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1
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Oxytocin release after bleeding in rat: the role of sympathetic and renin-angiotensin system

100%
Lipinska S., Zebrowska-Badalla A., Lipinska J.
Journal of Physiology and Pharmacology
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2006
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tom 57
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nr 4
The aim of this experiment was to compare the role of renin-angiotensin and sympathetic nervous system in post-haemorrhagic mechanism of oxytocin release. Oxytocin content in venous dialysates was determined by radioimmunoassay. In control rats the release of oxytocin into dialysates did not change during whole experiment. The injection of captopril induced 2-fold higher oxytocin release, but caused no change in oxytocin release after bleeding. Superior cervical ganglionectomy (SCGx) 20 days before, caused 5-fold higher increase in oxytocin release than in control group. Injection of captopril in rats after SCGx, did not decrease the high level of oxytocin in dialysate. However, bleeding increased oxytocin release and 1 hour after bleeding the highest - 14-fold increase, took place. In the contrary to 14-fold increase in oxytocin release in animals with superior cervical ganglia (SCG), bleeding after SCGx caused only 2-fold higher oxytocin release. When SCGx, bleeding and injection of captopril were done simultaneously, oxytocin release remained on the control concentration level. We assumed that blockade of renin angiotensin system and sympathetic dennervation prevent the increase in oxytocin release after bleeding. On basis of our present experiments, it can be assumed that, in posthaemorrhagic oxytocin release into the blood, sympathetic innervation derived from SCGx, as well as, renin-angiotensin system are involved.
2
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Bovine neonatal pancytopenia in calves in Poland

84%
Jaskowski J. M., Polak M. P., Marszalek A., Nowak K., Frackowiak H., Olechnowicz J.
Polish Journal of Veterinary Sciences
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2012
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tom 15
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nr 3
Bovine neonatal pancytopenia (BNP), a newly emerged syndrome of discussed etiology in calves, has been diagnosed since 2006. Here we describe first cases of BNP in Poland. Between September 2008 and April 2011, 62 cases of BNP were diagnosed in dairy calves. Bleeding skin lesions were mostly pronounced in summer and early autumn. Severe thrombocytopenia was observed in all sick animals. All calves came from herds vaccinated against BVDV infection with PregSure BVD vaccine (Pfizer). Substitution of colostrum from dams of BNP positive calves with colostrum from dams from herds free of BNP was the only effective measure to avoid new cases in affected herds.
3
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Long term evaluation of biomaterial application in surgical treatment of periodontitis

84%
Zietek M., Gedrange T., Mikulewicz M.
Journal of Physiology and Pharmacology. Supplement
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2008
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tom 59
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nr 5
81-86
4

Sampling small quantities of blood from microbats

84%
Smith C. S., Jong,de C. E., Field H. E.
Acta Chiropterologica
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2010
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tom 12
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nr 1
5
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The vasopressin and oxytocin neurohypophysial content as influenced by bleeding or dehydration: effect of cholecystokinin octapeptide

84%
Lewandowska A., Glowacka A., Guzek J. W.
Journal of Physiology and Pharmacology
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1992
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tom 43
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nr 2
The effect of CCK-8 (50 ng, i. c. v.) on the neurohypophysial vasopressin and oxytocin storage was estimated in haemorrhaged (1 ml per 100 g b. w.) male Wistar rats. In another experimental series rats dehydrated for three days were given CCK-8 in a daily i. c. v. dose of 50 ng. The neurohypophysial vasopressin and oxytocin content was bioassayed by pressor effect following Dekański or milk- ejection activity in vitro following van Dongen and Hays, respectively. The decrease of neurohypophysial vasopressin and oxytocin content, brought about by dehydration, was significantly less marked in animals treated with CCK-8. The depletion of neurohypophysial vasopressin and oxytocin content in haemorrhaged animals could be completely inhibited by earlier i. c. v. administration of CCK-8. It is suggested that hypothalamic cholecystokinin may serve as a modulator of neurohypophysial function.
6
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Aspirin’s ability to induce intestinal injury in rats is dependent on bile and can be reversed if pre-associated with phosphatidylcholine

67%
Lichtenberger L. M., Phan T., Okabe S.
Journal of Physiology and Pharmacology
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2011
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tom 62
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nr 4
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