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1

Influence of different pressures of pneumoperitoneum on the autonomic system function during laparoscopy

100%
Barczynski M., Herman R. M.
Journal of Physiology and Pharmacology. Supplement
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2001
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tom 52
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nr 2
2

The brain-gut axis, functional disturbances after vagotomy

100%
Thor P. J., Herman R. M.
Journal of Physiology and Pharmacology. Supplement
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2001
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tom 52
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nr 2
3
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Gastric emptying of liquids in patients with peptic ulcer disease

81%
Herman R. M., Thor P. J., Krystyniecki J., Weisbrodt N. W.
Acta Physiologica Polonica
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1989
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tom 40
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nr 2
Herman, R. M., Thor, P. J., Krystyniecki, J. and Weisbrodt, N. W.: Gastric emptying of liquids in patients with peptic ulcer disease. Acta Physiol. Pol., 1989, 40 .(2): 250-254. This study was designed to determine the rates of gastric emptying of Water, saline and a 20% glucose solution ,in patients with gastric and duodenal ulcers. In all subjects, gastric emptying was fastest after administration of the saline, slower with water and .the slowest with glucose. Significant statistical differences (p < 0.05) in emptying rates between normal subjects and the group with duodenal ulcer were seen when water and saline but not glucose were used. We conclude that inhibitory effects of the osmotic receptors are not changed in gastric and duodenal ulcer patients. However, the emptying rates were higher when osmotic stimulus was withheld.
4
Content available remote

Influence of vagal nerve stimulation on food intake and body weight - results of experimental studies

71%
Sobocki J., Krolczyk G., Herman R. M., Matyja A., Thor P. J.
Journal of Physiology and Pharmacology. Supplement
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2005
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tom 56
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nr 6
27-33
The paper reviews recent advances in vagal nerve stimulation for the control of food intake and body weight. The vagal nerves are the predominant pathway in the "brain-gut axis" responsible for short term regulation of food intake. Stimulation of afferent vagal traffic attenuates food intake by vagal projections to nucleus tractus solitarius, arcuate nucleus and its convergence’s to thalamic center of satiety. A few studies have been published in this field so far. All of them are consistent and show significant decrease in body mass during vagal stimulation. Due to promising results of experimental studies, clinical trials are expected in the near future.
5
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Disturbances of the parasympathetic branch of the autonomic nervous system in patients with gastroesophageal reflux disease [GERD] estimated by short-term heart rate variability recordings

71%
Dobrek L., Nowakowski M., Mazur M., Herman R. M., Thor P. J.
Journal of Physiology and Pharmacology. Supplement
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2004
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tom 55
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nr 2
77-90
Gastro-esophageal reflux disease (GERD) is the result of the acid contents regurgitation back from the stomach into the esophagus. According to the endoscopic findings, GERD can be divided into two main forms: non-erosive (NERD) and erosive reflux esophagitis. The pathogenesis of GERD is associated with the impaired function of the antireflux barrier. Disturbances of the autonomic nervous system (ANS), especially parasympathetic part of the ANS, may be also involved in the pathogenesis of this disease. The aim of our study was to establish the parasympathetic activity in patients with reflux esophagitis and in patients with symptomatic endoscopically negative reflux. Working hypothesis was the question, whether the possible parasympathetic activity disturbances, which are observed in all GERD patients, may be regarded as the primary or secondary to the esophagitis. All the participants (20 pts. with NERD, 20 pts. with reflux esophagitis and 20 healthy controls) underwent esophageal manometry, 24-hour ambulatory pH-monitoring, resting heart rate variability (HRV) recording and the deep breathing (DB) test with the continuous HRV recording. The results of the spectral analysis both of the short-term, resting HRV recordings and DB-evoked revealed the disturbances of the main power spectra components - LF and HF in both groups of patients in comparison with the control group. In our opinion, the observed HRV spectra changes in both groups of patients support the hypothesis that not only is the parasympathetic activity impairment associated with the pathogenesis of GERD but it is also the primary factor contributing to the pathophysiological mechanism of reflux.
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