1. Camejo G., Hurt-Camejo E., Wiklund O., Bondjers G.: Association of apo B lipoproteins with arterial proteoglycans: Pathological significance and molecular basis. Atherosclerosis 1998, 139, 205-22.
2. Cockeill G., Rye K., Gamble J., et al.: High density lipoproteins inhibit cytokine induced expression of endothelial cell adhesion molecules. Arterioscler. Thromb.Vasc. Biol., 1995, 15, 1987-93
3. Crea E, Biasucci L., Buffon A.: Role of inflammation in the pathogenesis of unstable coronary artery disease. Am. J. Cardiol., 1997, 80, Suppl. 10-6E.
4. Cybulsky M., Jiyama K., Li H., et al.: A major role for VCAM-1, but not ICAM-1, in early atherosclerosis. J. Clin. Invest., 2001, 107, 1255-62
5. Cyms T., et al.: Disruption of 12/15 lipoxygenase diminishes atherosclerosis in apo E deficient mice. J. Clin. Invest., 1999, 103, 1597- 1604.
6. De Catterina R., Libby P., Peng H., et al. Nitric oxide decreases cytokine-induced endothelial activation; nitric oxide selectively reduces endothelial expression of adhesion molecules and proinflammatory cytokines. J. Clin. Invest., 1995, 96, 60-8.
7. Gerszten R., Mach F., Sauty A., Rosenzweig A., Luster A.: Chemokines, leukocytes and atherosclerosis. J. Lab. Clin. Med., 2000, 136, 87-92.
8. Gimbrone M.A.: Vascular endothelium, hemodynamic forces, and atherogenesis. Am. J. Pathol., 1999, 155, 1-5.
11. Ivandic B., et al.: Role of group II secretory phospholipase A2 in atherosclerosis. Increased atherosclerosis and altered lipoproteins in transgenic mice expressing group II a phospholipase A2. Arterioscler. Thromb. Vase. Biol., 1999, 19, 1284-90.
12. Lange A., Lang E., Bielak F., et al.: Autosomal genome-wide scan for coronary artery calcification loci in sibships at high risk for hypertension. Arterioscler. Thromb. Vase. Biol., 2002, 22, 418-23.
13. Li H., Cybulsky M., Gimbrone M., Libby P.: An atherogenic diet rapidly induces VCAM-1, a cytokine regulatable mononuclear leukocyte adhesion molecule, in rabbit aortic endothelium. Arterioscler. Thromb. 1993, 13, 197-204.
18. Marathe S., Kuriakose G., Williams K., Tabas I.: Sphingomyelinase, an enzyme implicated in atherosclerosis, is present in atherosclerotic lession and binds to specific components of the subendothelial extracellular matrix. Arterioscler. Thromb. Vase. Biol., 1999, 19, 2648-58.
19. Nogal T., Resnick N., Atkinson W., Dewey C., Gimbrone M.: Shear stress selectively upregulates intercellular adhesion molecule-1 expression in cultured human vascular endothelial cells. J. Clin. Invest., 1994, 94, 885-91.
20. Nowicka G.: HDL-lipoproteiny o wysokiej gęstości. Teoria a praktyka kliniczna. Kardiol. Pol., 2002, 57, 150-4.
21. Nowicka G.: Współczesne spojrzenie na antyaterogenne działanie lipoprotein o wysokiej gęstości. Czynniki Ryzyka 1998, 4, 12-15.
22. Prescott S., Mclntyre T., Zimmerman G., Stafforini D.: Sol Sherry Lecture in Thrombosis: molecular events in acute inflammation. Arterioscler. Thromb. Vase. Biol., 2002, 22, 727-33.
23. Ross R.: The pathogenesis of atherosclerosis: a perspective for the 1990s. Nature 1993, 362, 801-9.
24. Subbanagounder G., et al.: Determinants of bioactivity of oxidized phospholipids. Arterioscler. Thromb. Vase. Biol., 2000, 20, 2248-54.
25. Terkeltaub R., Boisvert W., Curtiss L.: Chemokines and atherosclerosis. Curr. Opin. Lipidol.,
1998, 9, 397-405.
26. Tremoli E., Camera M., Toschi V., Colli S.: Tissue factor in atherosclerosis. Arteriosclerosis 1999, 144, 273-83.
27. Watson K.: Pathophysiology of coronary calcification. J. Cardiol. Risk 2000, 7, 93-7.
28. Winther M., van Dijk K., Havekes L., Hofker M.: Macrophage scavenger receptor closs A: a multifunctional receptor in atherosclerosis. Arterioscler. Thromb. Vase. Biol., 2000, 20, 290-7.