Wyniki wyszukiwania

1

The corticoclaustral connections in the rat studied by means of the fluorescent retrograde axonal transport method

100%

Kowianski P., Dziewiatkowski J., Berdel B., Lipowska M., Morys J.

Folia Morphologica

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1998

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tom 57

|

nr 2

2

Role of the subplate neurons in specification of neocortical areas

100%

Wozniak W.

Folia Morphologica

|

1995

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tom 54

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nr 3

3

Electrical activity of the cerebral cortex in men having high or low output alpha-frequencies while performing usual manual movements in response to sensory signals

86%

Morenko A., Tsos A., Kocan I.

Człowiek i Zdrowie

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2013

|

tom 07

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nr 3

A test group consisting of 104 healthy men from the ages of 19 to 21 was divided into two groups according to the magnitude of their individual α- frequency (ІАF) median―groups with high (n = 53, IA ≥ 10,04 Hz) and low (n = 51, IAF ≤ 10,03 Hz) levels of ІАF. Changes in power and coherence of the EEG oscillations during the usual manual movements as well as intergroup differences were evaluated. The higher level of the background tone of the cortex activation in men with a high IAF (individual alpha-frequency) was found as correlative with a signi􀏐icant selection of the relevant sensory information and a nonspecific input afferentation and its weakening under the performance of habitual manual movements. A functional state of the cortex is relatively lower in men with a low IAF and compensated by the tension of brain processes associated with a low level of the selective attention and the increase of the reticular influences and their significance during the finger movements. Men with a high IAF are characterized by the greater locality and asymmetry of processes of the cortical excitation under the dominant role of the left hemisphere. Activation changes are more diffuse in nature in subjects having a low frequency

4

Development of the human cerebral cortex: cortical patterning and migration

86%

Wozniak W.

Acta Biologica Cracoviensia. Series Botanica. Supplement

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2008

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tom 50

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nr 1

5

Cortical branches of the middle cerebral artery in fallow deer (Dama dama)

86%

Skoczylas B., Brudnicki W., Nowicki W., Jablonski R., Kirkillo-Stacewicz K., Wach J.

Electronic Journal of Polish Agricultural Universities. Series Veterinary Medicine

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2011

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tom 14

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nr 4

6

The effects of allogeneic bone marrow stromal cells on glial scar formation in the injured cerebral cortex of rats

86%

Opydo-Chanek M.

Acta Biologica Cracoviensia. Series Zoologia

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2009

|

tom 51

23-31

7

Glial scar instability after brain injury

86%

Frontczak-Baniewicz M., Walski M.

Journal of Physiology and Pharmacology. Supplement

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2006

|

tom 57

|

nr 4

97-102

Glial scar is formed following surgical damage to the cerebral cortex. In the present study we examined the ultrastructural status of the cerebral cortex 14 to 180 days following surgical damage to cerebral parenchyma. The results showed a contribution of astrocytes, but also mesodermal cells, to the process of scar formation. Furthermore, our study showed that the process initiated by trauma did not terminate with the formation of a glial scar. Late phases of repair following tissue damage were associated with lytic processes and a disassembly of the cerebral parenchyma. These findings indicate a changing and unstable nature of the glial scar and its components.

8

Developmental changes in nitric oxide synthase protein expression in the rat striatum and cerebral cortex

86%

Labuda C., Litwinowicz B., Kowianski P., Spodnik J. H., Luczynska A., Morys J.

Folia Morphologica

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2003

|

tom 62

|

nr 3

171-174

We examined the expression of brain nitric oxide synthase (bNOS) in two developing rat brain structures, the striatum and the cerebral cortex. For this purpose, we quantified the relative protein concentration level using the Western blotting method and densitometric scanning. 32 Wistar rats, divided according to survival period (P0-P120-postnatal days) were used in this study. Our results demonstrate that bNOS expression rises in these structures during the first week of postnatal life, reaching a maximum in the striatum on the 10th day and in the cerebral cortex on the 7th day of postnatal life. After the period of increase the expression declines and after the 14th day a stabilisation of bone protein concentration is observed, both in the striatum and the cerebral cortex. These changes in bone protein expression might be related to the important role of nitric oxide in the developing rat brain, especially in synaptogenesis, apoptosis and neurotransmission.

