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  1. 18 Journal of Physiology and Pharmacology. Supplement

  2. 3 Journal of Physiology and Pharmacology

  3. 2 Acta Neurobiologiae Experimentalis

  4. 1 Folia Histochemica et Cytobiologica

Autorzy help

  1. 3 Gorecka D.

  2. 3 Jernajczyk W.

  3. 3 Namyslowski G.

  4. 3 Plywaczewski R.

  5. 3 Scierski W.

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  1. 2 2015

  2. 1 2013

  3. 1 2011

  4. 5 2008

  5. 7 2007

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1
Content available remote

Mask leakage in continuous positive airway pressure and C-Flex

100%
Leidag M., Hader C., Keller T., Meyer Y., Rasche K.
Journal of Physiology and Pharmacology. Supplement
|
2008
|
tom 59
|
nr 6
401-406
2
Content available remote

The nasal airway evaluation in morbid obesity

75%
Namyslowski G., Mrowka-Kata K., Scierski W., Wylezol M., Pardela M.
Journal of Physiology and Pharmacology. Supplement
|
2005
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tom 56
|
nr 6
67-75
The aim of the present study was to evaluate objectively the nasal patency in the obese patients. A total of 18 morbidly obese patients were recruited for the study. All of them were surgically treated because of morbid obesity using Bilo-Pancreatic Diversion (BPD) or Laparoscopic Gastric Banding (LGB) methods. The patients were free of nasal abnormalities, such as septum deviation, polyps, nasal concha hypertrophy and paranasal sinus diseases. This group comprised 10 men and 8 women aged from 17 to 54. The mean Body Mass Index (BMI) was 51.6 kg/m2, ranged from 34.7 to 61.8 kg/m2. In all of the patients the nasal patency was examined by active anterior rhinomanometry according International Standardization Rhinomanometric Committee using air pressure 75, 100 and 150 dPa. The results were compared to the healthy control group. The correlation between BMI and nasal airflow pressure was also examined. We found that inspiration values for 75, 100 and 150 dPa as well as the expiration values for 100 and 150 dPa in standard method and expiration values for 100 and 150 dPa in Broms method using anterior rhinomanometry in morbidly obese patients were statistically significant higher in comparison with the healthy controls. No statistical significant correlation between BMI of obese patients and the airflow pressure values was found. We conclude that in the morbide obesity the nasal patency is reduced as compared to the healthy controls.
3
Content available remote

Sleep study in patients with overweight and obesity

75%
Namyslowski G., Scierski W., Mrowka-Kata K., Kawecka I., Kawecki D., Czecior E.
Journal of Physiology and Pharmacology. Supplement
|
2005
|
tom 56
|
nr 6
59-65
Obstructive sleep apnea syndrome (OSAS) is a disorder characterized by repetitive collapse of the pharyngeal airway during sleep, which leads to oxygen desaturation, sleep fragmentation and daytime somnolence. Obesity is one of the most important risk factor for the development of OSAS. The exact mechanisms responsible for the relationship between obesity and OSAS are still unclear. The fat deposits in the pharynx region as well as the reduction in the lung volume have been considered as factors that might be responsible for the increase of the upper airway collapsibility. The aim of our study was to evaluate the correlation between the Body Mass Index (BMI) and sleep study parameters in overweight and obese patients suffering from breathing disturbances during sleep. We studied a group of 106 consecutive obese or overweight patients with a primary complaint of snoring or other breathing disturbances during sleep. In all cases, BMI and sleep studies (PolyMESAM) were examined. We evaluated relationship between the BMI and sleep study parameters such as Respiratory Disturbance Index (RDI), Apnea Index (AI), Desaturation Index (DI) and Average of Lowest Saturation (LSAT). The results showed the lack of significant statistical correlations between BMI and all the sleep parameters studied in the overweight patients and the statistical positive correlation between the BMI and RDI in the obese cases. We conclude that BMI determination may be considered as a simple, yet important predictor, of the OSAS in the group of obese patients.
4

