PL EN


Preferencje help
Widoczny [Schowaj] Abstrakt
Liczba wyników
2014 | 08 | 2 |

Tytuł artykułu

Treatment of adult patients with craniocerebral trauma complicated by venitalory failure

Treść / Zawartość

Warianty tytułu

Języki publikacji

EN

Abstrakty

EN
This article contains the current state of knowledge on modern methods of treating patients with severe cranio-cerebral injuries, hospitalized in intensive care due to respiratory failure. Severe traumatic brain injury (TBI) can be divided into primary and secondary. Primary brain damage takes place in mechanism of direct injury most often due to an impact. Secondary damage is the result of subsequent disorders of physiological conditions such as ischemia and hypoxia of the brain areas covered by the primary injury. Treatment of patients with severe cranio-cerebral injuries is focused on maintaining proper ventilation, hemodynamic stability and optimal values of intracranial pressure. Maintaining adequate blood flow through brain vessels is necessary to prevent the development of ischemic changes. Respiratory failure develops in these patients by direct injury to the brain in the form of tachypnea, bradypnoea, shortness of breath, sleep apnea, or secondarily to trauma as pneumonia, acute respiratory distress syndrome (ARDS), or pulmonary edema. Mechanical ventilation in patients with TBI seeks to maintain an oxygen partial pressure in the arterial blood of more than 11 kPa and the partial pressure of carbon dioxide in the range of 4.5 to 5.0 kPa. The use of appropriate therapy is to prevent the secondary damage to the brain or reduce complications associated with secondary damage.

Słowa kluczowe

Wydawca

-

Rocznik

Tom

08

Numer

2

Opis fizyczny

p.4-9,ref.

Twórcy

autor
  • Department of Anaesthesiology and Intensive Care, The Zofia z Zamoyskich Tarnowska Regional Hospital in Tarnobrzeg, Tarnobrzeg, Poland
  • Laboratory of Neurological Rehabilitation, Department of Rehabilitation, Physiotherapy and Balneotherapy, Medical University of Lublin, Al.Raclawickie 1, 20-059 Lublin, Poland
autor
  • Laboratory of Neurological Rehabilitation, Department of Rehabilitation, Physiotherapy and Balneotherapy, Medical University of Lublin, Lublin, Poland
autor
  • Balneotherapy Unit, Department of Rehabilitation, Physiotherapy and Balneotherapy, Medical University of Lublin, Lublin, Poland
  • Department of Diagnostic Imaging, The Zofia Zamoyski Tarnowska Provincial Hospital in Tarnobrzeg, Tarnobrzeg, Poland

