Wyniki wyszukiwania - AGRO

1

The activity of aminoacyl-tRNA synthetases in stomach cancer

100%

Pasternak K., Paluszkiewicz P., Borkowski T.

Applied Biology Communications. Lublin Scientific Center

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1993

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tom 03

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nr 5-6

2

Bacteria in krill (Euphausia superba Dana) stomach

88%

Rakusa-Suszczewski S., Zdanowski M. K.

Acta Protozoologica

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1989

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tom 28

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nr 1

3

Preliminary evaluation of gastric endocrine cells in rats with experimental uremia

88%

Kasacka I., Azzadin A., Sawicki B., Dadan J., Malla H., Buczko W.

Folia Histochemica et Cytobiologica

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2001

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tom 39

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nr 2

4

Digenea in notothenioid fish in the Beagle Channel (Magellanic sub-region, sub-Antarctica)

75%

Jezewski W., Zdzitowiecki K., Laskowski Z.

Acta Parasitologica

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2014

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tom 59

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nr 1

Fish of five species of Notothenioidei (104 specimens), Cottoperca trigloides, Patagonotothen brevicauda, P. longipes, P. tessellata and Champsocephalus esox, caught in the Beagle Channel (Magellanic sub-region, sub-Antarctica) were infected with Digenea of nine species (1130 specimens). Faunistic data on the occurrence of all nine parasites are provided. The most abundant digenean species was Macvicaria magellanica found in the intestine of three host species of the genus Patagonotothen. The second most abundant digenean species was Elytrophalloides oatesi found in the stomach of four host species, with exception of P. brevicauda. Three digenean species: Stenakron kerguelense, Whitegonimus ozoufae and Genolinea bowersi, were more abundant in fish caught at the harbor of Ushuaia (depth 7–9 m), remaining six species: M. magellanica, Neolepidapedoides subantarcticus, Postmonorchis variabilis, Derogenes varicus, E. oatesi and Lecithaster macrocotyle, in the eastern mouth of the Beagle Channel (depth 30 m).

5

The presence of Helicobacter-like microorganisms in the gastric mucosa in dogs

75%

Sapierzynska R., Malicka E., Bielecki W., Sendecka H.

Polish Journal of Veterinary Sciences

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2003

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tom 06

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nr 4

6

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Effect of multiprobiotic “Symbiter acidophilic” concentrated on morphofunctional changes in stomach evoked by 28-days introduction of omeprazole

75%

Tsyryuk O. I., Beregova T. V.

Journal of Pre-Clinical and Clinical Research

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2010

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tom 04

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nr 1

7

Evaluation of the time-stability of an alternative research model based on isolated rat gastrointestinal strips

75%

Chlopecka M., Dziekan N., Mendel M., Bakala A., Maldyk J., Wiechetek M.

Journal of Physiology and Pharmacology. Supplement

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2007

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tom 58

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nr 3

73-86

8

Preliminary results on exogenous ghrelin administration via stomach tube influence on the crypt cell proliferation in the small intestine mucosa of neonatal piglets

75%

Slupecka M., Wolinski J.

Journal of Animal and Feed Sciences

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2007

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tom 16

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nr 3

445-451

9

The morphology and development of tunica muscularis in domestic pig's stomach in prenatal period

75%

Chroszcz A., Pospieszny N.

Acta Biologica Cracoviensia. Series Botanica. Supplement

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2008

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tom 50

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nr 1

10

The relationship between extracellular K-plus and Ca2-plus on aminopyrine accumulation in rat parietal cells

75%

Ostrowski J., Zych W., Dolowy K., Butruk E.

Journal of Physiology and Pharmacology

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1991

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tom 42

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nr 3

The effects of extracellular K⁺ in relation to extracellular Ca²⁺ on acid production were studied. Studies were performed in vitro using isolated cells from rat stomachs, and acid production was indirectly determined by ¹⁴C-aminopyrine (AP) accumulation. In the absence of K⁺ in the incubation medium histamine-stimulated AP accumulation ratios were significantly decreased independently in the presence or absence of extracellular Ca²⁺. Under basal conditions, in the absence of extracellular Ca²⁺ , increasing concentrations of extracellular K⁺ enhanced AP accumulation ratios to significantly higher than those found in the presence of Ca²⁺. In histamine-, cAMP-, and carbachol-stimulated parietal cells, high K⁺ concentrations increased AP accumulation significantly less in Ca²⁺-free than in Ca²⁺-containing media. High K⁺ also induced significantly both an increase in cytosolic free Ca²⁺ concentration and ⁴⁵ Ca²⁺ uptake. The present results confirmed the importance of K⁺ in gastric acid production and suggested a role for Ca²⁺ as a modulator of mechanisms of parietal cell stimulation.

