Wyniki wyszukiwania - AGRO

1

Investigations on the secretion mechanism of corticosteroid hormones

100%

Nussdorfer G. G.

Folia Histochemica et Cytobiologica

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1984

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tom 22

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nr 3-4

2

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Adenosine deaminase activity in the human duodenal mucosa in relation to gastric acid secretion

88%

Namiot Z., Stasiewicz J., Marcinkiewicz M., Gorski J.

Journal of Physiology and Pharmacology

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1992

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tom 43

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nr 2

Adenosine deaminase (ADA) activity was estimated in mucosal specimens obtained endoscopically from the duodenal bulb. Three groups of subjects were studied: 1. 9 patients with achlorhydria, 2. 12 subjects with normal gastric acid secretion, 3. 5 patients with hypersecretion. Enzyme activity was measured by determination of ammonia liberated from the substrate according to the Chaney and Marbach method. In patients with hypersecretion the ADA activity was lower than in those with achlorhydria (p < 0.001) and normal acid secretion (p < 0.02). A significant negative correlation between ADA activity in the duodenal bulb mucosa and basal and maximal gastric acid outputs was found. The present study seems to indicate a possible relationship between gastric acid secretion and duodenal ADA activity.

3

Secretion of pancreatic juice studied with cannulation of pancreatic duct in the pig

88%

Winnicki T., Brzeski W., Wasowski R.

Polish Journal of Veterinary Sciences

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1998

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tom 01

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nr 2

The following paper deals with the results of a long-lasting cannulation of the porcine pancreas performed using new methods. One method is based on a direct cannulation of the pancreatic duct after its incision. The other (designed by the authors) consists of a cannulation of the pancreas through the minor duodenal papilla. During the 14-day experimental period the efficiency of both methods was evaluated based upon certain indexes of the pancreatic juice secretions, including the level and daily dynamics of the secretions, as well as individual differentiations within two experimental groups. The method of cannulation through the minor duodenal papilla gave better results and a higher efficiency of the secretion, thus it was found to be more advantageous than the cannulation of the pancreatic duct after its incision.

4

Secretion of cytokines by testicular and peritoneal macrophages non-stimulated or stimulated with LPS

88%

Sawinski P., Klein P., Lukaszyk A., Balcerek M., Ludwiczak H.

Folia Histochemica et Cytobiologica. Supplement

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1997

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tom 35

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nr 2

5

Influence of opioids on LH secretion in gilts during the estrous cycle

88%

Okrasa S., Kalamarz H., Tilton J. E., Ziecik A. J.

Journal of Physiology and Pharmacology. Supplement

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1992

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tom 43

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nr 1

6

Gastric mucin secretion in response to beta-adrenergic G protein-coupled receptor activation is mediated by SRC kinase-dependent epidermal growth factor receptor transactivation

88%

Slomiany B. L., Slomiany A.

Journal of Physiology and Pharmacology

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2005

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tom 56

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nr 2

In many systems, the integration of converging regulatory signals that relay on G protein-coupled receptor (GPCR) activation into functional cellular pathways requires the involvement of receptor tyrosine kinase. In this report, we provide evidence that activation of GPCR by ß-adrenergic agonist leading to stimulation in gastric mucin secretion requires epidermal growth factor receptor (EGFR) participation. Using [3H]glucosamine-labeled gastric mucosal cells, we show that stimulatory effect of ß-adrenergic agonist, isoproterenol, on mucin secretion was inhibited by EGFR kinase inhibitor, PD153035, as well as wortmannin, a specific inhibitor of PI3K. Both inhibitors, moreover, blunted the mucin secretory responses to ß-adrenergic agonist-generated second messenger, cAMP as well as adenylate cyclase activator, forskolin. The gastric mucin secretory responses to isoproterenol, furthermore, were inhibited by PP2, a selective inhibitor of tyrosine kinase Src responsible for ligand-independent EGFR autophosphorylation, but not by ERK inhibitor, PD98059. The inhibition of ERK, moreover, did not cause attenuation in mucin secretion in response to cAMP and forskolin. The findings underline the role of EGFR as a convergence point in gastric mucin secretion triggered by ß-adrenergic GPCR activation, and demonstrate the requirement for Src kinase in EGFR transactivation.

