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1
Content available remote

Effect of low dose L-NAME pretreatment on nitric oxide/reactive oxygen species balance and vasoactivity in L-NAME/salt-induced hypertensive rats

100%
Vrankova S., Zemancikova A., Torok J., Pechanova O.
Journal of Physiology and Pharmacology
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2019
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tom 70
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nr 4
2

Localization of NADPH-diaphorase activity in the pineal gland of the domestic pig

100%
Lewczuk B., Wojtkiewicz J., Majewski M., Przybylska-Gornowicz B.
Folia Histochemica et Cytobiologica
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2001
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tom 39
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nr 2
3

The oxidative pentose phosphate pathway and glycolytic activity during dormancy breaking of Acer platanoides seeds

100%
Szczotka Z., Zymanczyk M.
Acta Physiologiae Plantarum
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1994
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tom 16
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nr 4
4
Artykuł dostępny w postaci pełnego tekstu - kliknij by otworzyć plik
Content available

Why chloroplastic ferredoxin: NADPplus oxidoreductase binds to lipids – model membranes study?

84%
Grzyb J.
BioTechnologia. Journal of Biotechnology Computational Biology and Bionanotechnology
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2013
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tom 94
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nr 3
5

9alpha,11beta-PGF2, a prostaglandin D2 metabolite, as a marker of mast cell activation in bee venom-allergic patients

84%
Nittner-Marszalska M., Cichocka-Jarosz E., Sanak M., Wujczyk M., Dor-Wojnarowska A., Lis G., Liebhart J.
Archivum Immunologiae et Therapiae Experimentalis
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2015
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tom 63
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nr 4
6
Artykuł dostępny w postaci pełnego tekstu - kliknij by otworzyć plik
Content available

Genetic analyses of cytosolic NADPH-regenerating systems reveal specific functions in oxidative stress responses

84%
Mhamdi A., Li S., Van Der Kelen K., Van Breusegem F., Noctor G.
BioTechnologia. Journal of Biotechnology Computational Biology and Bionanotechnology
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2013
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tom 94
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nr 2
7
Content available remote

15-deoxy-delta12,14-prostaglandin J2 suppresses RANTES expression by inhibiting NADPH oxidase activation in Helicobater pylori-infected gastric epithelial cells

67%
Cha B., Lim J. W., Kim K. H., Kim H.
Journal of Physiology and Pharmacology
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2011
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tom 62
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nr 2
Peroxisome proliferators-activated receptor-g (PPAR-g) is a ligand-activated transcription factor. 15 deoxy-12,14 prostaglandin J2 (15d-PGJ2) is a potent PPAR- ligand and acts as an anti-inflammatory agent via PPAR--dependent and independent mechanisms. Helicobacter pylori (H. pylori) induces gastric inflammation by inducing the activation of oxidant-sensitive transcription factor NF-B and cytokine expression in gastric epithelial cells. Since 15d-PGJ2 inhibits NF-B activation in various cells, it may suppress H. pylori-induced inflammatory signaling and cytokine expression in gastric epithelial cells. The present study aims to determined the effect of 15d-PGJ2 on the activation of inflammatory mediators Jak/Stat (Janus kinase/signal transducers and activators of transcription) and induction of cytokine RANTES in H. pylori-infected gastric epithelial AGS cells. Since NADPH oxidase is a candidate for the production of reactive oxygen species in H. pylori-infected gastric epithelial cells, we determined the effect of 15d-PGJ2 on the activation of NADPH oxdase. AGS cells were cultured in the presence of H. pylori treated with or without 15d-PGJ2. The activations of NADPH oxidase and Jak1/Stat3, the levels of H2O2 and RANTES in the medium, and DNA binding activity of Stat3 were assessed. A Jak/Stat3 specific inhibitor AG490 and an inhibitor of NADPH oxidase diphenyleneiodonium (DPI) were treated to determine the direct involvement of Jak/Stat and NADPH oxidase on the production of H2O2 and RANTES in H. pylori-infected cells. H. pylori induced the production of H2O2 and RANTES as well as the activations of NADPH oxidase and Jak1/Stat3, which were inhibited by the treatment of 15d-PGJ2. DPI suppressed H. pylori-induced alterations similar to 15d-PGJ2. However, AG490 had no effect on NADPH oxidase activation, but reduced the level of RANTES in the medium released from H. pylori-infected cells. Conclusion: NADPH oxidase activation is an upstream signaling of Jak1/Stat3 activation and induction of RANTES in H. pylori-infected AGS cells. 15d-PGJ2, inhibits the activations of NADPH oxidase and Jak1/Stat3 and RANTES expression, suggesting that 15d-PGJ2 may be beneficial for the treatment of H. pylori-induced gastric inflammation.
8

Neutrophil myeloperoxidase: soldier and statesman

59%
Prokopowicz Z., Marcinkiewicz J., Katz D. R., Chaim B. M.
Archivum Immunologiae et Therapiae Experimentalis
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2012
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tom 60
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nr 1
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