11

Gastric and duodenal ulcer and concomitant diseases among rural population

75%

Schabowski J. P.

Annals of Agricultural and Environmental Medicine

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1994

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tom 01

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nr 2

12

Gastric acid inhibitory profile of ebrotidine, a novel H2-receptor antagonist in humans

75%

Maczka M., Kwiecien N., Obtulowicz W., Konturek S. J., Kaminski K., Oleksy J., Gabryelewicz A., Torres J.

Journal of Physiology and Pharmacology

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1992

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tom 43

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nr 2

This study was designed to assess the gastric secretory effects of ebrotidine, a novel H₂ receptor antagonist, in humans. Three groups (A, В and C) of male subjects with normal gastric mucosa were used. Group A (6 subjects) was used to determine the dose-dependency of gastric inhibitory effect of ebrotidine on basal and pentagastrin- induced maximal acid output. Group В (8 subjects) was employed to examine the duration of the inhibitory effect of ebrotidine on basal and pentagastrin-induced acid secretion. In group C (6 subjects), the 24 h pH-metry was assessed using intraluminal pH-electrode placed in the gastric corpus and connected to a portable recording unit. Single oral dose of ebrotidine (200, 400 or 800 mg) caused a dose- dependent reduction in basal and pentagastrin-induced acid secretion that at a dose of 800 mg amounted to about 89% and 93%, respectively. This inhibition was still observed after 6h and averaged 72% and 50%, respectively. After 12 and 24 h upon the drug intake, both basal and pentagastrin-induced acid secretion returned to the control values. Single oral dose of ebrotidine (800 mg) caused a significant reduction in circadian acidity and resulted in a marked and significant reduction of intragastric acidity for about 6 h upon the administration. This inhibition was accompanied by a transient increase in basal and postprandial gastrin levels. We conclude that ebrotidine is highly effective inhibitor of basal, pentagastrin-induced and circadian gastric acid secretion in humans.

13

The detection of Helicobacter pylori in paraffin sections using the PCR technique and various primers as compared to histological techniques

75%

Ciesielska U., Dziegiel P., Jagoda E., Podhorska-Okolow M., Zabel M.

Folia Morphologica

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2004

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tom 63

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nr 2

Helicobacter pylori is thought to represent a significant etiopathogenic factor in diseases of the upper gastrointestinal tract. It seems, therefore, important to elaborate effective techniques for its detection. The aim of the present study was to evaluate the effectiveness of Helicobacter pylori detection using the PCR technique on paraffin sections with various pairs of primers and to compare the results with those of a histological appraisal. Material for the studies involved 50 paraffin blocks with gastric mucosa biopsies fixed in 4% buffered formalin. In this material 4 tests were performed with the aim of diagnosing Helicobacter pylori infection: 1) H+E staining, 2) staining by the Giemsa technique, 3) an immunocytochemical technique with antibodies against H. pylori and 4) the PCR technique with various primers. In the present study the most reliable results for H. pylori detection as well as the most pronounced correlation were obtained by using the PCR technique with primers for the ureC gene, immunohistochemistry and staining according to Giemsa. Less compatible results were obtained employing the two PCR techniques which utilise various primers. The experiments confirmed the usefulness of the PCR technique in the detection of Helicobacter pylori in paraffin sections by using a suitable pair of primers, and also indicated that Giemsa staining and immunohistochemistry should be taken into account.

14

Interleukin-1 and experimental gastric ulcer healing in the rat

75%

Schuerer-Maly C. C., Emmenegger U., Ott K., Flogerzi B., Halter F.

Journal of Physiology and Pharmacology

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1993

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tom 44

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nr 1

15

Metric analysis of stomach and intestine growth in the coypu fetuses

75%

Frackowiak H., Godynicki S., Jackowiak H.

Folia Morphologica

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1998

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tom 57

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nr 1

16

Ghrelin attenuates the development of acute pancreatitis in rats

75%

Dembinski A., Warzecha Z., Ceranowicz P., Tomaszewska R., Stachura J., Konturek S. J., Konturek P. C.