9

Developmental expression of SNAP-25 protein in the rat striatum and cerebral cortex

86%

Sidor-Kaczmarek J., Labuda C., Litwinowicz B., Spodnik J. H., Kowianski P., Dziewiatkowski J., Morys J.

Folia Morphologica

|

2004

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tom 63

|

nr 3

The developmental changes of 25-kDa synaptosomal-associated protein (SNAP-25) expression in the rat striatum and cerebral cortex were examined using Western-blotting and densitometric scanning of immunoblots. Analysis of the striatum extracts from postnatal day 0 (P0) to postnatal day 120 (P120) demonstrated that SNAP-25 is poorly expressed until P14. From this point the expression level gradually increases to reach a maximum on P60 and then decreases. The pattern of SNAP-25 expression in the rat cerebral cortex is different. Two peaks are observed, the first on P10 and the second on P60, after which the expression level decreases. These results appear to confirm the role of SNAP-25 protein in axon outgrowth and synaptogenesis in the nervous system.

10

Development of the archipallial and paleopallial structures in the human embryonic brain

86%

Rapalska K., Szwabe M., Czarniawska-Grzesinska M., Wozniak W.

Acta Biologica Cracoviensia. Series Botanica. Supplement

|

2000

|

tom 42

|

nr 1

11

Effects of excess vitamin B6 intake on cerebral cortex neurons in rat: an ultrastructural study

86%

Demir R., Acar G., Tanriover G., Seval Y., Kayisli U. A., Agar A.

Folia Histochemica et Cytobiologica

|

2005

|

tom 43

|

nr 3

12

Pituitary adenylate cyclase-activating polypeptide [PACAP] stimulates phosphoinositide metabolism and translocation of protein kinase C in chick cerebral cortex

86%

Nowak J. Z., Dejda A., Pigulowska A., Kuba K., Zawilska J. B.

Acta Neurobiologiae Experimentalis

|

2001

|

tom 61

|

nr 4

13

The phosphorylation of synaptosomal membrane proteins from cerebral cortex is modulated by neuroactive steroids

86%

Rebas E., Lachowicz L.

Biophysics of Membrane Transport

|

1994

|

tom 12

|

nr 2

14

A quantitative immunocytochemical study of blood-brain barrier to endogenous albumin in cerebral cortex and hippocampus of senescence-accelerated mice [SAM]

86%

Vorbrodt A. W., Dobrogowska D. H., Ueno M., Tarnawski M.

Folia Histochemica et Cytobiologica

|

1995

|

tom 33

|

nr 4

15

Cerebral and gastric histamine system is altered after portocaval shunt

86%

Fogel W. A., Michelsen K. A., Panula P., Sasiak K., Andrzejewski W.

Journal of Physiology and Pharmacology

|

2001

|

tom 52

|

nr 4,1

Biochemical parameters of the histamine (HA) system were examined in both rat brain and stomach, after portocaval anastomosis (PCA). These tissues become rich in histamine after PCA. Immunocytochemistry was used for brain histamine localisation. In addition to increased HA concentrations, monoamine oxidase B activity increased in both tissues. In hypothalamus HA was 15 fold; in cerebral cortex and in stomach mucosa 2.8 and 2.5 fold of the corresponding controls, respectively. MAO B activity was increased by approximately 50% in brain and 100% in stomach. A significant, uneven increase in tele-methylhistamine concentration was only found in the brain. In stomach mucosa higher histidine decarboxylase activity was found. PCA and sham rats treated with an irreversible inhibitor of MAO B, FA-73, 0.5 mg/kg i.p., showed 24 h later greatly reduced MAO activity and doubled t-MeHA concentration in brain structures. The treatment had no effect on gastric mucosal t-MeHA concentration and on urinary excretion of the t-MeHA metabolite, N-tele-methylimidazoleacetic acid. The HA rise in the stomach of PCA rats is associated with proliferation of histamine producing and storing cells (ECL cells) as demonstrated by others. However, in the brain we saw no indication for increased number of relevant cells either mast cells or neurons and our immunocytochemical findings suggest that in PCA rat brain, histamine deposits are located exclusively in neurons. The data indicate that the adaptative mechanisms to excessive histamine formation are tissue specific.

