Wyniki wyszukiwania - AGRO

1

Wplyw nadmiaru hormonow tarczycy na uklad sercowo-naczyniowy

100%

Noszczyk-Nowak A., Paslawska U., Nicpon J., Glinska K.

Medycyna Weterynaryjna

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2007

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tom 63

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nr 08

887-890

Thyroid hormones have an effect on every tissue of the organism because they carry out the functions of a catalyst of the oxidize reaction and the main regulator of metabolism. The thyroid hormones have an effect on the heart and cardiovascular system through β-receptors and they are synergistic with catecholamine. The excess and the deficiency of thyroid hormones in an organism is a cause of disorders in the cardiovascular system. These disorders are demonstrated as strong and nagging signs in animals and humans with hyperthyroidism and hypothyroidism. The disorders of heart functioning in hyperthyroidism may result in congestive heart failure (CHF). The development of heart failure in hyperthyroidism is connected with multidirectional changes in the cardiovascular system.

2

Znaczenie hormonów tarczycowych w laktacji u owiec

100%

Molik E., Flis Z., Staron M., Hell

Roczniki Naukowe Zootechniki

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2019

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tom 46

|

nr 1

3

Rewers trojjodotyronina - synteza i rola

100%

Bobek S.

Medycyna Weterynaryjna

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2006

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tom 62

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nr 12

1363-1365

The article discusses proposed physiological effects of reverse T3 (rT3), its synthesis and degradation. Until recently rT3 has been considered to be a metabolically inactive hormone present in high concentrations during the neonatal period and in various stress situations, including neoplasm development. Reverse T3 given to animals with unchanged hormonal homeostasis, especially of hormones participating in glucose and lipids metabolism seems to lack any metabolic effect. A fluctuation in circulating glucose and free fatty acids (FFA) levels is probably necessary for rT3 activity. In contrast to T3, rT3 acts hypo-metabolically by decreasing oxygen consumption, presumably as a result of reduced glucose and FFA utilization. A short latent period indicates that the action of rT3 is prevalently non-genomic. The article discusses the issue of low T3 syndrome or nonthyroidal syndrome which affects approximately 70% of hospitalized patients. The two hormones responsible for developing low T3 and high rT3 syndrome are: glucocorticoids . by inhibiting 5' deiodination and T3 synthesis followed by suppressed rT3 degradation, and adrenaline . by stimulating 5 deiodination followed by enhanced degradation of T3 and stimulated synthesis of rT3. Low T3 syndrome correlates with various cytokines. Cytokines treatment leads to the development of low Ta syndrome. In stress situations rT3 enhances the rise of glucose and FFA, whereas this rise is attenuated by rT3 when stress hormones i.e. glucocorticoids or catecholamine, are administered exogenously. We speculate that the molecular action of rT3 may be directed towards reducing T3 synthesis. It has been stated that equimolar concentrations of rT3 suppress 5.deiodination and synthesis of T3 with concomitant lower rT3 degradation. This leads to the development of T3 deficiency. T3 is a prohormone for synthesizing 3,5-diiodothyronine which, in turn, is necessary for binding and activating the cytochrom-c submit Va (Cyto-cVa) followed by increased oxidative phosphorylation and synthesis of ATP. Contrary to T3, rT3 decreases the ATP: ADP ratio. Finally, it is worth mentioning that the stimulatory-growth effect of rT3 on some tumor models in vitro was the most potent of all the various thyroid analogues. It may be suggested that lowered ATP reduces the energy supply of infected cells and thus suppresses the immune response.

4

Profil hormonów tarczycy w pęcherzykach jajnikowych kury podczas cyklu owulacyjnego

86%

Sechman A., Rzasa J., Paczoska-Eliasiewicz H., Proszkowiec M.

Zeszyty Naukowe. Przegląd Hodowlany

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2001

|

tom 57

5

Trijodotyronina i jej receptory jadrowe w procesie nowotworzenia.

86%

Puzianowska-Kuznicka M., Madej A., Krystyniak A., Stachlewska-Nasfeter A., Nauman A., Nauman J.

Postępy Biologii Komórki

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2001

|

tom 28

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nr 2

183-196

6

Molekularny mechanizm dzialania hormonow tarczycy

86%

Zabrocka L., Klimek J.

Postępy Biochemii

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2004

|

tom 50

|

nr 1

57-68

7

Zmiany poziomu hormonów tarczycy w białych pęcherzykach jajnikowych niosącej kury (Gallus domesticus)

86%

Sechman A., Rzasa J.