Bibliografia

  • 1. Caricato A., Conti G., Delia Corte F., Mancino A., Santilli F., Sandroni C. (2005), Effect of PEEP on the intracranial system of patients with head injury and subarachnoid hemorrhage: the role of respiratory system compliance. Trauma. 58(3): s. 571-576.
  • 2. Clifton GL., Miller ER., Coi SC., Levin HS. (2002), Fluid thresholds and outcome from severe brain injury. Crit Care Med. 30(4): s. 739-745.
  • 3. Diringer M.N. (2013), New trends in hyperosmolar therapy. Curr Optin Crit Care. 19(2): s. 77-82.
  • 4. Farag E., Manno EM., Kurz A. (2011), Use of hypothermia for traumatic brain injury: point of view. Minerva Anesthesiol. 77(3): s. 336-370.
  • 5. Geraldes R., Sousa P.R., Fonseca A.C., Falcao F., Canhao P., Pinho E., Melo T. (2014), Nontraumatic convexity subarachnoid hemorrhage: Different etiologies and outcomes. Journal of Stroke and Cerebrovascular Diseases. 23(1): s. 23-30.
  • 6. Ghajar J. (2000), Traumatic brain injury. Lancet. 356: s. 923-929.
  • 7. Grape S., Ravussin P. (2012), PRO: osmotherapy for the treatment of acute intracranial hypertension. J Neurosurg Anesthesiol. 24(4): s.402-406.
  • 8. Grände PO., Romner B. (2012), Osmotherapy in brain edema: a questionable therapy. J Neurosurg Anesthesiol. (4): s. 407-412.
  • 9. Helmy A., Vizcaychipi M., Gupta AK. (2007), Traumatic brain injury: intensive care management. Br J Anaesth. (1): s. 32-42.
  • 10. Johnston AJ., Steiner LA., Chatfield DA. (2003), Effect of propofol on cerebral oxygenation and methabolism after head injury. Br J Anesth. 91: s. 781-786.
  • 11. Johnston AJ, Steiner LA, Chatfield DA. (2004), Effect of cerebral perfusion pressure augmentation with dopamine and norepinephrine on global and focal brain oxygenation after traumatic brain injury. Itens Care Med.: s. 791-797.
  • 12. Kozler P., Riljak V., Pokorný J. (2011), Methylprednisolone reduces axonal impairment in the experimental model of brain oedema. Neuro Endocrinol Lett. 32(6): s. 831-835.
  • 13. Kumar MA., Urrutia VC., Thomas CE., Abou-Khaled KJ. (2005), The syndrome of irreversible acidosis after prolonged propofol infusion. Neurocrit Care. 3: 257-259.
  • 14. Macmillan CS., Grant IS., Andrews PJ. (2002), Pulmonary and cardiac sequelae of subarachnoid haemorrhage: time for active management? Intens Care Med. 28: s. 1012-1023.
  • 15. Mansour N., deSouza R.M., Sikorski C., Kahana M., Frim D. (2013), Role of barbiturane coma In the management of focally induced, severe cerebral edema In children. 12(1): s. 37-43.
  • 16. Marik PE., Varon J., Trask P. (2002), Management of head trauma. Chest. 122(2): s. 699-711.
  • 17. Petrucci N., Iacovelli W. (2004), Ventilation with lower tidal volumes versus traditional tidal volumes n adults for acute lung injury and acute respiratory distress syndrome. Cochrane Database Syst Rev. (2): s. 384-396.
  • 18. Polderman KH., Tjong Tjinoe R., Peerdeman SM., Vandertrop WP., Girbes AR. (2002), Effect of therapeutic hypothermia on intracranial pressure and outcome in patients with severe head injury. Intens Care Med. 28: s.1563-1567.
  • 19. Scalfani M.T., Dhar R., Zazulia A.R., Videen T.O., Diringer M.N. (2013), Effect of osmotic agents on regional cerebral blood flow in traumatic brain injury. J Crit Care. 27(5): s. 7-12.
  • 20. Solaiman O., Singh J.M. (2013), Hypocapnia in aneurismal subarachnoid hemorrhage: Incidence and association with poor clinical outcomes. Journal of Neurosurgical Anesthesiology. 25(3): s. 254-261.
  • 21. Stienen M., Abdulazim A., Hildebrandt G., Gautshi O.(2013), Emergency scenario: epidural hematoma- evalution and management. Praxis (Bern). 30: s.102-103.
  • 22. Sullivan G.M., Mierzwa A.J., Kijpaisalratana N., Tang H., wang Y., Song S.K., Selwyn R., Armstrong R.C. (2013), Oligodendrocyte lineage and subventricular zone response to traumatic axonal injury in the corpus callosum. J Neuropathol Exp Neurol. 72(12): s.1106-1125.
  • 23. Tagliaferri F., Compagnone C., Korsic M. (2006), A systematic review of brain injury epidemiology in Europe. Acta Neurochir (Wien). 148: s. 255-268.
  • 24. The Brain trauma Foundation and the Joint section of the American Association of Neurological Surgeons and Congress of Neurological Surgeons on Neurotrauma and Critical Care. (2007), Guidelines for the Management of Severe Traumatic Brain Injury. 3rd edition. IX Cerebral Perfusion Thresholds. J Neurotrauma. 24: s. 59-64.
  • 25. Tobias JD. (2000), Propofol: effect on the central nervous system. J. Intensive Care Med. 15(5): s. 237-246.
  • 26. Vrettou CS., Zakynthinos SG., Malachias S., Mentzelopoulos SD. (2013), High-frequency oscillation and tracheal gas insufflations in patients with severe acute respiratory distress syndrome adn traumatic brain injury: an interventional physiological study. CritCare. 17(4): s. 136-140.
  • 27. Yan E., Satgunaseelan L., Paul E., Bye N., Nyguyen P., Agyapomaa D., Kossmann T., Rosenfeld JV., Morganti-Kossmann C. (2013), Post-traumatic hypoxia is associated with prolonged cerebral cytokine production, higher serum biomarker levels and poor outcome in patients with severe traumatic brain injury. J Neurotrauma. 11(26): s. 1146-1160.
  • 28. Ye H., Su Y. (2013), Hemodynamic effect of mannitol infusion in patients with acute intracerebral hemorrhage. Acta Cir Bras. 28(2): s. 106-111.
  • 29. Ypung PJ., Pakeerathan S., Blunt MC., Subramanya S. (2006), A low-volume, low-pressure tracheal tube cuff reduces pulmonary aspiration. Crit Care Med. 34: s. 632-639.
  • 30. Zyguan DA., Kortbeek JB., Fick GH., Laupland KB., Doig CJ. (2005), Non-neurologic organ dysfunction in severe traumatic brain injury. Crit Care Med. 33: s. 654-660.

Uwagi

PL
Rekord w opracowaniu

Typ dokumentu

Bibliografia

Identyfikatory

Identyfikator YADDA

bwmeta1.element.agro-d62029bb-560e-4026-b6f9-88387bfdeef0
JavaScript jest wyłączony w Twojej przeglądarce internetowej. Włącz go, a następnie odśwież stronę, aby móc w pełni z niej korzystać.