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Czasopisma help

  1. 8 Journal of Physiology and Pharmacology

  2. 7 Journal of Physiology and Pharmacology. Supplement

  3. 3 Archivum Immunologiae et Therapiae Experimentalis

  4. 1 Acta Microbiologica Polonica

  5. 1 Bulletin of the Polish Academy of Sciences. Biological Sciences

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Autorzy help

  1. 6 Bak-Romaniszyn L.

  2. 6 Czkwianianc E.

  3. 5 Chmiela M.

  4. 5 Rudnicka W.

  5. 4 Planeta-Malecka I.

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Lata help

  1. 1 2013

  2. 1 2010

  3. 2 2009

  4. 1 2008

  5. 1 2003

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1

Helicobacter pylori eradication and antral intestinal metaplasia - two years follow-up study

100%
Ciok J., Dzieniszewski J., Lucer C.
Journal of Physiology and Pharmacology. Supplement
|
1997
|
tom 48
|
nr 4
2

Macroscopic and histopathological examination of the gastric mucosa in dogs with inflammatory bowel disease [IBD] treated with mesalazine

84%
Rychlik A., Nieradka R., Kander M., Depta A.
Polish Journal of Veterinary Sciences
|
2009
|
tom 12
|
nr 2
217-223
The objective of this study was to carry out a macroscopic and histopathological examination of the gastric mucosa in dogs with inflammatory bowel disease and to evaluate the effect of mesalazine therapy on histopathological changes in the gastric mucosa. The treatment was performed on 18 dogs with histopathologically confirmed inflammation of the duodenum and the jejunum. The animals were administered mesalazine at 12.5 mg/kg b.w. bid over a six-week period. The dogs investigated were diagnosed with chronic catarrhal gastritis with intense lymphocytic/plasmocytic infiltration. In 83% of the dogs, intestinal metaplasia had been noted in bioptates before examination. The results of macroscopic and microscopic examinations revealed that the applied treatment had a positive effect on gastric mucosal morphology. Cellular infiltration of the gastric mucosa was significantly inhibited, most probably, due to the anti-inflammatory effect of mesalazine.
3

Effects of high vitamin C doses on Helicobacter pylori infection and vitamin C concentration in gastric juice

84%
Jarosz M., Dzieniszewski J., Dabrowska-Ufniarz E., Wartanowicz M.
Journal of Physiology and Pharmacology. Supplement
|
1997
|
tom 48
|
nr 1
4

Pepsinogen I and II in serum in Helicobacter pylori [Hp] positive patients with NUD

84%
Duda D., Rzeszutko M., Knapik Z.
Journal of Physiology and Pharmacology. Supplement
|
1997
|
tom 48
|
nr 1
5

Serological assessment H.pylori infection in children and adults with chronic gastritis and gastroduodenitis

84%
Czkwianianc E., Chmiela M., Lawnik M., Rechcinski T., Bak-Romaniszyn L., Planeta-Malecka I., Rudnicka W.
Journal of Physiology and Pharmacology. Supplement
|
1997
|
tom 48
|
nr 1
6

Immune response to Helicobacter pylori in gastritis and duodenal ulcer in children

84%
Bak-Romaniszyn L., Zeman K., Czkwianianc E., Malecka-Panas E., Fornalczyk E.
Journal of Physiology and Pharmacology. Supplement
|
1997
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tom 48
|
nr 1
7

The frequency of Helicobacter pylori in gastric mucosa in patients with rosacea

84%
Ciesla B., Andrzejewska E., Klincewicz H., Szkaradkiewicz A.
Journal of Physiology and Pharmacology. Supplement
|
1997
|
tom 48
|
nr 1
8

Serological differentiation of Helicobacter pylori CagA[plus] and CagA[minus] infections

84%
Chmiela M., Wisniewska M., Bak-Romaniszyn L., Rechcinski T., Planeta-Malecka I., Bielanski W., Konturek S. J., Plonka M., Klink M., Rudnicka W.
Archivum Immunologiae et Therapiae Experimentalis
|
2003
|
tom 51
|
nr 2
9

Interleukin-1 gene polymorphisms in chronic gastritis patients infected with Helicobacter pylori as risk factors of gastric cancer development

67%
Hnatyszyn A., Wielgus K., Kaczmarek-Rys M., Skrzypczak-Zielinska M., Szalata M., Mikolajczyk-Stecyna J., Stanczyk J., Dziuba I., Mikstacki A., Slomski R.
Archivum Immunologiae et Therapiae Experimentalis
|
2013
|
tom 61
|
nr 6
10

Automated recognition and counting of the immunoreactive neuroendocrine cells in chronic gastritis (the preliminary study)

67%
Markiewicz T., Jochymski C., Slodkowska J., Kozlowski W.
Folia Histochemica et Cytobiologica
|
2009
|
tom 47
|
nr 4
11
Content available remote

Helicobacter pylori lipopolysaccharide activity in human peripheral blood mononuclear leukocyte cultures

