Wyniki wyszukiwania

1

Dissipation function and the extravascular influx of circulating noradrenaline during its thermogenic control

100%

Mejsnar J.

Acta Physiologica Polonica

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1990

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tom 41

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nr 7

2

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Current concepts in clinical and laboratory assessments of autonomic nervous system activity

75%

Dobrek L., Thor P.

Journal of Pre-Clinical and Clinical Research

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2015

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tom 09

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nr 1

Introduction and objective. As the autonomic nervous system (ANS) dysfunction is present in course of many disorders, an objective assessment of the ANS function is very important. In practice, the assessment is difficult, and based rather on indirect analysis of autonomically-controlled cardiovascular reflexes, than on direct recording of activity of central or peripheral autonomic structures. The aim of our paper was to discuss briefly current, clinical and scientific ANS investigations, as well as possible future methods of autonomic activity evaluation. A brief description of the state of knowledge. The review presents a short outline of autonomic function assessments based on clinical autonomic tests (e.g. “Ewing’s battery”) and discusses the heart rate variability (HRV) study, as currently popular and widespread option of analysis of the ANS activity. Other, complementary methods, including the baroreceptor sensitivity testing, microneurography or plasma norepinephrine measurement were also mentioned. The article also provides premises related to the determination of selected neuropeptides in plasma or saliva as an innovative concept of autonomic activity assessment. Summary. The available, clinical, non-invasive methods used for assessment of the ANS function are still relatively sparse and, in fact, a surrogate for direct ANS assessment. New methods of autonomic tension determination are still needed that would allow a more complete and reliable assessment. Reports of potential new laboratory markers of the ANS activity (NPY and VIP assay) bring some hope.

3

Increase in and in the outflowing from region after stimulation in

75%

Lipinska S., Orlowska-Majdak M., Traczyk W. Z.

Journal of Physiology and Pharmacology

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1996

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tom 47

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nr 2

4

Effects of infusion of corticotropin-releasing factor antagonist into the locus coeruleus on freezing behavior and brain catecholamines in rats

75%

Swiergiel A. H.

Acta Neurobiologiae Experimentalis

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2003

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tom 63

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nr 1

5

Aminergic control of vasopressin secretion in the conscious rat

75%

Wells T., Forsling M. L.

Journal of Physiology and Pharmacology

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1992

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tom 43

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nr 1

The influence of aminergic pathways on basal and stimulated vasopressin (AVP) release was studied in conscious rats, the stimulus for hormone release being an intracerebroventricular (ICV) injection of 5 µl 0.85M sodium chloride. The animals were treated with either phenoxybenzamine, propranolol or haloperidol prior to administration of the central hypertonic stimulus. Phenoxybenzamine elevated basal plasma vasopressin concentrations, while propranolol and haloperidol had no effect. The secretion of АVР in response to the hypertonic stimulus was potentiated by phenoxybenzamine and haloperidol, but the effect of propranolol was equivocal. The antagonists had no effect on basal arterial pressure at the time of hypertonic saline administration or the pressor response to ICV sodium chloride.

6

Role of the noradrenergic system in the secretory function of the corpus luteum

75%

Kotwica J.

Journal of Physiology and Pharmacology. Supplement

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1992

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tom 43

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nr 1

7

Changes in the vasoactive effects of nitric oxide, hydrogen sulfide and the structure of the rat thoracic aorta: the role of age and essential hypertension

63%

Berenyiova A., Drobna M., Cebova M., Kristek F., Cacanyiova S.

Journal of Physiology and Pharmacology

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2018

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tom 69

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nr 4

8

Noradrenaline and learning - dependent cortical plasticity

63%

Edeline J. M.

Acta Neurobiologiae Experimentalis

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2005

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tom 65

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nr 5

9

Age-dependent influence of prenatal stress on the catecholamine level in adrenals and blood glucose concentration in immobilized rats

63%

Beszczynska B., Siejka A.

Acta Biologica Cracoviensia. Series Zoologia

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2008

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tom 50

43-48

10

Modulation of sympathoadrenergic contractions by perivascular adipose tissue in mesenteric arteries of rats with different level of body adiposity

63%

Zemancikova A., Torok J., Balis P., Valovic P., Ulicna O., Chomova M.

Journal of Physiology and Pharmacology

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2020

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tom 71

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nr 4

11

Influence of fasting on adrenal medullary reaction to diazepam

63%

Beszczynska B.

Acta Biologica Cracoviensia. Series Zoologia

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2003

|

tom 45

12

Biogenic amines and the activity of the hypothalamo-pituitary-ovarian axis in ewes

63%

Chomicka L. K.

Journal of Physiology and Pharmacology. Supplement

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1992

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tom 43

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nr 1

13

Endothelial no release caused by red wine polyphenols

63%

Stoclet J. C., Kleschyov A., Andriambeloson E., Diebolt M., Andriantsitohaina R.

Journal of Physiology and Pharmacology

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1999

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tom 50

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nr 4

14

The influence of microwave power on the two-component ESR spectra of noradrenaline-melanin

63%

Pilawa B., Stepien K., Wilczok T.