Roczniki Naukowe Zootechniki. Suplement

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2005

|

tom 22

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nr 2

8

Heterodimeryczne receptory jadrowe. I. Receptory witamin i hormonow.

86%

Kwiatkowska D., Kwiatkowska-Korczak J.

Postępy Biochemii

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2000

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tom 46

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nr 2

115-124

9

Zmiany stezenia hormonow tarczycy w osoczu krwi kur w czasie cyklu owulacyjnego

86%

Sechman A.

Zeszyty Naukowe. Przegląd Hodowlany

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1996

|

tom 24

235

10

Przerost serca wyzwalany podaniem L-tyroksyny per os u rosnacych swin

86%

Noszczyk-Nowak A., Paslawska U., Zysko D., Gajek J., Nicpon J., Rabczynski J., Skrzypczak P.

Medycyna Weterynaryjna

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2007

|

tom 63

|

nr 01

113-117

The aim of the experiment was to evaluate the influence of oral administration of L-thyroxine on the heart. The research material included 8 pigs of the polska bia³a zwis³oucha breed. The experimental hyperthyroidism was induced by oral administration of L-thyroxine at a dose 20 µg/kg b.w. in 4 pigs. The echocardiography examination was carried out 3 times in each pig: before hyperthyroidism outset and twice in hyperthyroxinemia. Through echocardiographic examination a statistically significant increase in LWd was stated. An increase in IVSd, decrease in LVd and a statistically significant increase in relative wall thickness and left ventricular mass in the group of pigs administered L-tyroxine was observed. A statistically significant decrease in SF connected with an increase in the left ventricle wall thickness occurred as well. The performed autopsy revealed an increased thickness of the right and left heart ventricles in swine with the experimental hyperthyroidism. It was stated that hyperthyroxinemia induces heart muscle hypertrophy which concerns mainly the ventricular muscles. Observed secondary hypertrophic cardiomyopathy may be the consequence of hyperthyroxinemia.

11

Selen a metabolizm hormonów tarczycy

86%

Rosolowska-Huszcz D.

Żywienie Człowieka i Metabolizm

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1999

|

tom 26

|

nr 3

264-272

12

Genomowy kontekst i regulacja ekspresji receptorów hormonu tarczycy przez długie naturalnie występujące antysensowne transkrypty

86%

Master A., Nauman A.

Postępy Biologii Komórki

|

2014

|

tom 41

|

nr 1

13

Ostre zatrucie hormonami tarczycy

72%

DeClement Safrit C.

Weterynaria po Dyplomie

|

2004

|

tom 05

|

nr 5

49-52

14

Hormonalna aktywnosc tarczycy w okresie zimowego anestrus oraz wybranym cyklu plciowym sezonu rozrodczego klaczy

72%

Niezgoda J., Tischner M., Wronska-Fortuna D.

Medycyna Weterynaryjna

|

2002

|

tom 58

|

nr 11

877-880

15

Nerwowo-hormonalna kontrola procesów związanych z powstawaniem żółci. Część V. Rola hormonów wpływających na metabolizm komórkowy

72%

Romanski K.

Życie Weterynaryjne

|

2009

|

tom 84

|

nr 03

16

Zmiany morfologiczne narządów rozrodczych kury w stanie hipo- lub hipertyreozy

72%

Sechman A., Hrabia A., Rzasa J.

Zeszyty Naukowe. Przegląd Hodowlany

|

2001

|

tom 57

17

Endokrynopatie u koni. Niedoczynnosc tarczycy. Czesc III. Diagnostyka hormonalna

72%

Slebodzinski B.

Życie Weterynaryjne

|

2003

|

tom 78

|

nr 03

143-147

18

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Satellite association frequency in persons with goiter

72%

Ferenc T., Bratkowska W., Maziarz Z., Lopaczynska D., Gaszynska M.

Genetica Polonica

|

1994

|

tom 35

|

nr 4

333-344

19

Czynnosc tarczycy u nowo narodzonych zrebiat rasy konik polski

72%

Graffstein J., Niezgoda J., Tischner M.

Roczniki Naukowe Zootechniki. Suplement

|

2005

|

tom 22

|

nr 1

97-100

20

Wplyw niektorych czynnikow zywieniowych i wysilku fizycznego na metabolizm hormonow tarczycy

72%

Rosolowska-Huszcz D.

Rozprawy Naukowe i Monografie. Szkoła Główna Gospodarstwa Wiejskiego w Warszawie

|

1998

|

tom 218

1-134

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