in the diet.
Experiments were performed on 12 sheep divided into 2 groups and fed a standard diet. The trial group received the same nutritive fodder supplemented with 0.3 ppm of sodium selenite (Na₂SeO3). The concentrations of selenium, triiodothyronine and thyroxine in serum were determined every 4 weeks during 20 weeks, respectively. Oral supplements of sodium selenite increased the serum concentration of selenium in sheep from 0.34 ± 0.19 to 4.25 ± 0.69 μmol/l. Contrary to those in the control group, serum Se content ranged from 0.34 ± 0.08 to 0.36 ± 0.10 μmol/I. Alterations in the metabolism of thyroid hormones showed that supplements of selenium produced an insignificant decrease of thyroxine with a slight increase in triiodothyronine concentration.
Rats were fed a copper fortified diet (10 times the level in a standard diet) for a 4-week period and were concomitantly exposed to cadmium at a dose corresponding to 10 mg Cd/kg diet. The contents of cadmium in the carcass, and liver, kidneys, muscles and testicles and corresponding AUC values, calculated with respect to concentration-time curve, indicate that the copper supplement increased the absorption of cadmium from the gastrointestinal tract and the bodily and organ cadmium burden. On the other hand, an increase in body weight gain produced by the copper supplemental diet suggested that the toxic action of cadmium was limited.
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