Journal of Physiology and Pharmacology

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2003

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tom 54

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nr 4

Ghrelin, a circulating growth hormone-releasing peptide isolated from human and rat stomach, stimulates growth hormone secretion, food intake and exhibits gastroprotective properties. Ghrelin is predominantly produced by a population of endocrine cells in the gastric mucosa, but its presence in bowel, pancreas, pituitary and hypothalamus has been reported. In human fetal pancreas, ghrelin is expressed in a prominent endocrine cell population. In adult pancreatic islets the population of these cell is reduced. The aim of present study was to investigate the influence of ghrelin administration on the development of acute pancreatitis. Methods: Acute pancreatitis was induced in rat by caerulein injection. Ghrelin was administrated twice (30 min prior to the first caerulein or saline injection and 3 h later) at the doses: 2, 10 or 20 nmol/kg. Immediately after cessation of caerulein or saline injections the following parameters were measured: pancreatic blood flow, plasma lipase activity, plasma interleukin-1ß (IL-1ß) and interleukin 10 (IL-10) concentration, pancreatic DNA synthesis, and morphological signs of pancreatitis. Results: Administration of ghrelin without induction of pancreatitis did not affect significantly any parameter tested. Caerulein led to the development of acute edematous pancreatitis. Treatment with ghrelin at the dose 2 nmol/kg, during induction of pancreatitis, was without effect on pancreatic histology or biochemical and functional parameters. Treatment with ghrelin at the dose 10 and 20 nmol/kg attenuated the development of pancreatitis and the effects of both doses were similar. Administration of ghrelin (10 or 20 nmol/kg) reduced inflammatory infiltration of pancreatic tissue and vacuolization of acinar cells. Also, plasma lipase activity and plasma IL-1ß concentration were reduced, and caerulein-induced fall in pancreatic DNA synthesis was reversed. Administration of ghrelin at the dose 10 and 20 nmol/kg was without effect on caerulein-induced pancreatic edema and pancreatitis-related fall in pancreatic blood flow. Conclusions: (1) Administration of ghrelin attenuates pancreatic damage in caerulein-induced pancreatitis; (2) Protective effect of ghrelin administration seems to be related the inhibition in inflammatory process and the reduction in liberation of pro-inflammatory IL-1ß.

17

Mucosal adenosine deaminase activity and stump ulcer healing

75%

Namiot Z., Namiot A., Stasiewicz J., Marcinkiewicz M., Jaroszewicz W., Gorski J.

Journal of Physiology and Pharmacology

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1995

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tom 46

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nr 2

18

Cerebral and gastric histamine system is altered after portocaval shunt

75%

Fogel W. A., Michelsen K. A., Panula P., Sasiak K., Andrzejewski W.

Journal of Physiology and Pharmacology

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2001

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tom 52

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nr 4,1

Biochemical parameters of the histamine (HA) system were examined in both rat brain and stomach, after portocaval anastomosis (PCA). These tissues become rich in histamine after PCA. Immunocytochemistry was used for brain histamine localisation. In addition to increased HA concentrations, monoamine oxidase B activity increased in both tissues. In hypothalamus HA was 15 fold; in cerebral cortex and in stomach mucosa 2.8 and 2.5 fold of the corresponding controls, respectively. MAO B activity was increased by approximately 50% in brain and 100% in stomach. A significant, uneven increase in tele-methylhistamine concentration was only found in the brain. In stomach mucosa higher histidine decarboxylase activity was found. PCA and sham rats treated with an irreversible inhibitor of MAO B, FA-73, 0.5 mg/kg i.p., showed 24 h later greatly reduced MAO activity and doubled t-MeHA concentration in brain structures. The treatment had no effect on gastric mucosal t-MeHA concentration and on urinary excretion of the t-MeHA metabolite, N-tele-methylimidazoleacetic acid. The HA rise in the stomach of PCA rats is associated with proliferation of histamine producing and storing cells (ECL cells) as demonstrated by others. However, in the brain we saw no indication for increased number of relevant cells either mast cells or neurons and our immunocytochemical findings suggest that in PCA rat brain, histamine deposits are located exclusively in neurons. The data indicate that the adaptative mechanisms to excessive histamine formation are tissue specific.

19

The versatility of H.pylori in its adaptation to the human stomach

75%

Blaser M. J.

Journal of Physiology and Pharmacology. Supplement

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1997

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tom 48

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nr 1

20

Role of nitric oxide in gastroduodenal alkaline secretion

75%

Bilski J., Konturek S. J.

Journal of Physiology and Pharmacology

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1994

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tom 45

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nr 4

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