7

Co-transfection of EYFP-GH and ECFP-rab3B in an experimental pituitary GH3 cell: a role of rab3B in secretion of GH through porosome

75%

Matsuno A., Itoh J., Mizutani A., Takekoshi S., Osmura R. Y., Okinaga H., Ide F., Miyawaki S., Uno T., Asano S., Tanaka J., Nakaguchi H., Sasaki M., Murakami M.

Folia Histochemica et Cytobiologica

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2008

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tom 46

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nr 4

419-421

8

Penicillin G secretion by industrial mycelia of Penicillium chrysogenum

75%

Kurzatkowski W., Gebska-Kuczerowska A., Oliwa-Dobieszewska K., Gaber A., Kuczerowski M.

Postępy Mikrobiologii

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2016

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tom 55

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nr 4

9

Effect of a prostacyclin analogue on the vasopressin and oxytocin secretion in vitro

75%

Bojanowska E., Guzek J. W.

Acta Physiologica Polonica

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1990

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tom 41

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nr 7

Bojanowska E. and Guzek J. W.: Iloprost (ZK 36374; a stable prostacyclin analogue) increases basal as well as potassium-evoked vasopressin and oxytocin secretion from rat neurointermediate lobes in vitro. This finding suggests a possible regulatory role of endogenous prostacyclin in the release of neurohypophysial hormones.

10

Cytokine signalling-transcription factor cross-talk in T cell activation and Th1-Th2 differentiation

75%

Liberman A. C., Refojo D., Arzt E.

Archivum Immunologiae et Therapiae Experimentalis

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2003

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tom 51

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nr 6

11

ACE inhibitor and AT1 antagonist stimulate duodenal HCO3 secretion mediated by a common pathway - involvement of PG, NO and bradykinin

75%

Aihara E., Kagawa S., Hayashi M., Takeuchi K.

Journal of Physiology and Pharmacology

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2005

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tom 56

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nr 3

Recent study demonstrated that duodenal HCO3- secretion is affected by modulation of the renin-angiotensin system. We examined the effects of enalapril (angiotensin-converting enzyme (ACE) inhibitor) or losartan (angiotensin AT1 receptor antagonist) on duodenal HCO3- secretion in rats and investigated the mechanisms involved in the renin-angiotensin system-related HCO3- response. A proximal duodenal loop was perfused with saline, and HCO3- secretion was measured at pH 7.0 using a pH-stat method and by adding 2 mM HCl. Enalapril increased the HCO3- secretion in a dose-dependent manner, with a decrease in arterial blood pressure (MBP), and these effects were significantly attenuated by pretreatment with indomethacin, L-NAME and FR172357 (a selective bradykinin B2 receptor antagonist). Although losartan alone did not affect the HCO3- secretion, despite reducing MBP, the agent dose-dependently increased the HCO3- secretion in the presence of angiotensin II, and this response was totally antagonized by prior administration of FR172357, indomethacin and L-NAME. Bradykinin also dose-dependently increased the HCO3- secretion with no change in MBP, though transient, and again the effects were blocked by indomethacin, L-NAME and FR172357. Both prostaglandin (PG) E2 and the nitric oxide (NO) donor NOR-3 also increased the HCO3- secretion, the latter effect being inhibited by indomethacin. These results suggest that both an ACE inhibitor and AT1 antagonist (in the presence of angiotensin II) increase duodenal HCO3- secretion via a common pathway, involving bradykinin, NO and PGs. It is also assumed that bradykinin releases NO locally, which in turns stimulates HCO3- secretion mediated by PGs.

12

Notes on the method to investigate nectar secretion rate in flowers

75%

Jablonski B.

