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1
Content available remote

Angiotensin-(1-7): an active member of the renin-angiotensin system

100%
Kucharewicz I., Pawlak R., Matys T., Chabielska E., Buczko W.
Journal of Physiology and Pharmacology
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2002
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tom 53
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nr 4,1
Angiotensin-(1-7) [Ang-(1-7)] is an active member of renin-angiotensin system (RAS). It counterbalances vasoconstriction, mitogenic, arrhythmogenic and prothrombotic actions of Ang II. Inducing natiuresis and diuresis opposes also the water and sodium retention produced by Ang II. Till now the specific receptor side for Ang-(1-7) has been not cloned, but the current data strongly suggest that an interaction (cross- talk) between angiotensin receptors may play a role in the effects of Ang-(1-7).
2

The contribution of AT1 and AT2 angiotensin receptors to its cognitive effects

88%
Braszko J.
Acta Neurobiologiae Experimentalis
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1996
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tom 56
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nr 1
3
Content available remote

Angiotensin 1-7 formation in breast tissue is attenuated in breast cancer - a study on the metabolism of angiotensinogen in breast cancer cell lines

63%
Bujak-Gizycka B., Madej J., Bystrowska B., Toton-Zuranska J., Kus K., Kolton-Wroz M., Jawien J., Olszanecki R.
Journal of Physiology and Pharmacology
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2019
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tom 70
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nr 4
4

Immunochistochemical detection of angiotensin receptors AT1 and AT2 in adrenal tumors

63%
Pawlikowski M., Winczyk K., Sledz B.
Folia Histochemica et Cytobiologica
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2008
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tom 46
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nr 1
51-55
5

CGP 42112A abolishes facilitation of recognition caused by angiotensin II and angiotensin II[3-7] in rats

63%
Braszko J. J., Kulakowska A., Winnicka M. M., Karwowska-Polecka W.
Acta Neurobiologiae Experimentalis
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1997
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tom 57
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nr 3
6
Content available remote

Hypotensive function of the brain angiotensin-[1-7] in sprague dawley and renin transgenic rats

63%
Dobruch J., Paczwa P., Lon S., Khosla M. C., Szczepanska-Sadowska E.
Journal of Physiology and Pharmacology
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2003
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tom 54
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nr 3
Angiotensin-(1-7) (Ang-[1-7]) is present in the brain of normotensive Sprague Dawley (SD) rats, and its hypothalamic content is elevated in TGRmRen2(27) rats (TGR) with renin dependent transgenic hypertension. The purpose of the present study was to determine the role of intrabrain Ang-(1-7) in the regulation of cardiovascular functions in SD and TGR rats under resting conditions and during haemodynamic challenge produced by rapid bleeding. Two groups of experiments were performed on conscious SD and TGR rats that were chronically instrumented with a lateral cerebral ventricle (LCV) cannula and an intraarterial catheter. Blood pressure (MAP) and heart rate period (Hp = distance between two systolic peaks) were continuously monitored: 1) under resting conditions during an LCV infusion of either artificial cerebrospinal fluid (aCSF, 5µl/hr) or Ang-(1-7) in aCSF (100 pmol/5µl/hr), and 2) before and after haemorrhage performed during LCV infusion of either aCSF or Ang-(1-7) antagonist (A-779, 4nmol/5µl/hr). Cerebroventricular infusion of Ang-(1-7) did not affect baseline MAP in the SD rats but it caused a significant decrease in blood pressure in the TGR rats. In the control experiments, haemorrhage significantly reduced MAP in the SD and TGR rats and heart rate in the TGR rats. Cerebroventricular infusion of Ang-(1-7) antagonist eliminated posthaemorrhagic hypotension in both strains and bradycardia in the TGR rats. The results indicate that intrabrain Ang-(1-7) may contribute to posthaemorrhagic hypotension and bradycardia. Moreover, the manner in which it centrally regulates the cardiovascular functions in the SD and TGR rats may be considerably different.
7

Immunohistochemical detection of angiotensin receptors AT1 and AT2 in normal rat pituitary gland, estrogen-induced rat pituitary tumor and human pituitary adenomas

63%
Pawlikowski M.
Folia Histochemica et Cytobiologica
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2006
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tom 44
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nr 3
8
Content available remote

Beneficial anti-inflammatory effect of angiotensin-converting enzyme inhibitor and angiotensin receptor blocker in the treatment of dextran sulfate sodium-induced colitis in mice

51%
Salmenkari H., Pasanen L., Linden J., Korpela R., Vapaatalo H.
Journal of Physiology and Pharmacology
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2018
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tom 69
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nr 4
9

Influence of myocardial infarction on changes in the expression of angiotensin type 1 receptor in the rat prostate

51%
Piastowska-Ciesielska A. W., Drobnik J., Zarzynska J., Dominska K., Russell J. A., Ochedalski T.
Folia Histochemica et Cytobiologica
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2011
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tom 49
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nr 3
10
Content available remote

Altered expression of angiotensin AT1a and vasopressin V1a receptors and nitric oxide synthase mRNA in the brain of rats with renovascular hypertension

51%
Jackiewicz E., Szczepanska-Sadowska E., Dobruch J.
Journal of Physiology and Pharmacology
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2004
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tom 55
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nr 4
Previous studies revealed that the brain angiotensinergic, vasopressinergic and nitrergic systems are involved in regulation of blood pressure and that their function is altered in various forms of hypertension. The purpose of our investigation was to determine whether expression of AT1a angiotensin receptors (AT1aR) mRNA, V1a vasopressin receptors (V1aR) mRNA and neuronal nitric oxide synthase (NOS1) mRNA is altered in the brain of rats with the renovascular hypertension. Eight male Sprague Dawley (SD 2K,1C) rats were subjected to constriction of the left renal artery in order to produce the renovascular hypertension whereas nine SD rats underwent the sham surgery. In both groups blood pressure was determined before and after the surgery. Four weeks after the surgery the brain fragments were harvested for determination of mRNA expression. Competitive PCR method was applied for relative quantitative analysis of V1aR mRNA, AT1aR mRNA and NOS1 mRNA in the preoptic, diencephalic, mesencephalopontine, medullary and cerebellar fragments of the brain. Blood pressure was significantly higher in the 2K,1C than in the sham operated rats. In the preoptic, mesencephalopontine and medullary regions AT1aR mRNA expression was significantly lower in the 2K,1C rats than in the sham operated rats. The 2K,1C rats manifested also significantly higher expression of V1aR mRNA and NOS1 mRNA in the preoptic brain region in comparison to the sham operated rats. The study provides evidence for significant changes of expression of AT1aR mRNA, V1aR mRNA and NOS1 mRNA in the specific brain regions of rats with the renovascular hypertension.
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