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Reducing greenhouse gas emissions and governing pollutant emissions would cause real synergy. Therefore, China has proposed achieving the target of reducing fine particulate matter (PM₂.₅) concentrations to 35 ug/m3 , as it pollutes the most. The prioritized purpose of this dissertation is aimed at constructing a comprehensive framework by integrating the PM₂.₅ target, influencing factors, and countermeasures together to shed some new light on the PM₂.₅ governing problem. A computable general equilibrium (CGE) model containing a pollution abatement block is first introduced. Accordingly, four different scenarios about the PM₂.₅ target implementation plan are designed for analyzing the impacts on China’s macroeconomics, energy demand, and environmental quality, and we establish a PM₂.₅ system dynamics model in the principle of system dynamics theory afterward. Subsequently, the model offers six various countermeasures in terms of declining the concentration of PM₂.₅ on the basis of the causality diagram. Consequently, the scenario analysis and system simulation results illustrate that the decline in PM₂.₅ concentration at annual average rates of 3.07%, 4.61%, and 1.53% from 2016 to 2020, 2021 to 2025, and 2026 to 2030 is significantly beneficial for achieving the PM₂.₅ target. Additionally, three effective countermeasures could be approximately reaching the PM₂.₅ concentration target in 2030.
The ETHYLENE-INSENSITIVE3 (EIN3)/EIN3- Like (EIL) EIN3/EILs, novel nuclear proteins, are located at the downstream position of the ethylene signal transduction pathway. LeEIL1, which is expressed in fruit throughout ripening, is key transcription factor in the ethylene signaling pathway in tomato. To reveal its function, the LeEIL1 gene has been transformed into and expressed in the ein2 mutant of Arabidopsis. The expression levels of the transgene in the single copy line, LeEIL1-ein2-b, were higher than those in the multiple-copy line, LeEIL1-ein2-a. The ethylene-insensitive phenotype of the ein2 mutant plants has been partially recovered by expression of LeEIL1. The florescence of LeEIL1-ein2-a and LeEIL1- ein2-b exceeded that of the ein2 mutant but was still less than that of wild type of Arabidopsis. The expression of four pathology-related genes (AtPR3, 4, AtPDF1.2 and AtGST2) has been analyzed in LeEIL1 transgenic ein2 mutant plants. The expression of AtPR3 and AtPR4, which was reduced in the ein2 mutant, was enhanced in the two transgenic Arabidopsis plants. The expression of the AtPDF1.2 gene was unaffected in the two transgenic Arabidopsis lines, the ein2 mutant and wild-type Arabidopsis plants. In addition, the expression level of AtGST2 in transgenic Arabidopsis plants was lower even than that in ein2 mutant and wild-type Arabidopsis plants.
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