Intestinal parasitic diseases in Gaza Strip are a significant health problem. The main aim of this study is to determine the prevalence of parasitic infection among patients in the five governorates of the Gaza Strip. A crosssectional parasitological survey was conducted on 600 patients. Stool specimens were examined microscopically for the presence of parasite eggs or larvae/cysts per wet amount by the saline iodine and formol-ether concentration method. Of 600 subjects examined, 245 (40.8%) were infected with one or more intestinal parasites. Entamoeba histolytica/dispar and Giardia lamblia were the most prevalent parasites amongst the population (28.8%), (9.5%). Female patients demonstrated a higher prevalence of intestinal parasitic infections (42.7%) than males (39.0%). However, this difference was not significant (p>0.05). According to occupation, the rate of infection was highest among farmers (56.0%), followed by employers (44.2%) with laborers showing the lowest rate (30.17%). These differences in occupational prevalence were found to be statistically significant (p<0.05). The present study demonstrates that intestinal protozoan infections are still a public health problem in the Gaza strip, with Entamoeba and Giardia infections being most common. Therefore, it is necessary to develop effective prevention and control strategies, including health education and environmental sanitation improvement.
Eighty isolates of Escherichia coli were collected in Northern Palestine throughout the 1996 to 2000 period from hospitalized patients with urinary tract infections (UTIs). Resistance rates were ampicillin, 65%; co-trimoxazole, 55%; cefuroxime, 10%; cefotaxime, 7.5%; ceftazidime, 2.5%; ciprofloxacin, 12.5%; gentamicin, 6.25% and amikacin, 1.25%. No imipenem-resistant isolates were identified. To determine whether this was due to intra-hospital transmission of resistant strains, clonal structure of 10 multiple-resistant isolates was examined by genomic DNA fingerprinting by enterobacterial repetitive intergenic concensus-polymerase chain reaction (ERIC-PCR) and all were clonally distinct. Thus, these strains are likely resistant due to convergent acquisition of resistance determinants by genetically unrelated uropathogenic strains rather than epidemic spread of resistant isolates.
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