Olfactometry in fMRI studies: odor presentation using nasal continuous positive airway pressure

75%
Popp R., Sommer M., Muller J., Hajak G.
Acta Neurobiologiae Experimentalis
|
2004
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tom 64
|
nr 2
5
Content available remote

Exercise capacity in patients with obstructive sleep apnea syndrome

63%
Przybylowski T., Bielicki P., Kumor M., Hildebrand K., Maskey-Warzechowska M., Korczynski P., Chazan R.
Journal of Physiology and Pharmacology. Supplement
|
2007
|
tom 58
|
nr 5
6
Content available remote

The influence of obesity on sleep quality in male sleep apnea patients before and during therapy

63%
Antczak J., Horn B., Richter A., Jernajczyk W., Bodenschatz R., Schmidt E. W.
Journal of Physiology and Pharmacology. Supplement
|
2008
|
tom 59
|
nr 6
123-134
7
Content available remote

Obstructive sleep apnea syndrome: the effect of diabetes and autonomic neuropathy

63%
Keller T., Hader C., De Zeeuw J., Rasche K.
Journal of Physiology and Pharmacology. Supplement
|
2007
|
tom 58
|
nr 5
8
Content available remote

Analysis of the correlations between oxidative stress, gelatinases and their tissue inhibitors in the human subjects with obstructive sleep apnea syndrome

63%
Hopps E., Lo Presti R., Montana M., Canino B., Calandrino V., Caimi G.
Journal of Physiology and Pharmacology
|
2015
|
tom 66
|
nr 6
9
Content available remote

Oxidative stress markers in the blood of persons with different stages of obstructive sleep apnea syndrome

63%
Cofta S., Wysocka E., Piorunek T., Rzymkowska M., Batura-Gabryel H., Torlinski L.
Journal of Physiology and Pharmacology. Supplement
|
2008
|
tom 59
|
nr 6
183-190
10
Content available remote

Sleep related breathing disorders in patients with ischemic stroke and transient ischemic attacks: respiratory and clinical correlation

63%
Rola R., Wierzbicka A., Wichniak A., Jernajczyk W., Richter P., Ryglewicz D.
Journal of Physiology and Pharmacology. Supplement
|
2007
|
tom 58
|
nr 5
11
Content available remote

Melatonin and omentin: influence factors in the obstructive sleep apnoea syndrome?

63%
Zirlik S., Hildner K. M., Targosz A., Neurath M. F., Fuchs F. S., Brzozowski T., Konturek P. C.
Journal of Physiology and Pharmacology
|
2013
|
tom 64
|
nr 3
12
Content available remote

Disturbed angiogenic activity in sera from obstructive sleep apnea patients

63%
Chorostowska-Wynimko J., Radomska D., Plywaczewski R., Jonczak L., Stepniewska A., Gorecka D., Skopinska-Rozewska E.
Journal of Physiology and Pharmacology. Supplement
|
2005
|
tom 56
|
nr 4
71-77
It is increasingly recognized that obstructive sleep apnea (OSA) syndrome is a systematic rather than local disorder. There is also growing evidence that apart from the syndrome's major features: intermittent hypoxia and sleep fragmentation, functional activity of the immune system is altered in OSA patients, with several cytokines, such as tumor necrosis factor-alpha (TNF-alpha) and interleukin-6 (IL-6) taking active part in sleep regulation. Little is known about the effects exerted by chronic intermittent hypoxia combined with persistent pro-inflammatory activity of the immune system on the vascular micro milieu in OSA. In this study we attempted to confirm the hypothesized imbalance between pro- and anti-angiogenic factors by evaluating direct and indirect angiogenic activity of OSA patients' sera in the in vivo serum-induced angiogenesis (SIA) and leukocyte-induced (LIA) assays, respectively, in mice. Both tests revealed significantly inhibited angiogenic activity of OSA patients' sera compared with healthy controls (P<0.001). Moreover, differences related to the subject’s weight regarding in the mean number of newly-formed vessels were observed with a significantly greater inhibition in the normal-weighing apneic subjects than in the overweight or obese ones (P<0.01). The angiogenesis inhibition index was positively related to the serum IL-6 level (r=0.35; P<0.05) in the OSA group, but not to TNF-alpha, fasting serum leptin, or OSA syndrome severity as assessed by the AHI index. Our results demonstrate that OSA is accompanied by disturbed serum angiogenic activity, apparently resulting from an imbalance between pro- and anti-angiogenic factors, some of them being produced by the adipose tissue. The disordered angiogenic activity might be related to the pathophysiology of OSA and should be considered an important causative factor for the increased prevalence of cardiovascular diseases in OSA patients.
13
Content available remote

