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1
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Non-invasive ventilation in chronic obstructive pulmonary disease

100%
Rasche K., Hader C., Leidag M., Duchna H-W., Orth M.
Journal of Physiology and Pharmacology. Supplement
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2004
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tom 55
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nr 3
115-119
Non-invasive ventilation (NIV) via nasal or full face mask can be applied in respiratory failure due to chronic obstructive pulmonary disease (COPD) both in the acute and long-term settings. In acute exacerbation of COPD with respiratory failure it may be considered as a standard treatment. There is strong evidence that NIV might reduce intubation, complication, and mortality rate in patients with acute hypercapnia (PaCO2 >45 mmHg). The method is cost effective and reduces hospital stay. NIV might be also useful in the weaning of COPD patients from invasive ventilation. On the other hand, for severe stable disease the data concerning a positive effect of NIV are less convincing. Preliminary evidence suggests that NIV improves gas exchange, sleep quality and quality of life and might reduce the need for hospitalization. Especially, COPD patients with substantial chronic hypercapnia (PaCO2 >55 mmHg) and/or nocturnal hypoventilation, and those with repeated exacerbations may profit from NIV. In any case, NIV is a very valuable and effective tool in the non-pharmacological treatment of COPD. Every clinician who is involved in the management of COPD patients with respiratory insufficiency should be able to apply this technique.
2
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Reflex respiratory responses to progressive hyperoxic hypercapnia and normocapnic hypoxia in normocapnic and hypercapnic obstructive sleep apnea patients

75%
Tafil-Klawe M., Klawe J. J., Sikorski W., Drzewiecka B.
Journal of Physiology and Pharmacology. Supplement
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2004
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tom 55
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nr 3
135-138
Ventilatory responses to progressive hypercapnia were analyzed in the normocapnic and hypercapnic obstructive sleep apnea patients (OSA). The rebreathing hypercapnic and hypoxic tests were performed using the computerized equipment (Lungtest, MES), according to Read's method. The ventilatory response to hypoxia was impaired in all OSA patients. Concerning the hypercapnic ventilatory response, there were no differences between the OSA patients with normal end-tidal PCO2 and controls. Nine moderately hypercapnic OSA patients showed a right shift with a normal slope of the regression curve describing the relationship between the end-tidal PCO2 and minute ventilation. In contrast, three severely hypercapnic OSA patients showed a right shift with a decreased slope of this regression curve. We conclude that awake OSA patients who developed hypercapnic ventilatory insufficiency showed an impaired hypercapnic defense reaction.
3
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Respiratory responses to acute intermittent hypoxia and hypercapnia in awake rats

75%
Sokolowska B., Rekawek A., Jozwik A.
Journal of Physiology and Pharmacology. Supplement
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2008
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tom 59
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nr 6
659-667
4
Artykuł dostępny w postaci pełnego tekstu - kliknij by otworzyć plik
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The airway obstruction - important abrupt condition for pediatrics

75%
Tomaszewski M., Banakiewicz K., Jajko K., Szypowski R., Zielinski P., Zalewski G., Spisacka S., Olchowik G.
Health Problems of Civilization
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2015
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tom 09
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nr 1
The ability of fast response in case of increasing health problems of a child which lead directly to life-threatening situations is a necessary condition for creating opportunity of child’s survival until it will be transported to intensive care unit, where it will be provided with care by a specialist. In case of healthy children the most common cause of acute respiratory failure is obstruction of upper respiratory tract. There is an enormous variety of causes of upper airway obstruction, but the most important are the result of congenital defects, acute inflammation, anaphylactic reactions, foreign body aspiration and injuries. Consequence of the hypoventilation resulting from significant impediment of airflow through the obstructed airways is impaired gas exchange in the lungs. This leads to the increasing hypoxemia (PaO 2 <60 mmHg) and hypercapnia (PaCO2> 45 mmHg). This condition is called the total respiratory failure. The persistence of hypoventilation leads to hypoxia of vital organs (heart muscle, brain), increased anaerobic metabolism, acidosis, and inevitably to cardiac arrest as a result of homeostasis disorders. Respiratory failure is defined as acute when developing suddenly and is potentially reversible. We can find such a situation in the fast-increasing stenosis of the larynx. Symptoms of severe dyspnoea occur in a short time, but can be interrupted by an effective airway patency. Acute respiratory failure is a state of direct threat to life, which is why it is crucial to give a prompt aid to the sick child. The aim of this paper is to discuss the signs and symptoms, knowledge of which is essential for rapid identification and initial differentiation of the causes of acute upper airway obstruction in children. The principles of first-aid for children with acute respiratory failure and above all the description of life-saving procedures will be presented.
5
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Expression of anion exchanger 3 influences respiratory rate in awake and isofurane anesthetized mice

