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1

Effect of sodium butyrate treatment on the granule morphology, histamine level and elemental content of the bone marrow- derived mast cell

100%
Rydzynski K., Dalen H.
Folia Histochemica et Cytobiologica
|
1994
|
tom 32
|
nr 1
2
Content available remote

Sodium butyrate-dependent sensitization of human colon adenocarcinoma COLO 205 cells to TNF-alpha-induced apoptosis

67%
Pajak B., Orzechowski A.
Journal of Physiology and Pharmacology. Supplement
|
2007
|
tom 58
|
nr 3
163-176
3
Content available remote

Effect of sodium butyrate supplementation in milk replacer and starter diet on rumen development in calves

67%
Gorka P., Kowalski Z. M., Pietrzak P., Kotunia A., Kiljanczyk R., Flaga J., Holst J. J., Guilloteau P., Zabielski R.
Journal of Physiology and Pharmacology. Supplement
|
2009
|
tom 60
|
nr 3
47-53
4

Cytochrome P450 mRNA expressions along with in vitro differentiation of hepatocyte precursor cells from fetal, young and old rats

67%
Czekaj P., Bryzek A., Czekaj T. M., Koryciak-Komarska H., Wiaderkiewicz A., Plewka D., Sieron A. L.
Folia Histochemica et Cytobiologica
|
2010
|
tom 48
|
nr 1
46-57
5

The effect of sodium butyrate on calf growth and serum level of beta-hydroxybutyric acid

67%
Slusarczyk K., Strzetelski J. A., Furgal-Dzierzuk I.
Journal of Animal and Feed Sciences
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2010
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tom 19
|
nr 3
The experiment was carried out on 40 Polish Black-and-White HF bull calves (52-87% of HF blood) aged from 9 and 12 days at the beginning of the experiment to 90 days at its end. From the beginning of the trial the calves were offered restricted liquid feed to 56 days of age and concentrates ad libitum according to IZ-PIB-INRA recommendations. The concentrates were without sodium butyrate (control group, C), or with 1% Na-butyrate (group B1), 3% Na-butyrate (group B3) and 0.3% Na-butyrate (group B0.3), and included meadow hay from 0.10 kg/day during the liquid feeding period to 0.20 kg/day after weaning at 57 days of age. Na-butyrate at 3% in the diet reduced feed intake and had a beneficial effect on calf growth and nutrient utilization. The dietary level of Na-butyrate did not cause significant changes in serum β-hydroxybutyric acid concentration of the calves.
6
Content available remote

Effect of sodium butyrate on the small intestine development in neonatal piglets feed by artificial sow

67%
Kotunia A., Wolinski J., Laubitz D., Jurkowska M., Rome V., Guilloteau P., Zabielski R.
Journal of Physiology and Pharmacology. Supplement
|
2004
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tom 55
|
nr 2
59-68
Feeding of neonates with artificial milk formulas delays the maturation of the gastrointestinal mucosa. Na-butyrate has a complex trophic effect on the gastrointestinal epithelium in adults. The present study aimed to determine the effect of milk formula supplementation with Na-butyrate on the gut mucosa in neonatal piglets. Sixteen 3 day old piglets were randomly divided into two groups: control (C, n = 8), and Na-butyrate (B, n = 8). Animals were feed for 7 days with artificial milk formula alone (C) or supplemented with Na-butyrate (B). At the 10th day of life the piglets were sacrificed and whole thickness samples of the upper gut were taken for analyses. Administration of Na-butyrate led to significant increase in daily body weight gain as compared to control. In the duodenum, the villi length and mucosa thickness were reduced, however, in the distal jejunum and ileum, the crypt depth, villi length and mucosa thickness were increased in Na-butyrate supplemented piglets as compared to control. Supplementation with Na-butyrate did not affect the intestinal brush border enzyme activities but increased plasma pancreatic polypeptide and cholecystokinin concentrations. These results suggest that supplementation with Na-butyrate may enhance the development of jejunal and ileal mucosa in formula-fed piglets.
7

Comparative study of immunomodulatory agents to induce human T regulatory (Treg) cells: preferential Treg-stimulatory effect of prednisolone and rapamycin

67%
Janyst M., Kaleta B., Janyst K., Zagozdzon R., Kozlowska E., Lasek W.
Archivum Immunologiae et Therapiae Experimentalis
|
2020
|
tom 68
|
nr 4
8

Changes in the cellular behaviour of human colonic cell line Caco-2 in response to butyrate treatment

67%
Dzierzewicz Z., Orchel A., Weglarz L., Latocha M., Wilczok T.
Acta Biochimica Polonica
|
2002
|
tom 49
|
nr 1
Gut-derived adenocarcinoma Caco-2 cells were treated with sodium butyrate (NaB) at physiologically relevant concentrations. We characterized its effects on prolifera­tion, differentiation, apoptosis, adhesion to the solid support and interleukin-8 secre­tion. Differentiation was determined by brush border alkaline phosphatase activity. Apoptosis was assessed by acridine orange and Hoechst stains. Differentiation and apoptosis were analyzed in both adherent and floating cell populations. The trans­formed Caco-2 cells did not retain their malignant phenotype in the presence of NaB. They appeared to undergo a change in the phenotype induced by NaB, as indicated by reduced proliferation, enhanced differentiation, stimulation of apoptosis leading to decreased viability of cells, and stimulation of interleukin-8 secretion. Considering all the above facts and data, we postulate that Caco-2 cells cultured in NaB supplemented medium could regain the phenotypic characteristics of the phenotype of the parent cell from which originated the Caco-2 line.
9

Globotriaosyl ceramide (Gb3) expression in human tumour cells: Intracellular trafficking defines a new retrograde transport pathway from the cell surface to the nucleus, which correlates with sensitivity to verotoxin

59%
Lingwood C. A., Khine A. A., Arab S.
Acta Biochimica Polonica
|
1998
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tom 45
|
nr 2
The verotoxin receptor globotriaosyl ceramide (Gb3) is overexpressed in an ovarian tumour resistant to chemotherapy. An overlay of frozen tumour sections shows extensive staining of the tumour cells with verotoxin B subunit. In addition, blood vessels within the tumour mass are stained. The sensitivity of ovarian tumour cells in vitro to verotoxin can be modulated by culturing the cells in sodium butyrate to obtain an approximatly 5000-fold increase in susceptibility. This increased susceptibility is correlated with the intracellular targeting of verotoxin as monitored by using FITC-VT B subunit, in that prior to sodium butyrate treatment the toxin is internalized to a juxtanuclear (likely) Golgi location whereas, following butyrate treatment the intracellular toxin is distributed around the nucleus, consistent with endoplasmic reticulum and nuclear envelope location. This perinuclear location is similar to that found for drug-resistant variants of ovarian tumour cell lines. These results suggest that intracellular targeting of verotoxin to the perinuclear area results in increased cytotoxicity. Potentially such targeting may also occur in other human tumours.
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