Wyniki wyszukiwania - AGRO

1

Superoxide dismutases - the first line of defense against ROS

100%

Stanko A., Ludwikow A., Babula D., Sadowski J.

Polish Journal of Natural Sciences. Supplement

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2003

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tom 01

2

Oxidative stress as a factor limiting muscle performance

100%

Antosiewicz J.

Polish Journal of Environmental Studies

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1997

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tom 06

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nr 2

3

CAM induction and oxidative stress in Mesembryanthemum crystallinum

100%

Miszalski Z., Slesak I., Haag-Kerwer A., Surowka E., Libik M., Gawronska K., Niewiadomska E.

Polish Journal of Natural Sciences. Supplement

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2003

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tom 01

4

Coenzyme Q: a bioenergetic and antioxidant compound

100%

Lenaz G.

Polish Journal of Environmental Studies

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1999

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tom 08

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nr 5

5

Elevated CO2 ameliorated oxidative stress induced by elevated O3 in Quercus mongolica

88%

Yan K., Chen W., Zhang G., Xu S., Liu Z., He X., Wang L.

Acta Physiologiae Plantarum

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2010

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tom 32

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nr 2

Using open top chambers, the effects of elevated O₃ (80 nmol mol⁻¹) and elevated CO₂ (700 µmol mol⁻¹), alone and in combination, were studied on young trees of Quercus mongolica. The results showed that elevated O₃ increased malondialdehyde content and decreased photosynthetic rate after 45 days of exposure, and prolonged exposure (105 days) induced significant increase in electrolyte leakage and reduction of chlorophyll content. All these changes were alleviated by elevated CO₂, indicating that oxidative stress on cell membrane and photosynthesis was ameliorated. After 45 days of exposure, elevated O₃ stimulated activities of superoxide dismutase (SOD, EC 1.15.1.1) and ascorbate peroxidase (APX, EC 1.11.1.11), but the stimulation was dampened under elevated CO₂ exposure. Furthermore, ascorbate (AsA) and total phenolics contents were not higher in the combined gas treatment than those in elevated O₃ treatment. It indicates that the protective effect of elevated CO₂ against O₃ stress was achieved hardly by enhancing ROS scavenging ability after 45 days of exposure. After 105 days of exposure, elevated O₃ significantly decreased activities of SOD, catalase (CAT, EC 1.11.1.6) and APX and AsA content. Elevated CO₂ suppressed the O₃-induced decrease, which could ameliorate the oxidative stress in some extent. In addition, elevated CO₂ increased total phenolics content in the leaves both under ambient O₃ and elevated O₃ exposure, which might contribute to the protection against O₃-induced oxidative stress as well.

6

Harmful effect of high-dietary fructose and magnesium deficiency: role of inflammation and oxidative stress

88%

Rayssiguier Y., Gueux E., Nowacki W., Rock E., Mazur A.

Journal of Elementology

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2005

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tom 10

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nr 4 Suppl.1

85

7

4-Hydroxynonenal and oxidative stress in several organelles and its damaging effects on cell functions

88%

Gallo G., Sprovieri P., Martino G.

Journal of Physiology and Pharmacology

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2020

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tom 71

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nr 1

8

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Potential of karamunting (Rhodomyrtus tomentosa) fraction against kidney damage in diabetic rats

88%

Saleh M. I., Hidayat R., Febriyanto G., Parisa N.

Herba Polonica

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2021

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tom 67

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nr 2

9

Magnesium deficiency [MgD] enhances cardiac inflammation due to nucleoside reverse transcriptase inhibitor [NRTI] treatment

88%

Chmielinska J. J., Tejero-Taldo M. I., Mak I. T., Weglicki W. B.

Journal of Elementology

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2005

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tom 10

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nr 4 Suppl.1

25

10

Fever induced oxidative stress: the effect on thyroid status and the 5'-monodeiodinase activity, protective role of selenium and vitamin E

88%

Brzezinska-Slebodzinska E.

Journal of Physiology and Pharmacology

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2001

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tom 52

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nr 2

11

Antioxidant activity: what do we measure?

88%

Bartosz G., Bartosz M.

Acta Biochimica Polonica

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1999

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tom 46

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nr 1

Inhibition of oxidation of 2,2'-azinobis(3-ethylbenzothiazoline-6-sulfonic acid) (ABTS) by free radicals generated by decomposition of 2,2'-azobis(2-amidopropane) (ABAP) by antioxidants and biological material was studied. A correlation was found between the ability of various substances to delay the onset of ABTS oxidation and their rapid reduction of the ABTS+* cation radical, and between the ability to reduce the maximal rate of ABTS oxidation and slow reduction of ABTS+*. The length of the lag period of ABTS oxidation was found to be independent of ABTS concentration. Similar decrease of peroxynitrite-induced ABTS+* formation by antioxidants was observed when the antioxidants were added before and after peroxynitrite. All these findings indicate that the main effect of antioxidants in this system is reduction of ABTS+* and not prevention of its formation. Reduction of oxidation products rather than inhibition of their formation may be the predominant mode of action of antioxidants in various assays of antioxidant activity.

12

Activities of selected antioxidant enzymes in blood as dependent on training

88%

Hubner-Wozniak E., Panczenko-Kresowska B.

