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1

Oxidative metabolism of peripheral blood neutrophils in chronic renal failure on conservative and dialysis treatment

100%
Luciak M.
Archivum Immunologiae et Therapiae Experimentalis
|
1992
|
tom 40
|
nr 2
2

Influence of haemodialysis on magnesium metabolism in patient with chronic renal failure

88%
Walasek L., Rzeszotarski J., Marszalek A.
Journal of Elementology
|
2005
|
tom 10
|
nr 4 Suppl.1
107-108
3

Influence of disturbed calcium-phosphate metabolism on the cardiovascular system in patients with chronic renal failure

88%
Rzeszotarski J., Walasek L., Marszalek A.
Journal of Elementology
|
2005
|
tom 10
|
nr 4 Suppl.1
91-92
4

Preliminary evaluation of gastric endocrine cells in rats with experimental uremia

88%
Kasacka I., Azzadin A., Sawicki B., Dadan J., Malla H., Buczko W.
Folia Histochemica et Cytobiologica
|
2001
|
tom 39
|
nr 2
5

Suppressor of cytokine signaling and accelerated atherosclerosis in kidney disease

75%
Wesoly J., Sikorski K., Lee C.-K., Bluyssen A. R.
Acta Biochimica Polonica
|
2010
|
tom 57
|
nr 3
The prevalence of cardiovascular disease in patients with renal failure is extremely high and accounts for a large part of the morbidity and mortality. Inflammation participates importantly in host defense against infectious agents and injury, but also contributes to the pathophysiology of many diseases, including cardiovascular atherosclerosis, which is a main problem in patients with renal failure. Recruitment of blood leukocytes to the injured vascular endothelium characterizes the initiation and progression of atherosclerosis and involves many inflammatory mediators, modulated by cells of both innate and adaptive immunity. Excessive inflammatory and immune responses, communicated by these different cell types, are driven by inflammatory cytokines that promote associated tissue damage if cytokine signaling pathways remain unregulated. Thus, pathways capable of suppressing proinflammatory cytokine signaling hold the potential to limit life-threatening cardiovascular events caused by atherogenesis. Suppressor of cytokine signaling (SOCS) are a family of intracellular proteins, several of which have emerged as key physiological regulators of cytokine-mediated homeostasis, including innate and adaptive immunity. Accumulating evidence supports the idea that dysregulation of cytokine signaling by differential SOCS expression is involved in the pathogenesis of various inflammatory, and immunological diseases, including atherosclerosis. Based on recent observations, in which SOCS expression levels are profoundly altered in kidney disease, we discuss the possibilities of SOCS as new intracellular markers of inflammation as well as their potential atherogenic properties in renal failure related cardiovascular disease.
6

Decrease of PKCalpha translocation to the erythrocyte membrane in patients with chronic renal failure [CRF]

75%
Kaczmarek J., Komorowska G., Rybczynska M.
Cellular and Molecular Biology Letters
|
1998
|
tom 03
|
nr 2
7

Contemporary views on the role of nutrition in chronic renal failure - a review

75%
Pietrzak I., Formanowicz D., Grzymislawski M., Wojciechowski J.
Polish Journal of Food and Nutrition Sciences
|
2006
|
tom 15/56
|
nr 2
The pathological process in the kidneys in the course of chronic renal failure (CRF) leads to multiple metabolic disturbances in the whole body. Disturbances of protein, carbohydrate, fat, electrolyte and calcium-phosphate balance are the main reasons for protein-energy malnutrition in patients. The use of a proper, individually adapted diet may slow down the progression of the disease and reduce the intensity of the symptoms.
8

Increased whole blood chemiluminescence in patients with chronic renal failure independent of hemodialysis treatment

75%
Rysz J., Potargowicz E., Banach M., Luczynska M., Stolarek R., Bialasiewicz P., Kasielski M., Cialkowska-Rysz A., Nowak D.
Archivum Immunologiae et Therapiae Experimentalis
|
2006
|
tom 54
|
nr 5
9

Preliminary evaluation of thyroid C cells in rats with chronic renal failure

75%
Azzadin A., Kasacka I., Sawicki B., Dadan J., Malla H., Buczko W.
Folia Histochemica et Cytobiologica
|
2001
|
tom 39
|
nr 2
10

Analysis of calcium, phosphorus and magnesium intake in diet of patients with chronic renal failure treated with haemodialysis

63%
Rzeszotarski J., Walasek L., Ligocki P.
Journal of Elementology
|
2005
|
tom 10
|
nr 4 Suppl.1
90-91
11
Content available remote

What influences the level of oxidative stress as measured by 8-hydroxy-2'-deoxyguanosine in patients on hemodialysis?