16

Ultrastructural organization of the visual zone in the claustrum of the cat

86%

Kubasik-Juraniec J., Dziewiatkowski J., Morys J., Narkiewicz O.

Folia Morphologica

|

1998

|

tom 57

|

nr 4

17

New vessel formation after surgical brain injury in the rat's cerebral cortex. II. Formation of the blood vessels distal to the surgical injury

86%

Walski M., Frontczak-Baniewicz M.

Acta Neurobiologiae Experimentalis

|

2003

|

tom 63

|

nr 2

18

The structure of the primordial plexiform layer in staged human embryos

86%

Rapalska K., Wozniak W.

Folia Morphologica

|

1998

|

tom 57

|

nr 1

19

Traumatic brain injury results in a concomitant increase in neocortical expression of vasopressin and its V1A receptor

72%

Pascale C. L., Szmydynger-Chodobska J., Sarri J. E., Chodobski A.

Journal of Physiology and Pharmacology. Supplement

|

2006

|

tom 57

|

nr 11

161-167

Arginine vasopressin (AVP) has been shown to promote the disruption of the blood-brain barrier (BBB) and the formation of edema in various animal models of brain injury. However, the source(s) of this AVP have not been identified. Since the cerebral cortex was considerably affected in some of these brain injury models, we sought to determine if AVP was produced in the cerebral cortex, and, if so, whether or not this cortical AVP expression was up regulated after injury. In the present study, a controlled cortical impact model of traumatic brain injury (TBI) in rats was used, and the temporal changes in expression of AVP and its V1a receptor were analyzed by real-time reverse-transcriptase polymerase chain reaction. The expression of AVP and its V1a receptor in the ipsilateral cortex adjacent to the lesion area was significantly up regulated between 4 h and 1day post-TBI. The maximum increase in mRNA for AVP (4.3-fold) and its receptor (2.6-fold) in the ipsilateral vs. contralateral cortex was observed at 6 h post-TBI. Compared to sham-injured rats, no statistically significant changes in expression of AVP or its receptor were found in the contralateral cortex. These results suggest that the cerebral cortex is an important source of AVP in the injured brain, and the parallel increase in the expression of AVP and its cognate receptor may act to augment the actions of AVP related to promoting the disruption of the BBB and the formation of post-traumatic edema.

20

Tangential migration of neocortical neurons in early human foetuses

72%

Rapalska K., Szwabe M.

Folia Morphologica

|

2003

|

tom 62

|

nr 3

223-225

The aim of the present study is to describe the tangential migration of cortical neurons in early human foetuses aged 9–11 weeks. Histological sections showed that in this early period of development most of the neurons migrate along radial glia, but there is also tangential migration, especially from the lateral eminence.

Ograniczanie wyników

The corticoclaustral connections in the rat studied by means of the fluorescent retrograde axonal transport method

Role of the subplate neurons in specification of neocortical areas

Electrical activity of the cerebral cortex in men having high or low output alpha-frequencies while performing usual manual movements in response to sensory signals

Development of the human cerebral cortex: cortical patterning and migration

Cortical branches of the middle cerebral artery in fallow deer (Dama dama)

The effects of allogeneic bone marrow stromal cells on glial scar formation in the injured cerebral cortex of rats

Glial scar instability after brain injury

Developmental changes in nitric oxide synthase protein expression in the rat striatum and cerebral cortex

Developmental expression of SNAP-25 protein in the rat striatum and cerebral cortex