Positron emission tomography findings in obstructive sleep apnea patients with residual sleepiness treated with continuous positive airway pressure

63%
Antczak J., Popp R., Hajak G., Zulley J., Marienhagen J., Geisler P.
Journal of Physiology and Pharmacology. Supplement
|
2007
|
tom 58
|
nr 5
14
Content available remote

Saliva and oxidative stress in oral cavity and in some systemic disorders

63%
Buczko P., Zalewska A., Szarmach I.
Journal of Physiology and Pharmacology
|
2015
|
tom 66
|
nr 1
15
Content available remote

Leptin measurement in urine is a reliable method of monitoring its secretion in patients with obstructive sleep apnea syndrome

63%
Chorostowska-Wynimko J., Plywaczewski R., Jonczak L., Bednarek M., Stepniewska A., Borowska A., Gorecka D.
Journal of Physiology and Pharmacology. Supplement
|
2007
|
tom 58
|
nr 5
16
Content available remote

Age-related response of the genioglossus muscle EMG-activity to hypoxia in humans

63%
Klawe J. J., Tafil-Klawe M.
Journal of Physiology and Pharmacology. Supplement
|
2003
|
tom 54
|
nr 1
14-19
The prevalence of snoring and obstructive sleep apnea syndrome increases with age. Upper airway obstruction during sleep occurs when the dilator muscles are unable to overcome the negative pharyngeal pressure. The very important muscle dilator of the upper airway is the genioglossus muscle (GG). Our previous study showed an increase in GG-EMG activity during progressive hypoxia. In the present study a hypoxic increase in GG-EMG activity in response to hypoxia was investigated in two groups of 10 healthy subjects each: aged 29.1 ±1.8 and aged 53.2 ±2.3. The older humans showed reduced response of GG to hypoxia. We suggest that the biological aging process may be a risk factor for the obstructive sleep apnea syndrome.
17
Content available remote

Sleep apnea syndrome and snoring in patients with hypothyroidism with relation to overweight

63%
Misiolek M., Marek B., Namyslowski G., Scierski W., Zwirska-Korczala K., Kazmierczak-Zagorska Z., Kajdaniuk D., Misiolek H.
Journal of Physiology and Pharmacology. Supplement
|
2007
|
tom 58
|
nr 1
77-85
18
Content available remote

Does night-shift work induce apnea events in obstructive sleep apnea patients?

51%
Laudencka A., Klawe J. J., Tafil-Klawe M., Zlomanczuk P.
Journal of Physiology and Pharmacology. Supplement
|
2007
|
tom 58
|
nr 5
19

The rheological properties of blood and the risk of cardiovascular disease in patients with obstructive sleep apnea syndrome (OSAS)

51%
Tazbirek M., Slowinska L., Kowalski M., Pierzchala W.
Folia Histochemica et Cytobiologica
|
2011
|
tom 49
|
nr 2
20
Content available remote

Frequency of distribution of inflammatory cytokines IL-1, IL-6 and TNF-alpha gene polymorphism in patients with obstructive sleep apnea

51%
Popko K., Gorska E., Potapinska O., Wasik M., Stoklosa A., Plywaczewski R., Winiarska M., Gorecka D., Sliwinski P., Popko M., Szwed T., Demkow U.
Journal of Physiology and Pharmacology. Supplement
|
2008
|
tom 59
|
nr 6
607-614
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