63%
Mastalerz-Migas A., Steciwko A., Pokorski M., Pirogowicz I., Drobnik J., Bunio A., Muszynska A., Jasinska A.
Journal of Physiology and Pharmacology. Supplement
|
2006
|
tom 57
|
nr 4
199-205
Oxidative stress is at play in the progression of chronic renal failure (CRF) and in the genesis of atherosclerosis. The aim of the present study was to evaluate the factors that might influence the oxidative-antioxidative balance in patients on hemodialysis. The study group consisted of 71 hemodialysis patients due to CRF. Sixteen healthy subjects constituted a control group. The levels of 8-hydroxy-2’-deoxyguanosine (8-OHdG), C-reactive protein (CRP), and the blood lipid profile were measured in both groups. The results showed significantly higher mean levels of both 8-OHdG and CRP in the hemodialysis patients compared with that in the control subjects. The highest level of 8-OHdG was found in the subgroups of the patients with CRF primarily caused by diabetes (16.4 ng/ml) and with hypertensive nephropathy (15.8 ng/ml). More than a 2.5-fold higher level of 8-OHdG in the hemodialysis patients compared with the control subjects points to the presence of intensive oxidative stress in the patients.
12

TGF-beta1 immunohistochemistry and promoter methylation in chronic renal failure rats treated with uremic clearance granules

63%
Miao X.-H., Wang C.-G., Hu B.-Q., Li A., Chen C.-B., Song W.-Q.
Folia Histochemica et Cytobiologica
|
2010
|
tom 48
|
nr 2
284-291
13

Assessment of the concentrations of total magnesium, ionized magnesium and magnesium in red blood cells in patients with chronic renal failure treated with haemodialysis

63%
Walasek L., Rzeszotarski J., Marszalek A.
Journal of Elementology
|
2005
|
tom 10
|
nr 4 Suppl.1
109
14

Expression of vascular endothelial growth factor and transforming growth factor alpha in rat testis during chronic renal failure

63%
Seval-Celik Y., Akkoyunlu G., Kocamaz E., Koksal I. T., Ozer S., Baykara M., Demir R.
Folia Histochemica et Cytobiologica
|
2014
|
tom 52
|
nr 4
15

Thyroid C cells in male and female rats with chronic renal failure

63%
Kasacka I., Azzadin A., Malla H.
Folia Histochemica et Cytobiologica
|
2002
|
tom 40
|
nr 2
16

Red blood cell damage in chronic renal failure

63%
Gwozdzinski K.
Cellular and Molecular Biology Letters
|
2002
|
tom 07
|
nr Suppl.
17

Changes in red blood cell membrane structure in patients with chronic renal failure

63%
Gwozdzinski K., Janicka M., Brzeszczynska J., Luciak M.
Acta Biochimica Polonica
|
1997
|
tom 44
|
nr 1
The properties of red blood cell membranes in patients with chronic renal failure were investigated using electron paramagnetic resonance spectroscopy. Using spin traps, 5,5-dimethylpirroline-l-oxide and N-tert-butyl-a-phenylni- trone, we found generation of hydroxyl radicals in the blood of patients with chronic renal failure after 20 min of regular hemodialysis. The physical state of membrane proteins and membrane osmotic fragility and reductive properties of red blood cells were studied. The increase in the relative correlation time of 4-(2-iodoacetamido)-2,2,6,6-tetramethylpiperidine-l-oxyl in­dicates the immobilization of membrane protein molecules in erythrocytes of chronic renal failure patients. The decrease in membrane protein mobility was observed in whole blood incubated with tert-butylhydroperoxide, regardless of the presence of iron. We found that the addition of ferrous ions did not aggravate profound changes in membrane proteins induced with tert-butylhy­droperoxide. We also demonstrated higher osmotic fragility of erythrocytes in the patients with renal failure as compared to normal subjects.
18

Kardionefrologia - nowy punkt widzenia w medycynie weterynaryjnej

51%
Szczepankiewicz B., Paslawski R., Paslawska U.
Weterynaria w Praktyce
|
2020
|
tom 17
|
nr 03
19
Content available remote

Comparison of a statin vs. hypolipemic diet on the oxidant status in hemodialyzed patients with chronic renal failure

51%
Mastalerz-Migas A., Reksa D., Pokorski M., Steciwko A., Muszynska A., Bunio A., Drobnik J., Pokorna-Kalwak D.
Journal of Physiology and Pharmacology. Supplement
|
2007
|
tom 58
|
nr 5
20

Quantitative characteristics of calcitonin-producing cells in the thyroid and lungs of uremic rats

51%
Kasacka I.
Folia Histochemica et Cytobiologica
|
2008
|
tom 46
|
nr 4
525-530
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