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1

Carbon allowance allocation on Chinese industrial sectors in 2030 under multiple indicators

100%
Zhou J., Jin B.
Polish Journal of Environmental Studies
|
2019
|
tom 28
|
nr 3
Aiming at China’s “2030 target,” and given the key role of industrial sectors in CO₂ emission reduction, this paper selects the path of allocating carbon emission allowance among different industrial sectors. By selecting CO₂ accumulation emissions, the industry-added value and the CO₂ emission efficiency which is calculated by the super-SBM model, a carbon intensity distribution model has been established for the allocation. Considering the different preferences of policymakers, different weights are used, meanwhile, an entropy weighting method is proposed to make a contrast. The main conclusions of this paper are: 1) The tendency of decision makers to assign principles has a great impact on the distribution of responsibility for the industry’s emission reduction responsibilities. 2) CO₂ emission intensity and carbon dioxide emission efficiency of various industries in China are very different, which is mainly related to the energy consumption demand, input, and output of various industries. 3) More historical accumulated emission and carbon intensity will shoulder more intensity reduction burden, and the industrial sector with the heaviest reduction burdens are those with the two high indicators, regardless of the tendency of decision makers.
2

Elevated IL-33 promotes expression of MMP2 and MMP9 via activating STAT3 in alveolar macrophages during LPS-induced acute lung injury

71%
Liang Y., Yang N., Pan G., Jin B., Ji W.
Cellular and Molecular Biology Letters
|
2018
|
tom 23
Background: Pulmonary inflammation and endothelial barrier permeability increase in acute lung injury (ALI) and acute respiratory distress syndrome (ARDS) induced by pro-inflammatory cytokines and matrix metalloproteinases (MMPs). However, the relationship between pro-inflammatory cytokines and MMPs in ALI/ARDS remains poorly understood. Methods: A lipopolysaccharide (LPS)-induced ALI rat model was established through intratracheal instillation. The wet/dry ratios of lung tissues were measured, and bronchoalveolar lavage fluid (BALF) was collected to test protein concentrations, total cell/macrophage numbers, and pro-inflammatory cytokine levels. LPS-treated alveolar macrophages were utilized in in vitro experiments. The expression and secretion of MMPs were respectively detected using quantitative PCR, Western blotting and ELISA assays. Results: The levels of IL-33 and MMP2/9 in BALF increased in all the ALI rats with severe lung injury. LPS-induced IL-33 autocrine upregulated the expression of MMP2 and MMP9 through activating STAT3. Neutralizing IL-33 in culture medium with specific antibodies suppressed the expression and secretion of MMP2 and MMP9 in LPS-treated alveolar macrophages. Consistently, eliminating IL-33 decreased the levels of MMP2 and MMP9 in BALF and alleviated lung injury in ALI rats. Conclusion: The IL-33/STAT3/MMP2/9 regulatory pathway is activated in alveolar macrophages during acute lung injury, which may exacerbate the pulmonary inflammation.
3

Candidate gene expression affects intramuscular fat content and fatty acid composition in pigs

61%
Wang W., Xue W., Jin B., Zhang X., Ma F., Xu X.
Journal of Applied Genetics
|
2013
|
tom 54
|
nr 1
The objective of this study was to correlate the expression pattern of candidate genes with the intramuscular fat (IMF) content and fatty acid composition of the Longissimus dorsi muscle of Duroc × Shanzhu commercial crossbred pigs. Animals of both sexes were slaughtered at a body weight of about 90 kg. The IMF content and fatty acid composition of the Longissimus dorsi muscle were measured and correlated with candidate genes mRNA expression (AdPLA, ADRB3, LEPR, MC4R, PPARγ, PPARα, LPL, PEPCK, and SCD). Females presented higher IMF content (p < 0.05) than males. The total saturated fatty acid (SFA) in males was greater (p < 0.01), whereas the total monounsaturated fatty acid (MUFA) (p < 0.01) and polyunsaturated fatty acid (PUFA) (p < 0.05) were lower than in females. The expressions of AdPLA, MC4R, PEPCK, and SCD correlated with the IMF content (p < 0.05). AdPLA showed a positive association with MUFA and a negative association with SFA (p < 0.05). LEPR and MC4R were both positively and significantly associated with C18:3 and C20:0 (p < 0.05). PPARα and PPARγ were negatively correlated with SFA, and PPARγ was positively associated with MUFA (p < 0.05). LPL was positively associated with MUFA and negatively associated with SFA (p < 0.05). PEPCK was negatively correlated with PUFA (p < 0.05). SCD was positively associated with MUFA (p < 0.05). The revealed correlations may confirm that these candidate genes are important for fat deposition and fatty acid composition in pigs, and the evaluation and use of these genes may be useful for improving porcine meat quality.
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