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The current paper aims to highlight: 1) Major problems due to urbanization, including land cutting, erosion, overgrazing, biodiversity loss, and climate change. 2) The impact of grazing on plant community structure and ecosystem functioning. 3) Management and conservation of natural ecosystems in Sheikh Muhammdi Peshawar. For the current work, three different sites (Zones 1, 11, 111) were selected in the local area. The populations of the local area have increased very rapidly in the last 40 to 50 years. Anthropogenic activities associated with population and industrialization in the district, with vegetation clear for developing of towns and roads, has resulted in the substitution of vegetations with the dark color surface, the temperature of the environments much higher than before, leading to the phenomenon of the urban heat island effect. This urbanization and construction work at Amman plots Sheikh Muhammadi Peshawar is causing the extinction of vegetation and there would be no more wild vegetation in the near future in that particular area.
In this study, the methanol extract of Artemisia scoparia was evaluated for its protective potential against carbon tetrachloride (CCl₄)-induced hepatic toxicity. Seven groups of mature albino rats were used in the course of the experiment and each group was treated with specific doses of plant extract and CCl₄. Silymarin was used as a standard protective drug. The results of the experiment revealed that Artemisia scoparia plant extract was successful in fighting CCl₄ toxicity as it clearly reduced the elevated levels of liver serum markers (alkaline phosphatase and alkaline aminotransferase), lipid peroxidation, nitrite content, and H₂O₂ on one side while enhancing the levels of antioxidant enzymes (catalase, peroxidase, superoxide dismutase, glutathione-s-transferase, γ-Glutamyltranspeptidase, and glutathione reductase) and protein content. It also protected DNA from the damaging effects of CCl₄. The findings of this study demonstrate that Artemisia scoparia plant extract plays a significant role in preventing the hepatic damages instigated with CCl₄ and can be used as a protective agent against oxidative stress-associated disorders.
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