Wyniki wyszukiwania

1

Negative signals from FcepsilonRI engagement attenuate mast cell functions

100%

Molfetta R., Peruzzi G., Santoni A., Paolini R.

Archivum Immunologiae et Therapiae Experimentalis

|

2007

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tom 55

|

nr 4

2

The small intestine in experimental giardiasis of rats. Morphological changes and mast cell response

88%

Kasprzak W., Majewska A. C., Gustowska L., Ruitenberg E. J.

Acta Protozoologica

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1991

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tom 30

|

nr 1

3

Signalling pathways regulating transcription, mRNA stabilisation and intracellular transport of cytokines in activated mast cells

88%

Dastych J., Walczak-Drzewiecka A., Adamczewska V., Trzaska D., Olszewski M., Wyczolkowska J.

Cellular and Molecular Biology Letters

|

2002

|

tom 07

|

nr Suppl.

4

Presence of histamine and mast cells in chicken oviduct

88%

Hrabia A., Rzasa J., Paczoska-Eliasiewicz H., Slomczynska M.

Folia Biologica

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2001

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tom 49

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nr 3-4

5

Mast cells in elastic cartilage of the human epiglottis

88%

Pawlicki R.

Folia Histochemica et Cytobiologica

|

1993

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tom 31

|

nr 4

6

The evaluation of quantity and distribution of murine pulmonary mast cells in experimental hemorrhagic shock

88%

Kasacka I., Humenczyk-Zybala M., Niczyporuk M., Mycko G.

Folia Histochemica et Cytobiologica

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2003

|

tom 41

|

nr 2

7

Histamine secretion from human mesenteric and adenoidal mast cells

88%

Brzezinska-Blaszczyk E.

Archivum Immunologiae et Therapiae Experimentalis

|

1992

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tom 40

|

nr 2

8

Disruption of histamine/H3 receptor signal reduces collagen deposition in cultures scar myofibroblasts

75%

Piera L., Olczak S., Kun T., Galdyszynska M., Ciosek J., Szymanski J., Drobnik J.

Journal of Physiology and Pharmacology

|

2019

|

tom 70

|

nr 2

9

Post-irradiation atrophy of spleen is not followed by depletion of local tissue mast cell

75%

Wlodarski K. H.

Bulletin of the Polish Academy of Sciences. Biological Sciences

|

1987

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tom 35

|

nr 1-3

10

Renal interstitial mast cell counts differ across classes of proliferative lupus nephritis

75%

Kaczmarczyk K., Kosalka J., Soja J., Kuzniewski M., Musial J., Okon K.

Folia Histochemica et Cytobiologica

|

2014

|

tom 52

|

nr 3

11

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The molecular mechanism of nondegranulative release of biogenic amines

75%

Uvnas B.

Journal of Physiology and Pharmacology

|

1991

|

tom 42

|

nr 2

According to current teaching biogenic amines are released by exocytos- is, i. e. by evacuation of amine storing vesicles or granules into the extracellular space. The release of transmitter amines is quantal, i. e. occurs in packs of transmitter molecules. These packs are assumed to be identical with vesicle contents, in other words, the smallest releasable quantum equals the amine content of one vesicle. However, there are experimental observations which do not fit in with this version of an exocytotic release theory. Observed quantitative discrepancies could be explained if the release mechanism allowed a fractional release of transmitter amine from several vesicles instead of the total evacuation of a few. The lack of adequate knowledge about the mechanisms of storage of biogenic amines within the vesicles has up til now rendered it difficult to envisage the machinery behind a fractional release of the amine content of a vesicle. In extensive in-vitro studies we have found that the matrices of amine storing granules (i. e. from mast cells, chromaffin cells and nerve terminals) show the properties of weak cation exchanger materials, carboxyl groups serving as amine binding ionic sites. When exposed to cations like sodium and potassium ions, the amines are released from their storage sites according to kinetics characteristic of weak cation exchangers. In vivo, amine release from cat adrenals on splanchnic nerve stimulation also occurs according to ion echange kinetics. Histamine release from mast cells is considered to occur as the result of degranulation, i. e. the expulsion of histamine storing granules to the extracellular space, a typical example of exocytosis. The granules are assumed to loose their histamine by ion exchange, Na⁺ Hi⁺, on exposure to the sodium-rich extracellular medium. However, recent observations on histamine release from superfused mast cells suggest that the release of histamine, although caused by ion exchange, is due to intracellular ion exchange at granule sites between cytoplasmic potassium and activated mast cells as the consequence of intracellular potassium ion flux across the histamine carrying granules, degranulation and extracellular histamine release from expelled granules occurring only as the result of more extensive activation. The possibility of potassium ions being involved also in an ion exchange process behind the release of other biogenic amines e. g. at nerve terminals will be proposed. The amine release will still be quantal but the size of the released quanta will not depend on the total amine content of a vesicle but on the size of the fractions thereof being released, thereby explaining many of the quantitative discrepancies attached to the current exocytotic release theory. A fractional release theory may have interesting consequences for our thinking as to the physiology and pharmacology of processes involving storage and release of biogenic amines.

