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1

Intestinal nematode infection afects metastasis of EL4 lymphoma cells

100%
Donskow-Lysoniewska K., Krawczak K., Machcinska M., Brodaczewska K.
Archivum Immunologiae et Therapiae Experimentalis
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2020
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tom 68
|
nr 5
2

BW5147 and derivatives for the study of T cells and their antigen receptors

100%
White J., O'Brien R. L., Born W. K.
Archivum Immunologiae et Therapiae Experimentalis
|
2020
|
tom 68
|
nr 3
3
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Lethal acute liver failure in a 60 y/o female patient with AILD III (Ann Arbor) T-cell lymphoma in remission state - case report

100%
Kuczynska M., Dziaduch E., Grzywa-Celinska A., Lachowska-Kotowska P., Mosiewicz J.
Journal of Pre-Clinical and Clinical Research
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2014
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tom 08
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nr 1
4

Changes in glucocorticoid-induced apoptosis and in expression of Bcl-2 protein during long-term culture of thymic lymphoma

100%
Kobzdej M., Matuszyk J., Ziolo E., Strzadala L.
Archivum Immunologiae et Therapiae Experimentalis
|
2000
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tom 48
|
nr 1
5

Thy28 partially prevents apoptosis induction following engagement of membrane immunoglobulin in WEHI-231 B lymphoma cells

84%
Toyota H., Jiang X. Z., Asakura H., Mizuguchi J.
Cellular and Molecular Biology Letters
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2012
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tom 17
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nr 1
Thy28 protein is conserved among plants, bacteria, and mammalian cells. Nuclear Thy28 protein is substantially expressed in testis, liver, and immune cells such as lymphocytes. Lymphocyte apoptosis plays a crucial role in homeostasis and formation of a diverse lymphocyte repertoire. In this study, we examined whether Thy28 affects induction of apoptosis in WEHI-231 B lymphoma cells following engagement of membrane immunoglobulin (mIg). Once they were established, the Thy28-overexpressing WEHI-231 cells showed similar expression levels of IgM and class I major histocompatibility complex (MHC) molecule compared with controls. The Thy28-overexpressing cells were considerably resistant to loss of mitochondrial membrane potential (ΔΨm), caspase-3 activation, and increase in annexin-positive cells upon mIg engagement. These changes were concomitant with an increase in G1 phase associated with upregulation of p27Kip1. The anti-IgM-induced sustained activation of c-Jun N-terminal kinase (JNK), which was associated with late-phase hydrogen peroxide (H2O2) production, was partially reduced in the Thy28-expressing cells relative to controls. Taken together, the data suggest that in WEHI-231 B lymphoma cells, Thy28 regulates mIg-mediated apoptotic events through the JNK-H2O2 activation pathway, concomitant with an accumulation of cells in G1 phase associated with upregulation of p27Kip1 in WEHI-231 B lymphoma cells.
6

Pretreatment levels of vascular endothelial growth factor in plasma predict a complete remission rate and time to relapse or progression in patients with diffuse large B-cell lymphoma

84%
Lech-Maranda E., Bienvenu J., Broussais-Guillaumot F., Michallet A.-S., Warzocha K., Bilinski P., Boyle P., Coiffier B., Salles G.
Archivum Immunologiae et Therapiae Experimentalis
|
2013
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tom 61
|
nr 2
7

The influence of toll-like receptor stimulation on expression of EBV lytic genes

84%
Siennicka J., Trzcinska A., Czescik A., Dunal-Szczepaniak M., Lagosz B.
Polish Journal of Microbiology
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2013
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tom 62
|
nr 3
Epstein-Barr virus (EBV) establishes latency in the resting memory B-cell compartment. It has been recently suggested that maintenance of chronic infection is dependent on periodic reactivation. Although the stimuli for EBV reactivation in vivo during natural infections are largely unknown, there is evidence indicating that heterologous infections could trigger herpesviruses reactivation. The purpose of this work was to identify the influence of Toll-like receptors stimulation on EBV replication in EBV latently infected Burkitt lymphoma cells (P3HR-1, Raji and Namalwa). The cells were stimulated with Pam3CSK4 (synthetic triacylated lipoprotein), PolyI:C (synthetic analog of dsRNA), LPS (lipopolysaccharide from E.coli), measles virus (MeV) and PMA (phorbol myristate acetate). Non-stimulated cells (NS) served as control. EBV expression was investigated at mRNA level for three viral lytic genes: BZLF1 (immediate early, ZEBRA), BALF2 (early, EA) and BcLF1 (late, VCA). Additionally, the effect of stimulation on NF-kBp65 and inflammatory cytokines (IL-1b, IL-6, IL-8, IL-10, IL-12p70, and TNF) was investigated. Stimulation of TLRs led to limited changes in EBV expression manifesting as increase of ZEBRA at mRNA level in cells treated with PolyI:C and Pam3CSK4. Stimulation with PolyI:C, Pam3CSK4 and LPS also lead to considerable increase of NF-kBp65, while increased levels of inflammatory cytokines were observed for IL-8, TNF and IL-6 in cells treated with PMA and MeV. In conclusion, the results of our experiments support the suggestion that TLRs stimulation with microbial ligands influences EBV virus replication.
8

Loss of T cell receptor diminished tumorigenicity of thymocyte-derived lymphoma cells in the T cell receptor transgenic mice

67%
Kobzdej M., Matuszyk J., Ziolo E., Strzadala L.
Archivum Immunologiae et Therapiae Experimentalis
|
1997
|
tom 45
|
nr 4
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