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1
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The role of serotonergic 5-HT1A receptors in central cardiovascular regulation in hemorrhagic shock in rats

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Sowa P., Adamczyk-Sowa M., Zwirska-Korczala K., Namyslowski G., Misiolek M., Pierzchala K.
Journal of Physiology and Pharmacology
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2013
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tom 64
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nr 2
2
Content available remote

Proopiomelanocortin but not vasopressin or renin-angiotensin system induces resuscitative effects of central 5-HT1A activation in haemorrhagic shock in rats

100%
Sowa P., Adamczyk-Sowa M., Zwirska-Korczala K., Pierzchala K., Adamczyk D., Paluch Z., Misiolek M.
Journal of Physiology and Pharmacology
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2014
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tom 65
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nr 5
3
Content available remote

Effects of hypovolemia on hypercapnic ventilatory response in experimental hyperthermia

100%
Zila I., Brozmanova A., Javorka M., Calkovska A., Javorka K.
Journal of Physiology and Pharmacology. Supplement
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2007
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tom 58
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nr 5
4
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Vasopressin release in response to hypovolaemia in the conscious rat and the effect of opioid and aminergic receptor antagonists

100%
Peysner K., Forsling M. L.
Journal of Physiology and Pharmacology
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1991
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tom 42
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nr 3
Conscious rats were given i. p. polyethylene glycol (PEG) or dextran injections to compare their efficacy in inducing moderate hypovolaemia. Dextran was found unsuitable, producing large variability in the the plasma vasopressin (A VP) concentrations. Putative neurotransmitters involved in the A VP response to hypovolaemia and in basal release were examined using opioid, and ß-adrenoceptor and dopamine receptor-blocking agents. A dose of PEG was chosen to produce a decrease in blood volume of approx 14.5% giving plasma AVP concentrations of 19.0±4.6 pmol/1. Naloxone and phenoxy- benzamine failed to influence AVP release under both hypovolaemic and basal conditions. Prazosin also failed to influence the AVP response. In contrast propranolol elevated the plasma AVP concentrations in both conditions. Haloperidol enhanced basal AVP release but did not influence release during hypovolaemia. Guanethidine pretreatment partially blocked the response to hypovolaemia, but did not affect basal plasma AVP. Thus it appears that aminergic pathways have an inhibitory influence on AVP release under hypovolaemic and basal conditions. However, endogenous opioids do not appear to contribute significantly to the hypovolaemic response.
5
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Galanin affects vasopressin and oxytocin release from the hypothalamo-neurohypophysial system in haemorrhaged rats

100%
Ciosek J., Cisowska A., Dabrowski R.
Journal of Physiology and Pharmacology
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2003
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tom 54
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nr 2
The effect of centrally administered galanin (Gal; 100 pM i.c.v.) on the hypothalamo-neurohypophysial storage as well as blood plasma level of vasopressin and oxytocin was estimated in haemorrhaged (1 ml per 100 g b.w.) male Wistar rats. Gal i.c.v. treatment did not alter vasopressin and oxytocin content both in the hypothalamus and neurohypophysis as well as their concentration in blood plasma of not haemorrhaged rats. Haemorrhage decreased the hypothalamic and neurohypophysial vasopressin and oxytocin storage but increased the neurohormones plasma level in animals injected with vehicle solution. During the haemorrhage, the increase in plasma vasopressin and oxytocin was inhibited in rats previously treated i.c.v. with galanin. The hypothalamic and neurohypophysial vasopressin as well as oxytocin content significantly increased in animals treated with galanin and subsequently haemorrhaged. These results suggest that galanin may have a regulatory role in the hypothalamo-neurohypophysial function especially under condition of hypovolemia.
6

Cisnienie tetnicze psow - wskaznik hemodynamiki i poziomu znieczulenia

70%
Ratajczak K., Treter T.
Acta Scientiarum Polonorum. Medicina Veterinaria
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2002
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tom 01
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nr 1
5-19
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