Polish Journal of Environmental Studies

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1997

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tom 06

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nr 2

13

The correlation between oxidative stress and leaf senescence during plant development

88%

Zimmermann P., Zentgraf U.

Cellular and Molecular Biology Letters

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2005

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tom 10

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nr 3

14

Oxidative stress in flax seedling under low water potential - protective role of exogenous polyamines

88%

Niedzwiedz-Siegien I., Tokarewicz A.

Polish Journal of Natural Sciences. Supplement

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2003

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tom 01

15

The preventive role of Spirulina platensis (Arthrospira platensis) in immune and oxidative insults in a stress-induced rat model

76%

Seyidoglu N., Koseli E., Gurbanli R., Aydin C.

Journal of Veterinary Research

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2021

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tom 65

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nr 2

16

Redox properties and prooxidant cytotoxicity of a neuroleptic agent 6,7-dinitrodihydroquinoxaline-2,3-dione (DNQX)

76%

Sarlauskas J., Nemeikaite-Ceniene A., Miseviciene L., Krikstopaitis K., Anusevicius Z., Cenas N.

Acta Biochimica Polonica

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2013

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tom 60

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nr 2

In order to characterize the possible mechanism(s) of cytotoxicity of a neuroleptic agent 6,7-dinitrodihydroquinoxaline-2,3-dione (DNQX) we examined the redox properties of DNQX, and its mononitro- (NQX) and denitro- (QX) derivatives. The irreversible electrochemical reduction of the nitro groups of DNQX was characterized by the reduction peak potentials (Ep,7) of -0.43 V and -0.72 V vs. Ag/AgCl at pH 7.0, whereas NQX was reduced at Ep,7 = -0.67 V. The reactivities of DNQX and NQX towards the single-electron transferring enzymes NADPH:cytochrome P-450 reductase and NADPH:adrenodoxin reductase/adrenodoxin complex were similar to those of model nitrobenzenes with the single-electron reduction potential (E17) values of -0.29 V - -0.42 V. DNQX and NQX also acted as substrates for two-electron transferring mammalian NAD(P)H:quinone oxidoreductase (DT-diaphorase). The cytotoxicity of DNQX in bovine leukemia virus-transformed lamb kidney fibroblasts (line FLK) was prevented by antioxidants and an inhibitor of NQO1, dicoumarol, and was enhanced by the prooxidant alkylating agent 1,3-bis(2-chloromethyl)-1-nitrosourea. A comparison with model nitrobenzene compounds shows that the cytotoxicity of DNQX and NQX reasonably agrees with the ease of their electrochemical reduction, and/or their reactivities towards the used enzymatic single-electron reducing systems. Thus, our data imply that the cytotoxicity of DNQX in FLK cells is exerted mainly through oxidative stress.

17

Cytoprotective action of 17beta-oestradiol against iron-induced hepatic oxidative stress in vitro

76%

Wojcik M., Kosior-Korzecka U., Bobowiec R.

Bulletin of the Veterinary Institute in Puławy

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2010

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tom 54

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nr 2

259-263

The objective of this study was to analyse the response of hepatocytes on various concentrations of 17ß-oestradiol (17ß-E) under iron-induced oxidative stress in vitro. Isolated by in situ collagenase perfusion hepatocytes were cultured in DMEM/HAMS-12 (v/v) medium without any additional agents (control), with Fe³⁺ alone, and with Fe³⁺ aild 0.2%, 0.02%, and 0.002% solution of 17ß-E (17ß-EI, 17ß-EII, and 17ß-EIII, respectively). After 24, 48, and 72 h, medium malonylodialdehyde (MDA), haptoglobin (Hpt) concentration and proliferative activity were determined. In comparison to control samples, and samples collected at 24 and 72 h, hepatocytes exposition to Fe³⁺, caused a significant increase in MDA (0.056 ±0.011 nM/mL) only after 48 h of incubation. Each of 17ß-E concentrations resulted in a decrease in MDA in samples obtained after 24 and 48 h. In comparison to the first 24 h, Fe³⁺ alone and together with 17ß-EI, 17ß-EII, and 17ß-EIII caused a significant augmentation of Hpt level in 48 h and 72 h of the experiment. Each of the 17ß-E concentrations added to the culture medium resulted in inhibition of hepatic proliferative activity, especially in the 72 h of cell culture.

18

Identification of Triticum durum genotypes showing increased tolerance to oxidative stress

76%

Lesniowska-Nowak J., Sozoniuk M., Kawecka M., Maga K., Mrozek M.

Agronomy Science

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2020

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tom 75

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nr 1

19

The effect of water deficit and UV-B radiation on oxidative stress markers and growth parameters in barley seedlings

76%

Paczkowska M., Bandurska H.

Acta Physiologiae Plantarum

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2007

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tom 29

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nr 1 Suppl.

20

Sperm mitochondrial dysfunction and oxidative stress as possible reasons for isolated asthenozoospermia

76%

Nowicka-Bauer K., Lepczynski A., Ozgo M., Kamieniczna M., Fraczek M., Stanski L., Olszewska M., Malcher A., Skrzypczak W., Kurpisz M. K.

Journal of Physiology and Pharmacology

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2018

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tom 69

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nr 3

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