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1
Content available remote

Association between PNPLA3[G]/I148M variant, steatosis and fibrosis stage in hepatitis C virus - genetics matters

100%
Crisan D., Grigorescu M., Crisan N., Craciun R., Lupsor M., Radu C., Grigorescu M. D., Suciu A., Epure F., Avram L., Leach N.
Journal of Physiology and Pharmacology
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2019
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tom 70
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nr 4
2

Hepatic complications in acute pancreatitis

100%
Dlugosz J. W.
Journal of Physiology and Pharmacology. Supplement
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1998
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tom 49
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nr 2
3
Content available remote

The role of PPAR gamma agonists - rosiglitazone and 15-deoxy-delta12,14-prostaglandin J2 in experimental cyclosporine a hepatotoxicity

84%
Sikora-Wiorkowska A., Smolen A., Czechowska G., Wiorkowski K., Korolczuk A.
Journal of Physiology and Pharmacology
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2019
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tom 70
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nr 6
4

Green tea as an antioxidant which protects against alcohol induced injury in rats - a histopathological examination

84%
Baltaziak M., Skrzydlewska E., Sulik A., Famulski W., Koda M.
Folia Morphologica
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2004
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tom 63
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nr 1
Our study with animal models was designed to test the hypothesis that green tea protects against chronic (over 4 weeks) alcohol induced liver injury in rats. The research was conducted on Wistar male rats divided into 4 research groups: I — received the Libera-De Carli control diet (L-DC), II — received (L-DC) and green tea, III — received (L-DC) and ethanol and IV — received (L-DC), green tea and ethanol. When comparing groups I and II we saw less intensive steatosis in group II than in group I, which can suggest that green tea may affect the accumulation of fat in the hepatocytes and protect them against steatosis and disruption. In III, the ethanol group, the steatosis of the liver increased considerably and the green tea which was given with ethanol in group IV did not halt this, as in group IV we also observed intensive steatosis in the liver. From this data we conclude that green tea has an important, although not fully understood role in preventing liver injury.
5
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Is fatty liver always benign and should not consequently be treated?

67%
Fierbinteanu-Braticevici C., Negreanu L., Tarantino G.
Journal of Physiology and Pharmacology
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2013
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tom 64
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nr 1
6
Content available remote

Metabolic and antioxidative changes in liver steatosis induced by high-fat, low-carbohydrate diet in rabbits

67%
Birkner E., Kasperczyk S., Kasperczyk A., Zalejska-Fiolka J., Zwirska-Korczala K., Stawiarska-Pieta B., Grucka-Mamczar E.
Journal of Physiology and Pharmacology. Supplement
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2005
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tom 56
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nr 6
45-58
Significantly decreased amounts of carbohydrates and increase of fat in diet (Atkins’ diet, Jan Kwaœniewski’s diet) may interfere with liver metabolism because main source of liver energy are carbohydrates. Large amount of fatty acid in diet may induce overproduction of reactive oxygen species in mitochondria because of increased oxidation and may cause steatosis. The influence of high-fat, low-carbohydrate diet (contained 14 g% protein, 25 g% fat, 8 g% carbohydrate) and compared to usual habitual diet (13 g% protein, 3 g% fat, 28 g% carbohydrate) as a control on liver function was examined in adult male rabbits. Fodder and water was ad libitum. The experiment lasted 24 weeks. At the beginning and every six weeks rabbits were weighed and blood was taken. Plasma glucose and cholesterol concentration, activity of glutamate dehydrogenase, alanine and aspartate in plasma and liver did not change. Concentrations of triacylglycerols (TG) in plasma were lower in the study group. Activity of aldolase increased in plasma and in liver in the study group while activity of sorbite dehydrogenase decreased in plasma at the end of the experiment. Malondialdehyde (MDA) concentration increased in aorta with no changes in liver and erythrocytes. Activity of glutathione peroxidase increased in erythrocytes and liver while total SOD increased only in the liver. High-fat, low-carbohydrate diet despite the lack of growth of the body mass, modifies significantly the homeostasis of carbohydrates and antioxidants in liver and enhanced production of TG in this organ, resulting in its steatosis.
7
Content available remote

Biological role of liver X receptors

59%
Baranowski M.
Journal of Physiology and Pharmacology. Supplement
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2008
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tom 59
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nr 7
8
Content available remote

Therapy of nonalcoholic fatty liver disease: current status

51%
Duvnjak M., Tomasic V., Gomercic M., Smircic Duvnjak L., Barsic N., Lerotic I.
Journal of Physiology and Pharmacology. Supplement
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2009
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tom 60
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nr 7
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