EN
Responses (knock-down and mortality) of large pine weevil (Hylobius abietis L.) beetles to insecticides of different mechanism of action such as neonicotinoids (clotianidin, acetamiprid) acting on nicotinic receptors of acetylcholine (ACh), pyrethroids (deltamethrin, α-cypermethrin) blocking sodium channels in nerve cell membranes, organophosphates (chlorpiryfos) and carbamates (carbosulfan) both inhibiting acetylcholinesterase (AChE) activity and fenylpirazoles (fipronil) interfering with γ-butyric acid (GABA) receptors were studied. Taking into account speed of insect decay the order of examined groups of substances may be established as follows: first place is occupied by insecticides interferring with GABA receptors, second place by insecticides blocking AChE activity, and third place in parallel insecticides blocking sodium channels and those acting on nicotinic receptors of ACh.