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Respiratory symptoms and peak expiratory flow rates among furniture-decoration students

100%

Arbak P., Bilgin C., Balbay O., Yesildal N., Annakkaya A. N., Ulger F.

Annals of Agricultural and Environmental Medicine

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2004

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tom 11

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nr 1

This study was designed to evaluate the effects of furniture production, mainly including fir tree (aberia mulleriana), on respiratory health of young workers and to compare the results with those obtained from previous studies. Sixty-four furniture-decoration students (57 males and 7 females) and 62 controls (54 male, 8 female) from different departments in the same school were included into the study. All participants were assessed with a questionnaire (concerning history of occupational exposure, work-related respiratory and other symptoms, smoking history, previous asthma history), full physical examination, spirometric evaluation and chest radiograph. Participants then performed serial monitoring of peak expiratory flow rates (PEFR) at work and away from work within a month. Mean age of students was 20.9 ± 3.7 years, 20.5 ± 2.6 years in controls. There was no difference between study and control groups with regard to age, gender, smoking status and previous asthma history. Reported cough (23.4% vs. 8.1%) and shortness of breath (18.8% vs. 6.5%) were significantly higher in furniture-decoration students than in controls (p = 0.016 and p = 0.034, respectively). Furniture-decoration students had higher conjunctivitis (34.4% vs. 9.7%, p = 0.001) and rhinitis (34.4% vs. 19.4%, p = 0.044) history when compared with controls. Both students and controls were normal in terms of respiratory examination. PEF recordings were performed for approximately one month. Diurnal variability greater than 20% was seen in 12/64 (18.7%) of students at work, whereas it was detected in 4/62 (6.4%) of controls (p = 0,034). When comparing for the presence of diurnal variability greater than 20% in weekends, no difference was found between groups (p = 0.457). In conclusion, early detection of work-related respiratory changes by serial monitoring of peak expiratory flows should save the workers from hazardous respiratory effects of the furniture production, especially in young population.

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Occupational asthma diagnosis workers exposed to organic dust

100%

Sigsgaard T., Schlunssen V.

Annals of Agricultural and Environmental Medicine

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2004

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tom 11

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nr 1

The clinical evaluation of newly developed asthma in an adult should always include consideration of his occupational environment, since an abundance of different exposures, which are known causes of asthma, occur in workplaces. Two types of occupational asthma (OA) are distinguished, by whether they appear after a latency period: 1) Immunological OA, characterised by a latency period, caused by high and low-molecular-weight agents, with or without an IgE mechanism 2) Non-immunological, i.e. irritant induced asthma. The first step of the clinical evaluation is to confirm a diagnosis of asthma. Second step is to find out if there is a temporo-spatial distribution of symptoms and lung function that are indicative of OA. Third step is to determine if the disease at hand is an IgE or a non-IgE mediated disease. Last step is a challenge test that can be either unspecific, in order to assess the responsiveness of the lung, or specific challenge test, especially for the non-IgE mediated OA. The depth of clinical evaluation may vary from a situation in which a classical history confirms the clinical symptoms in e.g. a baker with confirmed allergy towards well-known allergens and a characteristic pattern in serial measurements of lung function, to more elaborate investigations in a situation with no or unknown allergen. In the latter situation, a specific challenge test might be necessary in order to find the offending agent. Finally, challenge tests are important in order to distinguish a causal relation from unspecific hyperresponsiveness in persons with pre-existing asthma. In these situations, extended sick leave and challenge tests can be the only way to find the answer.

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Combined asthmatic-alveolitic reaction in a case of farmer's lung

84%

Baur X., Huber H., Weber K.

Annals of Agricultural and Environmental Medicine

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1996

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tom 03

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nr 1

4

Occupational asthma due to wood dust exposure [ash, oak, beech and pine] - a case study

84%

Spiewak R., Bozek A., Maslowski T., Brewczynski P. Z.

Annals of Agricultural and Environmental Medicine

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1994

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tom 01

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nr 1

5

Non-malignant respiratory diseases and occupational exposure to wood dust. Part I. Fresh wood and mixed wood industry

67%

Jacobsen G., Schaumburg I., Sigsgaard T., Schlunssen V.

Annals of Agricultural and Environmental Medicine

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2010

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tom 17

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nr 1

15-28

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Non-malignant respiratory diseases and occupational exposure to wood dust. Part II. Dry wood industry

67%

Jacobsen G., Schaumburg I., Sigsgaard T., Schlunssen V.

Annals of Agricultural and Environmental Medicine

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2010

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tom 17

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nr 1

29-44

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Prevalence and risk factor for airway diseases in farmers - summary of results of the European farmers' project

67%

Radon K., Monso E., Weber C., Danuser B., Iversen M., Opravil U., Donham K., Hartung J., Pedersen S., Garz S., Blainey D., Rabe U., Nowak D.

Annals of Agricultural and Environmental Medicine

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2002

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tom 09

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nr 2

8

Organic dusts and lung disease: the role of inflammation

67%

Rylander R.

Annals of Agricultural and Environmental Medicine

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1994

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tom 01

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nr 1

Ograniczanie wyników

Respiratory symptoms and peak expiratory flow rates among furniture-decoration students

Occupational asthma diagnosis workers exposed to organic dust

Combined asthmatic-alveolitic reaction in a case of farmer's lung

Occupational asthma due to wood dust exposure [ash, oak, beech and pine] - a case study

Non-malignant respiratory diseases and occupational exposure to wood dust. Part I. Fresh wood and mixed wood industry

Non-malignant respiratory diseases and occupational exposure to wood dust. Part II. Dry wood industry

Prevalence and risk factor for airway diseases in farmers - summary of results of the European farmers' project

Organic dusts and lung disease: the role of inflammation