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The paper reviews the concept of public goods, indicating its various dimensions. The fundamental objective of the paper is conceptualizing public goods and answering a question whether a public goods delivered by agricultural land is valorized in land prices. The aim of the study was also to identify the mechanism that makes intrinsic land utility transformed into productivity in monetary units. A conducted research consists in deriving a land rent capitalized in land prices and estimating its share in land value in comparison with the share of lease fees in the different regions of Poland in years 2000–2009. In the authors opinion since accession of Poland to the UE a market has valorized intrinsic utilities of land, whereas the new role of capital and labour is distribution of those utilities in favour of consumers. Given the fact that there is a lack of Polish scientifi c studies on agricultural areas as a provider of public goods fi ndings of foreign scientists were also used in the research. Since the beginning of human civilization, the land has been creating certain utilities which satisfy human needs. When the dangerous side effects of industrial agriculture have occurred intrinsic land utilities are being discovered anew. They have a nature of public goods and constitute a hard core of the sustainable agriculture paradigm. Despite irreversible accumulation of capital in the anthropogenic environment many new utilities of the land come into existence without additional capital and labour inputs. Since they are public goods, they are paid from taxes in great measure. This way an intrinsic land utility takes a form of a paid product and can be called “intrinsic productivity” of land.
The disease is characterised by cobalamin (Cbl) deficiency in children 0-5 years old, causing failure to thrive, infections, megaloblastic anaemia, neuropathy, and mild general malabsorption; slight proteinuria is common. Cbl injections produce remission, but Cbl malabsorption and proteinuria persist. About 250 cases have been reported. Dogs also have it. The heredity is autosomal and recessive. The physiological and pathological absorption mechanisms are described: Cbl liberated from food by digestion is first bound to haptocorrin, but in the intestine it is transferred to intrinsic factor. In the ileum the complex attaches to a receptor on the enterocytes; this requires neutral pH and Ca2+. The receptor is a membrane-bound glycoprotein consisting of multiple subunits. The receptor-ligand complex is endocytosed and degraded in lysosomes, and the vitamin is transferred to transcobalamin which carries it to tissues. The same receptor is strongly expressed in the kidneys, but urine also contains its activity which can be assayed for diagnosis. The basic lesion is an error in the ileal receptor. In the affected dogs the synthesised receptor is retained intracellularly. Urine and ileal biopsies from human cases contained little receptor but it had conserved affinity for the ligand. Recently examined Arab patients did not excrete reduced amounts of the receptor. Apparently, the disease has subsets, such as different structural errors in the receptor and possibly faulty transport inside the enterocyte. The cause of the proteinuria is unknown but kidney damage due to severe Cbl deficiency and an error in a multiligand renal receptor are among the possibilities.
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