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New ketoimine sorbents in solid phase extraction of HPLC analysis of bisphenol A and other 'endocrine disrupting' residues in drinking water

100%
Szymanski A., Rykowska I., Wasiak W.
Polish Journal of Environmental Studies
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2006
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tom 15
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nr 3
Our study was undertaken to compare the behaviour and properties of two new ketoimine sorbents with a commercial sorbent C-18. The sorbents were used to concentrate the endocrine-disrupting compounds (EDCs) in the water, by the use at solid phase extraction (SPE). EDCs were analyzed by high performance liquid chromatography (HPLC) coupled to UV detection. The residues of bisphenol A, bisphenol A diglycidyl ether, bisphenol F and bisphenol F diglycidyl ether in the water having contact with polycarbonate plastic have been determined. The applicable concentration range was 0.5 to 100 µg/l in water samples. Detection limits were of about 0.20 µg/l for BPA and BPF, and 0.50 µg/l for diglycidyl ether derivatives. The recovery of bisphenol A introduced into water ranged from 93.3% to 97.0%, of BPF from 91.6% to 95.9%, of BADGE from 82.0% to 86.4%, and of BFDGE from 79.7 to 82.5%. The proposed method is simple and sensitive, and thus well suited for analysis of ECDs in the water.
2
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Effects of peroxisome proliferator-activated receptors-gamma ligands on dextran sodium sulphate-induced colitis in rats

80%
Celinski K., Dworzanski T., Korolczuk A., Piasecki R., Slomka M., Madro A., Fornal R.
Journal of Physiology and Pharmacology
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2011
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tom 62
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nr 3
Recent studies indicate the involvement of peroxisone proliferator-activated receptor- (PPAR-) in the inflammatory reaction. The exact mechanism of PPAR- action has not been elucidated. It is supposed that PPAR- regulates transcription of genes responsible for encoding cytokines involved in the inflammatory response. The latest studies, carried out to explain the pathogenesis of non-specific colitis, confirm beneficial effects of PPAR- agonists on attenuation of colon inflammation. The aim of the present study was to assess the effects of nuclear PPAR- activity on the course of experimental acute colitis induced by intragastric administration of dextran sodium sulphate (DSS) using the PPAR- agonist rosiglitazone and the antagonist BADGE in rats. Colitis in Wistar rats was induced by 1.5% DSS administered in drinking water for 8 days. Animals with induced colitis received rosiglitazone, bisphenol A diglycidyl ether (BADGE) or both substances. After decapitation, colons were macroscopically and histopathologically evaluated. Levels of interleukin-1ß (IL-1ß), interleukin-6 (IL-6), interleukin-10 (IL-10), tumor necrosis factor- (TNF-) and myeloperoxidase (MPO) were determined in serum and colon homogenates using ELISA. In rats with experimentally induced colitis receiving rosiglitazone, the inflammatory reaction was found to be markedly limited; ulceration, oedema and infiltration activity were reduced. The activated PPAR- inhibit the expression of proinflammatory factors, such as IL-6, TNF-, and neutrophil chemotaxis, which was evidenced by MPO reduction in serum and colon homogenates mediated by rosiglitazone. The positive effects of rosiglitazone on expression of IL-10 were also demonstrated. During the short period of observation, BADGE did not increase histopathological inflammatory markers.
3
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Additional effects of bisphenol A and paraben on the induction of calbindin-D9k and progesterone receptor via an estrogen receptor pathway in rat pituitary GH3 cells

80%
Kim S. M., Jung E. M., An B. S., Hwang I., Vo T. T., Kim S. R., Lee S. M., Choi K. C., Jeung E. B.
Journal of Physiology and Pharmacology
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2012
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tom 63
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nr 5
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