67%
Grebowska A., Moran A. P., Bielanski W., Matusiak A., Rechcinski T., Rudnicka K., Baranowska A., Rudnicka W., Chmiela M.
Journal of Physiology and Pharmacology
|
2010
|
tom 61
|
nr 4
437-442
Helicobacter pylori (H. pylori) have been recognized as a major cause of chronic gastritis, gastric and duodenal ulcers and gastric cancer. Macrophages are the targets of lipopolysaccharide (LPS), which is a constituent of the outer membrane of Gram-negative rods. In this study we focused on a potential role of macrophages in the proliferation of human peripheral blood mononuclear leukocytes (PBML) in the milieu of H. pylori LPS and standard E. coli LPS. First, we found that H. pylori and E. coli LPS induced proliferation of total PBML (tPBML) from 5 out 21 healthy blood donors (LPS responders). In the LPS milieu, tPBML from the majority of volunteers (LPS non-responders) showed a significant decrease in the [3H]-thymidine incorporation as compared to tPBML in medium alone. The decreased cell proliferation was associated with a diminished metabolic activity of non-adherent lymphocytes. Then, non-adherent lymphocytes were stimulated with autologous macrophages pulsed with bacterial LPS. Still, the lymphocytes from the non-responders did not proliferate in the cultures with LPS exposed macrophages. In the group of LPS responders, the macrophages pulsed with H. pylori LPS significantly reduced the proliferation of non-adherent lymphocytes. The possible mechanism regulating the responses of PBML to bacterial LPS with an implication for the outcome of H. pylori infections is discussed.
12

Serum gastrin in dogs with acute or chronic gastritis and positive or negative for Helicobacter sp. in the stomach

67%
Guzelbektes H., Coskun A., Ortatatli M., Sen I.
Bulletin of the Veterinary Institute in Puławy
|
2008
|
tom 52
|
nr 3
The relationship between gastrin levels and presence of Helicobacter-like organisms (HLO) in dogs with acute or chronic gastritis was investigated. For this purpose, a total of 52 crossbred Kangal Dogs aging 2-5 years were used; including 12 dogs with acute gastritis, 25 dogs with chronic gastritis, and 15 healthy dogs. Blood samples were collected from the vena cephalica. Serum gastrin levels were measured using commercial competitive binding radioimmunoassay. HLO were found in 64%, 58.3%, and 60% of animals with chronic gastritis or acute gastritis, and controls, respectively. The gastrin levels in dogs with chronic gastritis were higher than in those with acute gastritis and controls. There were no significant differences between the gastrin levels in all groups, with regard to HLO positive versus HLO negative dogs. In conclusion, HLO incidence was generally found in high levels in all the dogs. However, HLO did not primarily affect the stomach mucosal damage. In the mean time, there was no relation between the levels of serum gastrin in dogs with gastritis, either being HLO positive or negative. The results of the study showed that higher gastrin levels in dogs with gastritis were related to severe gastric lesions and mucosal damage.
13

Evaluation of three serological tests for the detection of Helicobacter pylori associated infection

67%
Gosciniak G., Matysiak-Budnik T., Przondo-Mordarska A.
Archivum Immunologiae et Therapiae Experimentalis
|
1993
|
tom 41
|
nr 5-6
14
Artykuł dostępny w postaci pełnego tekstu - kliknij by otworzyć plik
Content available

Mucosal chemokines in Helicobacter pylori infection

67%
Shimoyama T., Crabtree J. E.
Journal of Physiology and Pharmacology
|
1997
|
tom 48
|
nr 3
15

Primary resistance of Helicobacter pylori to antimicrobial agents in Polish children

67%
Rozynek E., Dzierzanowska-Fangrat K., Celinska-Cedro D., Jozwiak P., Madalinski K., Dzierzanowska D.
Acta Microbiologica Polonica
|
2002
|
tom 51
|
nr 3
16

Isotypic analysis of anti-H.pylori antibodies in sera from children after anti-H.pylori treatment

67%
Czkwianianc E., Chmiela M., Bak-Romaniszyn L., Rudnicka W., Planeta-Malecka I.
Journal of Physiology and Pharmacology. Supplement
|
1997
|
tom 48
|
nr 1
17
Artykuł dostępny w postaci pełnego tekstu - kliknij by otworzyć plik
Content available

H.pylori corpus gastritis - relation to acid output

67%
Sipponen P., Hyvarinen H., Siurala M.
Journal of Physiology and Pharmacology
|
1996
|
tom 47
|
nr 1
18
Artykuł dostępny w postaci pełnego tekstu - kliknij by otworzyć plik
Content available

Prevalence of Helicobacter pylori in children dependently on age and living conditions

67%
Czkwianianc E., Bak-Romaniszyn L., Malecka-Panas E., Suski S., Woch G.
Journal of Physiology and Pharmacology
|
1996
|
tom 47
|
nr 1
19

The ingestion of Helicobacter pylori by macrophages in the presence of sera from children with or without dyspeptic symptoms

67%
Rudnicka W., Chmiela M., Czkwianianc E., Paziak-Domanska B., Rozalska B., Planeta-Malecka I.
Bulletin of the Polish Academy of Sciences. Biological Sciences
|
1998
|
tom 46
|
nr 2
20
Artykuł dostępny w postaci pełnego tekstu - kliknij by otworzyć plik
Content available

Relationship between antibody to cytotoxin and Helicobacter pylori infection

67%
Gosciniak G., Przondo-Mordarska A., Poniewierka E., Iwanczak B., Bilska A.
Journal of Physiology and Pharmacology
|
1997
|
tom 48
|
nr 3
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