63%
Meier S., Hubner C. A., Groeben H., Peters J., Bingmann D., Wiemann M.
Journal of Physiology and Pharmacology. Supplement
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2007
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tom 58
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nr 5
6
Content available remote

Normocapnia following noninvasive ventilation in acute exacerbations and chronic state of obstructive pulmonary disease

63%
Laier-Groeneveld G., Gietl C., Bauer J. U.
Journal of Physiology and Pharmacology. Supplement
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2007
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tom 58
|
nr 5
7
Artykuł dostępny w postaci pełnego tekstu - kliknij by otworzyć plik
Content available

Role of the endogenous nitric oxide in the vasodilatory tone and CO2 responsiveness of the rostral ventrolateral medulla microcirculation in the rat

63%
Wolk R., Nowicki D., Sieminska J., Trzebski A.
Journal of Physiology and Pharmacology
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1995
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tom 46
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nr 2
8
Content available remote

Transient O2-dependent effects of CO2 on ventilation in the anesthetized mouse

63%
Pokorski M., Izumizaki M., Homma I.
Journal of Physiology and Pharmacology
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2005
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tom 56
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nr 3
In this study we sought to determine the effects of background hyperoxia on the ventilatory response to hypercapnia. We addressed this issue by examining the temporal profile of the first minute transients of minute ventilation, and its frequency and tidal components, in response to 5% and 10% CO2 each co-applied with the natural (balanced with air) and hyperoxic (balanced with O2) levels of oxygen. The study was performed on the urethane-anesthetized, tracheostomized, spontaneously breathing mouse, placed in a flow-through body plethysmograph. We identified an early suppressant effect of CO2-in-O2 on breathing frequency. The frequency declined to 88.5 ±1.4% and 87.8 ±1.9% relative to the pre-test, baseline level for 5% and 10% CO2, respectively. There was a compensatory rise in tidal volume and no major change in the overall ventilation. In contrast, CO2-in-Air resulted in ventilatory stimulation caused in equal measure by frequency and tidal components. Thus, the inhibitory effect on breathing frequency of the CO2-in-O2 resulted from the O2 content in the mixture and had the temporal characteristics consistent with carotid body function. In conclusion, transient O2-dependent effects can bear on the nascent hypercapnic ventilatory response. The complexity of the O2-CO2 interaction regarding the breathing pattern components should be taken into account while designing the optimal conditions for a hypercapnic test.
9
Artykuł dostępny w postaci pełnego tekstu - kliknij by otworzyć plik
Content available

Modulation of human by mild, brief and

63%
Trzebski A., Smith M. L., Beightol L. A., Fritsch-Yelle J. M., Rea R. F., Eckberg D. L.
Journal of Physiology and Pharmacology
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1995
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tom 46
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nr 1
10

Dual response of cerebrocortical blood flow and arterial blood pressure to transient CO2 stimulus after inhibition of nitric oxide synthesis in rats

63%
Wolk R., Sieminska J., Trzebski A.
Acta Neurobiologiae Experimentalis
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1995
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tom 55
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nr 2
11
Content available remote

Significance of pulmonary vagal afferents for respiratory muscle activity in the cat

51%
Marek W., Muckenhoff K., Prabhakar N. R.
Journal of Physiology and Pharmacology. Supplement
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2008
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tom 59
|
nr 6
407-420
12

The influence of lidocaine on the permeability of the blood-cerebrospinal fluid barrier in experimental acute hypercapnia in the rabbit

51%
Naskret M., Platkiewicz M., Billert H., Drobnik L., Dyderski S.
Acta Neurobiologiae Experimentalis
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2001
|
tom 61
|
nr 2
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