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  1. 7 Archivum Immunologiae et Therapiae Experimentalis

  2. 5 Folia Histochemica et Cytobiologica

  3. 4 Cellular and Molecular Biology Letters

  4. 2 Journal of Physiology and Pharmacology. Supplement

  5. 1 BioTechnologia. Journal of Biotechnology Computational Biology and Bionanotechnology

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  1. 1 Akiyama T.

  2. 1 Bienvenu J.

  3. 1 Bilinski P.

  4. 1 Birckbichler P. J.

  5. 1 Blanck G.

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  1. 1 2020

  2. 2 2019

  3. 1 2018

  4. 2 2014

  5. 3 2013

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1

Manipulation of non-canonical NF-κB signaling by non-oncogenic viruses

100%
Struzik J., Szulc-Dabrowska L.
Archivum Immunologiae et Therapiae Experimentalis
|
2019
|
tom 67
|
nr 1
2

Role of immunohistochemical and histochemical profiling in H and E-based diagnosis of scrotal leiomyosarcoma of dartos muscle

100%
Wincewicz A., Lewitowicz P., Sulkowska U., Sulkowski S., Horecka-Lewitowicz A., Koziel D., Gluszek S.
Folia Histochemica et Cytobiologica
|
2013
|
tom 51
|
nr 4
3
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Content available

Activation-induced cytidine deaminase (AID): single activity – pleiotrophic effect

84%
Budzko L., Jackowiak P., Figlerowicz M.
BioTechnologia. Journal of Biotechnology Computational Biology and Bionanotechnology
|
2013
|
tom 94
|
nr 1
4

The impact of Di(2-ethylhexyl)phthalate on cancer progression

84%
Chou C.-K., Yang Y.-T., Yang H.-C., Liang S.-S., Wang T.-N., Kuo P.-L., Wang H.-M. D., Tsai E.-M., Chiu C.-C.
Archivum Immunologiae et Therapiae Experimentalis
|
2018
|
tom 66
|
nr 3
5

Bim: guardian of tissue homeostasis and critical regulator of the immune system, tumorigenesis and bone biology

84%
Akiyama T., Tanaka S.
Archivum Immunologiae et Therapiae Experimentalis
|
2011
|
tom 59
|
nr 4
6

Nucleolin and nucleophosmin expression in seminomas and non-seminomatous testicular tumors

84%
Masiuk M., Lewandowska M., Teresinski L., Dobak E., Urasinska E.
Folia Histochemica et Cytobiologica
|
2019
|
tom 57
|
nr 3
7

The stage-specific function of gap junctions during tumourigenesis

84%
Czyz J.
Cellular and Molecular Biology Letters
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2008
|
tom 13
|
nr 1
92-102
Tumour development is a process resulting from the disturbance of various cellular functions including cell proliferation, adhesion and motility. While the role of these cell parameters in tumour promotion and progression has been widely recognized, the mechanisms that influence gap junctional coupling during tumorigenesis remain elusive. Neoplastic cells usually display decreased levels of connexin expression and/or gap junctional coupling. Thus, impaired intercellular communication via gap junctions may facilitate the release of a potentially neoplastic cell from the controlling regime of the surrounding tissue, leading to tumour promotion. However, recent data indicates that metastatic tumour cell lines are often characterized by relatively high levels of connexin expression and gap junctional coupling. This review outlines current knowledge on the role of connexins in tumorigenesis and the possible mechanisms of the interference of gap junctional coupling with the processes of tumour invasion and metastasis.
8
Content available remote

Sirt7-dependent inhibition of cell growth and proliferation might be instrumental to mediate tissue integrity during aging

84%
Vakhrusheva O., Braeuer D., Liu Z., Braun T., Bober E.
Journal of Physiology and Pharmacology. Supplement
|
2008
|
tom 59
|
nr 9
201-212
9
Content available remote

Role of autophagy in mammary gland development

84%
Gajewska M., Sobolewska A., Kozlowski M., Motyl T.
Journal of Physiology and Pharmacology. Supplement
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2008
|
tom 59
|
nr 9
237-249
10

Epidermal differentiation complex (locus 1q21) gene expression in head and neck cancer and normal mucosa

84%
Tyszkiewicz T., Jarzab M., Szymczyk C., Kowal M., Krajewska J., Jaworska M., Fraczek M., Krajewska A., Hadas E., Swierniak M., Markowski J., Lange D., Poltorak S., Wiench M., Krecicki T., Jarzab J., Maciejewski A.
Folia Histochemica et Cytobiologica
|
2014
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tom 52
|
nr 2
11

Pretreatment levels of vascular endothelial growth factor in plasma predict a complete remission rate and time to relapse or progression in patients with diffuse large B-cell lymphoma