12

Diagnostic immunohistochemistry for canine cutaneous round cell tumours — retrospective analysis of 60 cases

75%

Pazdzior-Czapula K., Mikiewicz M., Gesek M., Zwolinski C., Otrocka-Domagala I.

Folia Histochemica et Cytobiologica

|

2019

|

tom 57

|

nr 3

13

Correlation between scharf - bloom index and stromal modifications in the breast cancer

75%

Raica M.

Folia Morphologica

|

1988

|

tom 47

|

nr 1-4

14

Toxoplasma gondii and mast cells

75%

Dlugonska H.

Annals of Parasitology

|

2014

|

tom 60

|

nr 4

Mast cells, discovered by Paul Burnet over one century ago, have been long recognized only as inductors of IgE-dependent allergic diseases (allergy of type I, Th2 lymphocytes dependent). However, numerous recent studies have indicated that they play an essential role in many other immunological and non-immunological processes. Infection with Toxoplasma gondii elicits the induction of a strong cell-mediated immunity characterized by a highly-polarized Th1 response, which can protect against allergy. Knowledge of the contribution of mast cells to Toxoplasma invasion is still limited, and the present article discusses aspects of the relationship between mast cells and T. gondii.

15

Resident peritoneal macrophages and mast cells are important cellular sites of COX-1 and COX-2 activity during acute peritoneal inflammation

75%

Kolaczkowska E., Goldys A., Kozakiewicz E., Lelito M., Plytycz B., Van Rooijen N., Arnold B.

Archivum Immunologiae et Therapiae Experimentalis

|

2009

|

tom 57

|

nr 6

459-466

16

Preliminary evaluation of mast cells in rats with experimental fibrosarcoma induced by methylcholantrene

75%

Sawicki B., Kasacka I., Chyczewski L., Sobolewski K.

Folia Histochemica et Cytobiologica. Supplement

|

2001

|

tom 39

|

nr 1

17

Intracellular signalling pathways in IgE-dependent mast cell activation

75%

Kopec A., Panaszek B., Fal A. M.

Archivum Immunologiae et Therapiae Experimentalis

|

2006

|

tom 54

|

nr 6

18

Topographical and quantitative post mortem assessment of the rat skin mast cells

75%

Brzezinski P. M., Godlewski A.

Folia Histochemica et Cytobiologica

|

2001

|

tom 39

|

nr 2

19

Effect of sodium butyrate treatment on the granule morphology, histamine level and elemental content of the bone marrow- derived mast cell

75%

Rydzynski K., Dalen H.

Folia Histochemica et Cytobiologica

|

1994

|

tom 32

|

nr 1

20

The electrolyte barrier of the gastric mucosa and the barrier breakers

75%

Szlachcic A., Dziadus-Sokolowska A.

Biophysics of Membrane Transport

|

1994

|

tom 12

|

nr 2

Ograniczanie wyników

Negative signals from FcepsilonRI engagement attenuate mast cell functions