Current Topics in Biophysics

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1994

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tom 18

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nr 2

15

Interaction of the rat C-terminal fragment [Tyr 0]CGRP [28-37] with noradrenaline and acetylcholine in isolated uterine artery of the pig

63%

Dynarowicz I., Jaroszewski J. J., Markiewicz W.

Polish Journal of Veterinary Sciences

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1999

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tom 02

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nr 2

In the present paper the influence of the rat C-terminal fragment [Tyr°]CGRP(28-37) (Ct-CGRP) was compared with the influences of noradrenaline (NA) and acetylcholine (ACh), respectively on isolated pig uterine artery (UA). It was shown that Ct-CGRP (10 -8 mol/1) caused contraction of UA, similar to NA (10-7 mol/1) action. ACh addition before Ct-CGRP treatment inhibited the action of this peptide. ACh administration after Ct-CGRP pretreatment negated the action of this peptide, whereas Ct-CGRP given before and after NA treatment caused additive contractile reactions of UA. Moreover, it was also shown that susceptibility of UA reaction on Ct-CGRP was higher in the postovulatory phase than in other phases of the oestrous cycle. But the effect of Ct-CGRP addition in NA pretreatment vessels as well as the influence of NA added after Ct-CGRP pretreatment were more marked in the luteal phase than in pre- and postovulatory phases of the cycle. Furthermore it was showed that CGRP (10 -9 mol/1) caused very strong dilatation of UA, equal to the reaction observed after ACh (10-8 mol/1) treatment. We concluded that Ct-CGRP in contrast to the whole molecule of calcitonin gene-related peptide (CGRP) caused vasocontractile activity in the isolated pig uterine artery. This effect was synergistic with NA action and was inhibited by ACh.

16

Mechanisms of catecholamine secretion from chromaffin cells

63%

Burgoyne R. D.

Journal of Physiology and Pharmacology

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1995

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tom 46

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nr 3

17

Effect of L-NAME, a specific nitric oxide synthase inhibitor, on corticotropin-releasing hormone-elicited ACTH and corticosterone secretion

63%

Bugajski J., Borycz J., Gadek-Michalska A., Glod R.

Journal of Physiology and Pharmacology

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1998

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tom 49

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nr 4

18

Blockade of nitric oxide formation impairs adrenergic-induced ACTH and corticosterone secretion

63%

Bugajski J., Gadek-Michalska A., Glod R., Borycz J., Bugajski A. J.

Journal of Physiology and Pharmacology

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1999

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tom 50

|

nr 2

19

Bulbospinal catecholamine neurones and sympathetic pattern generation

63%

Coote J. H., Lewis D. I.

Journal of Physiology and Pharmacology

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1995

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tom 46

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nr 3

20

Early effects of short-term endurance training on hormonal responses to graded exercise

63%

Chwalbinska-Moneta J., Kruk B., Nazar K., Krzeminski K., Kaciuba-Uscilko H., Ziemba A.

Journal of Physiology and Pharmacology

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2005

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tom 56

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nr 1

Twelve male, sedentary volunteers (22.0 ± 0.7 yrs) were submitted to three weeks of a bicycle ergometer training, consisting of 45 min exercise (at 70% VO2max), 4 times in the first week and 3 times in the next 2 weeks. They performed four incremental exercise tests with the power output increased by 50 W every 3 min until volitional exhaustion: two before training (C1 and C2), and after one (T1) and three (T3) weeks of training. Before and after each load the plasma noradrenaline (NA), adrenaline (A) and blood lactate (LA) concentrations were determined in venous blood samples as well as plasma growth hormone (HGH) and cortisol concentrations before and at the end of exercise. A decrease in NA concentration was found already after 1 week of training at power output of 100 W (p<0.01) and 200 W (p<0.05). Similar decline was maintained after 3 weeks of training. No significant training-induced differences in plasma A concentration were found, however, the thresholds for both catecholamines were significantly shifted towards higher values after 3 weeks of training. One week of training caused a decrease in the pre-exercise (p<0.01), as well as post-exercise (p<0.05) plasma cortisol and HGH concentrations. It was concluded that endurance training induced a decrease in HGH, cortisol and NA concentration already after one week of training. A decline of pre-exercise plasma HGH and cortisol levels with time of experiment may, in part, indicate familiarization to exercise protocol.

Ograniczanie wyników

Dissipation function and the extravascular influx of circulating noradrenaline during its thermogenic control

Current concepts in clinical and laboratory assessments of autonomic nervous system activity

Increase in and in the outflowing from region after stimulation in

Effects of infusion of corticotropin-releasing factor antagonist into the locus coeruleus on freezing behavior and brain catecholamines in rats

Aminergic control of vasopressin secretion in the conscious rat

Role of the noradrenergic system in the secretory function of the corpus luteum

Changes in the vasoactive effects of nitric oxide, hydrogen sulfide and the structure of the rat thoracic aorta: the role of age and essential hypertension

Noradrenaline and learning - dependent cortical plasticity

Age-dependent influence of prenatal stress on the catecholamine level in adrenals and blood glucose concentration in immobilized rats

Modulation of sympathoadrenergic contractions by perivascular adipose tissue in mesenteric arteries of rats with different level of body adiposity