84%
Lech-Maranda E., Bienvenu J., Broussais-Guillaumot F., Michallet A.-S., Warzocha K., Bilinski P., Boyle P., Coiffier B., Salles G.
Archivum Immunologiae et Therapiae Experimentalis
|
2013
|
tom 61
|
nr 2
12

Tumor models for prostate cancer exemplified by fibroblast growth factor 8-induced tumorigenesis and tumor progression

84%
Tuomela J., Harkonen P.
Reproductive Biology
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2014
|
tom 14
|
nr 1
13

Upregulated miR-96-5p inhibits cell proliferation by targeting HBEGF in T-cell acute lymphoblastic leukemia cell line

84%
Xu K., Yan X., Ouyang G., Feng J., Ye L., Hu X., Liu D.
Folia Histochemica et Cytobiologica
|
2020
|
tom 58
|
nr 3
14

The inhibition of in vivo tumorigenesis of osteosarcoma [OS]-732 cells by antisense human osteopontin RNA

84%
Liu S. J., Zhang D.-Q., Sui X.-M., Zhang L., Cai Z.-W., Sun L.-Q., Liu Y.-J., Xue Y., Hu G.-F.
Cellular and Molecular Biology Letters
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2008
|
tom 13
|
nr 1
11-19
Osteopontin (OPN) is a secreted, non-collagenous, sialic acid-rich protein which functions by mediating cell-matrix interactions and cellular signaling via binding with integrins and CD44 receptors. An increasing number of studies have shown that OPN plays an important role in controlling cancer progression and metastasis. OPN was found to be expressed in many human cancer types, and in some cases, its over-expression was shown to be directly associated with poor patient prognosis. In vitro cancer cell line and animal model studies have clearly indicated that OPN can function in regulating the cell signaling that ultimately controls the oncogenic potential of various cancers. Previous studies in our laboratory demonstrated that OPN is highly expressed in human osteosarcoma (OS) cell line OS-732. In this study, we successfully reduced the tumorigenecity of OS-732 cells xenotransplanted into nude mice, using the antisense human OPN (hOPN) RNA expression vector.
15

Components of the IFN-gamma signalling pathway in tumorigenesis

84%
Blanck G.
Archivum Immunologiae et Therapiae Experimentalis
|
2002
|
tom 50
|
nr 3
16

Chemoprevention, cell adhesion, and apoptosis: report from the 87th annual meeting of the American Association for Cancer Research

84%
Waliszewska M. K., Waliszewski P., Birckbichler P. J., Hurst R. E., Hemstreet G. P.
Biotechnologia
|
1997
|
nr 3
17

Concurrent mutation in exons 1 and 2 of the K-ras oncogene in colorectal cancer

67%
Palmirotta R., Savonarola A., Ludovici G., De Marchis M. L., Covello R., Ettorre G. M., Ialongo C., Guadagni F.
Folia Histochemica et Cytobiologica
|
2011
|
tom 49
|
nr 4
18

Targeting NF-kappaB and HIF-1 pathways for the treatment of cancer: Part I

67%
Wilczynski J., Duechler M., Czyz M.
Archivum Immunologiae et Therapiae Experimentalis
|
2011
|
tom 59
|
nr 4
19

RNA interference against Biot2, a novel mouse testis - specific gene, inhibits the growth of tumor cells

67%
Wang C. T., Zhang P., Wang Y.-S., Ruan X.-Z., Li Z.-Y., Peng F., Yang H.-S., Wei Y.-Q.
Cellular and Molecular Biology Letters
|
2009
|
tom 14
|
nr 3
363-376
Biot2 is a novel murine testis-specific gene that was first identified using the SEREX technique, and named by our laboratory. Using conventional RT-PCR and real time RT-PCR, we tested the expression profile of Biot2 in normal tissues and various murine tumor cell lines. Using RNA interference, we studied the biological function of Biot2 in tumorigenesis. We applied various types of growth assay, such as the in vitro MTT, colony-forming and BrdU incorporation assays, along with in vivo tumorigenicity assays, to reveal its inhibition of tumor cell proliferation. The results revealed that the Biot2 transcript was detected only and strongly in the testis tissues and abundantly in five types of murine cancer cell line. Treating B16 murine melanoma, LL/2 murine Lewis lung carcinoma and CT26 murine colorectal adenocarcinoma with special shRNA targeting Biot2 can significantly reduce the proliferation rate of these three tumor cell lines in vitro, as measured by the MTT, colony-forming and BrdU incorporation assays. The tumorigenicity of the CT26 cells transfected with special shRNA targeting Biot2 was also decreased distinctly in vivo compared with the control. It was therefore concluded that Biot2 plays a key role in tumorigenesis and could be a potential target for biotherapy.
20
Content available remote

Mechanisms promoting physiological cells progression into tumorigenesis

67%
Korbut E., Ptak-Belowska A., Brzozowski T.
Journal of Physiology and Pharmacology
|
2012
|
tom 63
|
nr 6
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