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Earthquake is a major disaster responsible for vast losses both life and infrastructure. Pakistan is one of the highly earthquake prone areas in Asia. The present study is carried out to investigate the dynamics of disastrous Awaran earthquake. The 2013 Mw 7.7 Awaran earthquake and its Mw 6.8 aftershock caused numerous loss of lives and vast infrastructure damages. The earthquake triggered along Hoshab fault rupturing 230 km of the fault. The rupture propagated at 3 Km/s on average and was concentrated in top 10 km with no major displacement in the underlying decollement. The rupture released a cumulative moment of 5.4 x 1020 Nm. This study reveals that although Hoshab fault originated as thrust fault, the slip was purely strike slip during the earthquake and that the rigid block rotation of southeastern Makran is responsible for the Awaran earthquake. The study concludes that the earthquake significantly increased the coulomb stress on Makran mega thrust and strike slip faults in Chaman fault system, hence increasing the risk of a major seismic event. Therefore, in order to prevent major loss of lives and infrastructure damages; designing of new building codes, reassessing the seismic hazard of the region and marking of hidden faults is of utmost importance.
This paper presents a case study of the chromosomal complement of 16 mice Mus , musculus domesticus Linnaeus, r 1758 from the region near one of the most recent disastrous earthquakes in Italy (named Umbria-Marche 1997), with the aim of ex­amining chromosomal variability among the mice from the seismically active zones. For the present investigation, the sampling sites were chosen in the vicinity of some active faults, supposedly the main earthquake generators in the area. In the three localities, that lie approximately on the fault lines, mice with a reduced chromosomal number (2n = 36 to 39) were trapped. This reduction is due to the presence of three different Robertsonian metacentrics - Rb(9.14), Rb(10.12) and Rb(15.17) - in both the homo­zygous and heterozygous states. Mice trapped in four localities more distant from the fault zone only had the standard karyotype (2n = 40). These results increase the need to analyze in more detail the distribution of karyotypes in relation with active faults.
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