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1
Artykuł dostępny w postaci pełnego tekstu - kliknij by otworzyć plik
Content available

The methods of normalization used in the analysis of two-color microarrays

100%
Siatkowski I., Zyprych J., Handschuh L., Figlerowicz M.
Colloquium Biometricum
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2009
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tom 39
2

Gene expression profiling in Arabidopsis as influenced by lead - microarray analysis

100%
Gawronska H., Ciurzynska M., Wiench M., Gawronski S. W.
Polish Journal of Natural Sciences. Supplement
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2003
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tom 01
3
Content available remote

Toll-like receptors and the tendency of normal mucous membrane to transform to polyp or colorectal cancer

75%
Niedzielska I., Niedzielski Z., Tkacz M., Orawczyk T., Ziaja K., Starzewski J., Mazurek U., Markowski J.
Journal of Physiology and Pharmacology. Supplement
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2009
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tom 60
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nr 1
65-71
The aim of this study was to identify differences in Toll-like receptor (TLR) expression patterns in normal and diseased tissues of patients with polyps and colorectal cancer. Eight patients were included in the study group (aged 38 to 72 years). Sixteen HG-U133A oligonucleotide microarrays were analysed including four of colonic polyps, four of adenocarcinoma with different degree of histological differentiation (2 poorly and 2 highly differentiated), and eight of macroscopically normal tissue. The levels of selected TLR mRNA transcripts were analysed. An analysis of all per cent variability values with regard to malignancy stage increasing from polyp to stages I to III adenocarcinoma, and normal colon mucosa shows a statistically significant relationship for TLR2 (increasing) and TLR3 (decreasing). In polyps, copy numbers of TLR3, TLR4 and TLR5 mRNA were the highest and TLR7 mRNA the lowest. In normal colon mucosa of polyposis patients the highest mRNA copy numbers were observed for TLR3, and the lowest for TLR7. TLR3 may serve as a marker of colon tissue metaplasia and may indicate the tendency of normal tissue to form polyps transforming to colorectal cancer.
4

Expression of estrogen, estrogen related and androgen receptors in adrenal cortex of intact adult male and female rats

75%
Trejter M., Jopek K., Celichowski P., Tyczewska M., Malendowicz L. K., Rucinski M.
Folia Histochemica et Cytobiologica
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2015
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tom 53
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nr 2
5

Gene expression profiling studies using microarray in osteoarthritis: genes in common and different conditions

75%
Liu W., Jiao Y., Tian C., Hasty K., Song L., Kelly D. M., Li J., Chen H., Gu W., Liu S.
Archivum Immunologiae et Therapiae Experimentalis
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2020
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tom 68
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nr 5
6
Content available remote

Analysis of microRNA expression in canine mammary cancer stem-like cells indicates epigenetic regulation of transforming growth factor-beta signaling

75%
Rybicka A., Mucha J., Majchrzak K., Taciak B., Hellmen E., Motyl T., Krol M.
Journal of Physiology and Pharmacology
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2015
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tom 66
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nr 1
7
Artykuł dostępny w postaci pełnego tekstu - kliknij by otworzyć plik
Content available

Elevated CO2 mitigates the impact of a combination of heat wave and drought in Arabidopsis thaliana

75%
Zinta G., AbdElgawad H., Domagalska M., Vergauwen L., Nijs I., Beemster G., Asard H., Janssens I.
BioTechnologia. Journal of Biotechnology Computational Biology and Bionanotechnology
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2013
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tom 94
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nr 2
8
Artykuł dostępny w postaci pełnego tekstu - kliknij by otworzyć plik
Content available

Identification of novel genes potentially involved in rice (Oryza sativa L.) drought tolerance

63%
Zinati Z.
BioTechnologia. Journal of Biotechnology Computational Biology and Bionanotechnology
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2017
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tom 98
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nr 3
9
Artykuł dostępny w postaci pełnego tekstu - kliknij by otworzyć plik
Content available

Identification of candidate genes related to aroma in rice by analyzing the microarray data of highly aromatic and nonaromatic recombinant inbred line bulks

63%
Zinati Z., Delavari A.
BioTechnologia. Journal of Biotechnology Computational Biology and Bionanotechnology
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2019
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tom 100
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nr 3
10

Minichromosome maintenance (MCM) and AgNOR proteins expression in desmoid tumours: a tissue microarray analysis

51%
Ferenc T., Kopczynski J., Stalinska L., Tosik D., Sidor M., Lopaczynska D., Kulig A., Dziki A., Sygut J.
Folia Histochemica et Cytobiologica
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2010
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tom 48
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nr 4
11

Relapse of acute lymphoblastic leukemia in children in the context of microarray analyses

51%
Szczepanek J., Styczynski J., Haus O., Tretyn A., Wysocki M.
Archivum Immunologiae et Therapiae Experimentalis
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2011
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tom 59
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nr 1
12

Microarray analysis reveals the role of matrix metalloproteinases in mouse experimental autoimmune myocarditis induced by cardiac myosin peptides

51%
Tang Q., Huang J., Qian H., Xiong R., Shen D., Wu H., Bian Z., Wei X.
Cellular and Molecular Biology Letters
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2007
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tom 12
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nr 2
Autoimmune myocarditis develops after the presentation of heart-specific antigens to autoaggressive CD4+ T cells and after inflammation has infiltrated the tissues. To shed light on global changes in the gene expression of autoimmune myocarditis and to gain further insight into the molecular mechanisms underlying the genesis of myocarditis, we conducted a comprehensive microarray analysis of mRNA using an experimental mouse autoimmune myocarditis model via immunization with α-myosin heavy chain-derived peptides. Of over 39,000 transcripts on a high density oligonucleotide microarray, 466 were under-expressed and 241 over-expressed by ≥ 1.5-fold compared with the controls in BALB/C mouse with autoimmune myocarditis. In this paper, we list the top 50 up-regulated genes related to the immune response. These altered genes encode for leukocyte-specific markers and receptors, the histocompatibility complex, cytokines/receptors, chemokines/receptors, adhesion molecules, components of the complement cascade, and signal transduction-related molecules. Interestingly, matrix metalloproteinases (MMPs) such as MMP-3 and MMP-9 were up-regulated, as further revealed by the reverse transcriptase-polymerase chain reaction (RT-PCR) and immunohistochemistry assays. This indicates that MMPs may act as major regulators of the cytokine profile. Together, these findings provide new insight into the molecular events associated with the mechanism of the autoimmune genesis of myocarditis.
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