Wyniki wyszukiwania

1

Protein catabolism in chronic uremia: is it due to malnutrition?

100%

Ahuja T. S., Mitch W. E.

Archivum Immunologiae et Therapiae Experimentalis

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2004

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tom 52

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nr 5

2

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Modulation of LDL catabolism by sodium nitroprusside and [PGE2] in human peripheral blood lymphocytes

100%

Miszczuk-Jamska B., Wybranska I., Hartwich J., Dembinska-Kiec A., Gryglewski R. J.

Journal of Physiology and Pharmacology

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1994

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tom 45

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nr 2

3

Age-dependent lipid and glycogen synthesis in the cricket fat body and regulation by adipokinetic hormone

84%

Lorenz M. W.

Pestycydy

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2005

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nr 3

117-124

4

Pathways of purine metabolism: effects of exercise and training in competitive athletes

84%

Zielinski J., Kusy K.

Trends in Sport Sciences

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2015

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tom 22

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nr 3

Introduction. The main part of skeletal muscle adenosine- 5'-triphosphate (ATP) is restored by inosine monophosphate (IMP) reamination in the purine nucleotide cycle. The intramuscular resources of IMP may be resynthesized via the quick and economical salvage pathway, in which muscle hypoxanthine (Hx) is reconverted to IMP by hypoxanthineguanine phosphoribosyl transferase (HGPRT). IMP is subsequently reutilized in the adenine nucleotide (AdN) pool. Inosine and Hx, which flow out of the skeletal muscle, represent the loss of AdN precursors. In the latter case, full restoration of resting ATP levels depends on a slow and energy-consuming de novo pathway. Plasma Hx and erythrocyte HGPRT are indirect indicators of muscle metabolism, particularly of AdN degradation, that reflect exercise- and training-induced muscle energy status. Results. Our analyses of long-term training cycles in different sports show that plasma Hx concentration and erythrocyte HGPRT activity significantly change in consecutive training phases. Both high-intensity sprint training and endurance training incorporating high-intensity exercise lead to a decrease in plasma Hx levels and an increase in erythrocyte HGPRT activity. The lowest Hx concentration and the highest HGPRT activity are observed in the competition phase characterised by low-volume and high-intensity training loads. Training cessation in the transition phase brings about a reverse phenomenon: an increase in Hx levels and a decrease in HGPRT activity. Conclusions. Low plasma purine levels indicate that the administered training adapts the athletes to high-intensity exercise (more economical AdN use, limited purine efflux from muscle into the blood). Such an adaptation is of great importance for contemporary elite athletes. Purine metabolites are more sensitive markers of training status and better performance predictors than typical biochemical and physiological indicators (e.g. blood lactate and oxygen uptake) in highly-trained athletes of different specializations and ages. The use of Hx and HGPRT for monitoring and control of the training process is worthy of consideration.

5

Variability of selected hematological and biochemical markers in marathon runners

84%

Kaminska J., Podgorski T., Pawlak M.

Trends in Sport Sciences

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2015

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tom 22

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nr 3

Introduction. The marathon is an increasingly popular form of athletic competition, especially among amateur runners. It seems, therefore, important to determine how this type of exercise affects the runner’s body. Aim of Study. The aim of the study was to identify hematological and biochemical changes in the blood of amateur runners after a marathon run. Material and Methods. The study involved 10 male runners aged from 28 to 42 years. Their blood was collected from the fingertip four times: a day before the competition and one, three and five days after the race, and subjected to hematological and biochemical analysis. Results. The results revealed a statistically significant increase in the number of leukocytes, including monocytes and granulocytes, creatine kinase (CK) and lactate dehydrogenase (LDH) activity, urea concentration, and ferric reducing ability of plasma (FRAP). On the other hand, the values of certain indices, such as the number of erythrocytes, hemoglobin concentration, hematocrit, and red blood cell distribution width, decreased. There were no statistically significant changes in the number of lymphocytes and thrombocytes, the concentration of total protein, albumin, and polyphenols. Conclusions. Prolonged and intense physical eexercise, such as a marathon run, causes multiple significant changes in the body. These changes can lead to health disorders and deterioration of physical fitness.

6

Expression of IgG immunoglobulin heavy chain IGHGA1 and IGHGA2 genes and absorption and catabolism of these proteins in lambs

84%

Krzyscin P., Skiba E., Wegrzyn J.

Annals of Animal Science

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2002

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tom 02

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nr 1

7

Purine nucleotide catabolism in rat liver labelling of uric acid and allantoin after administration of various labelled precursors

84%

Marinello E., Pagani R., Arezzini L., Porcelli B., Cinci G., Terzuoli L.

Cellular and Molecular Biology Letters

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1999

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tom 04

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nr 3

8

Katabolizm glikokoniugatow tkanek szczurow narazonych na metale ciezkie

51%

Koniusz Z., Zwierz K., Moniuszko-Jakoniuk J., Arciuch L., Pikutin A.

Acta Poloniae Toxicologica. Suplement

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1994

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tom 02

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nr 1

75-82

Ograniczanie wyników

Protein catabolism in chronic uremia: is it due to malnutrition?

Modulation of LDL catabolism by sodium nitroprusside and [PGE2] in human peripheral blood lymphocytes

Age-dependent lipid and glycogen synthesis in the cricket fat body and regulation by adipokinetic hormone

Pathways of purine metabolism: effects of exercise and training in competitive athletes

Variability of selected hematological and biochemical markers in marathon runners

Expression of IgG immunoglobulin heavy chain IGHGA1 and IGHGA2 genes and absorption and catabolism of these proteins in lambs

Purine nucleotide catabolism in rat liver labelling of uric acid and allantoin after administration of various labelled precursors

Katabolizm glikokoniugatow tkanek szczurow narazonych na metale ciezkie