Journal of Apicultural Science

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2002

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tom 46

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nr 2

13

Relations between FcRI crosslinking-induced mast cell activation and adhesion to fibronectin

75%

Wyczolkowska J., Dastych J., Slusarczyk A., Kolago B.

Journal of Physiology and Pharmacology

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1994

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tom 45

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nr 4

14

Ecophysiological aspects of nectar reabsorption

75%

Stpiczynska M., Nepi M.

Acta Agrobotanica

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2006

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tom 59

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nr 1

A number of approaches, both direct and indirect, have shown that nectar is reabsorbed by numerous plant species, irrespective of the age or sex of the flower. Furthermore, reabsorption occurs regardless of whether or not the flower has been pollinated. Reabsorption helps to maintain concentration of nectar and their viscosity and thereby encourages continued visits by pollinators. Conversely, the capacity to vary concentration of nectar sugars may confer evolutionary advantage by encouraging visits by more than one kind of pollinator and this is particularly important in regions where there is a paucity of pollinators. A further important role of nectar reabsorption is the maintenance of the energy equilibrium of the plant. A number of studies have shown that nectar production involves considerable energy expenditure requiring as much as 37% of the plant's daily production of energy by photosynthesis. The increased metabolic costs incurred by the plant during nectar production and secretion can reduce its growth and reproduction during the following season. Reabsorption of nectar that has not been collected by pollinators enables the plant to conserve at least some of the energy reserved for the secretion of nectar. Sugars reabsorbed from nectar can be re-used for the development of fruit and ovules - processes which demand large quantities of sugar. Despite convincing evidence for the reabsorption of nectar, few detailed studies have addressed the transport and incorporation of reabsorbed sugars. One of the questions that remain to be answered is 'What is the cellular basis for nectar reabsorption by the nectary?'.

15

The involvement of catecholamines in the pituitary release of luteinizing hormone [LH] and prolactin [PRL] in progesterone [P4] - treated gilts

75%

Siawrys G., Przala J.

Folia Histochemica et Cytobiologica. Supplement

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1997

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tom 35

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nr 2

16

Schistosoma japonicum: the chemical nature of the secretion produced by the Mehlis' gland

75%

Moczon T., Swiderski Z.

Wiadomości Parazytologiczne

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1998

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tom 44

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nr 3

17

Role of nitric oxide in gastroduodenal alkaline secretion

75%

Bilski J., Konturek S. J.

Journal of Physiology and Pharmacology

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1994

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tom 45

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nr 4

18

Aminergic control of vasopressin secretion in the conscious rat

75%

Wells T., Forsling M. L.

Journal of Physiology and Pharmacology

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1992

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tom 43

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nr 1

The influence of aminergic pathways on basal and stimulated vasopressin (AVP) release was studied in conscious rats, the stimulus for hormone release being an intracerebroventricular (ICV) injection of 5 µl 0.85M sodium chloride. The animals were treated with either phenoxybenzamine, propranolol or haloperidol prior to administration of the central hypertonic stimulus. Phenoxybenzamine elevated basal plasma vasopressin concentrations, while propranolol and haloperidol had no effect. The secretion of АVР in response to the hypertonic stimulus was potentiated by phenoxybenzamine and haloperidol, but the effect of propranolol was equivocal. The antagonists had no effect on basal arterial pressure at the time of hypertonic saline administration or the pressor response to ICV sodium chloride.

19

IP6 modulates interleukin-8 secretion in colon epithelial cells by influencing various signalling pathways

75%

Weglarz L., Wawszczyk J., Orchel A., Dzierzewicz Z.

Cellular and Molecular Biology Letters

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2005

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tom 10

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nr Suppl.2

20

The influence of indomethacin on the ACTH secretion induced by central stimulation of adrenergic receptors

75%

Glod R., Gadek-Michalska A., Bugajski J.

Journal of Physiology and Pharmacology

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2000

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tom 51

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nr 2

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