Wyniki wyszukiwania - AGRO

1

mTOR pathway novel modulator of astrocyte activity

100%

Latacz A., Russell J. A., Oclon E., Zubel-Lojek J., Pierzchala-Koziec K.

Folia Biologica

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2015

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tom 63

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nr 2

2

The lumbosacral plexus in coypu

100%

Langenfeld M.

Folia Morphologica

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1987

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tom 46

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nr 3-4

3

Inflammation in Alzheimer’s disease and molecular genetics: recent update

100%

Zhang Z.-G., Li Y., Ng C. T., Song Y.-Q.

Archivum Immunologiae et Therapiae Experimentalis

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2015

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tom 63

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nr 5

4

Neuropeptide Y - a neuromodulatory link between nutrition and reproduction at the central nervous system level

100%

Wojcik-Gladysz A., Polkowska J.

Reproductive Biology

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2006

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tom 06

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nr Suppl.2

5

Effect of diazepam on adrenal cortex stress reaction

100%

Beszczynska B., Siejka A.

Acta Biologica Cracoviensia. Series Zoologia

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2003

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tom 45

6

The role of dendritic cells in neurodegenerative diseases

100%

Iribarren P., Cui Y. H., Le Y., Wang J. M.

Archivum Immunologiae et Therapiae Experimentalis

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2002

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tom 50

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nr 3

7

MIB-I as a proliferative activity marker of the multiform glioblastomas

100%

Lebelt A., Lemancewicz D., Szkudlarek M., Zimnoch L., Lewko J., Lebkowski W., Dzieciol J.

Folia Histochemica et Cytobiologica. Supplement

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2001

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tom 39

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nr 1

8

RNA interference: a potential novel therapeutic combating HIV-1 in the central nervous system

100%

Pomerantz R. J.

Archivum Immunologiae et Therapiae Experimentalis

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2004

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tom 52

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nr 6

9

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Functional status of the central nervous system in girls aged 16-18 years old

86%

Bosenko A., Bobro O., Topchii M., Kholodov S.

Health Problems of Civilization

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2019

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tom 13

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nr 4

Background. The objective of the study was to explore the dynamics of the functional state of the central nervous system (CNS) and neurophysiological processes of girls 16-18 years old in the context of their transition to a new level of social organization. Material and methods. The functional state of the CNS was studied in girls 16-18 years old in a state of relative muscular rest through observation of visible motive reactions, Loskutova’s method, and in the fifth minute of renewal after the physical loading with variable power. Results. We found that 17 year-old girls had the best functional CNS capabilities. The reduced adaptive capacity of the CNS in 16 year-old girls is due to their necessity for adaptive reorganization of regulatory mechanisms to new social conditions: examinations and university entry. After physical activity, 16 and 18 year-old girls registered stress reactions, indicating decreased CNS adaptation. Conclusions. 17 year-old girls were observed at rest to have better CNS functionality compared to the other age groups. Analysis of the dynamics of the functional CNS states of 16–18 year-old girls after exercise resulted in the identification of two main reaction types: 1) reduction of the basic criteria at high initial values; 2) increase of criteria of a general functional state of a brain at low raw data.

10

Orexin A and its role in the regulation of the hypothalamo-pituitary axes in the rat

86%

Martynska L., Polkowska J., Wolinska-Witort E., Chmielowska M., Wasilewska-Dziubinska E., Bik W., Baranowska B.

Reproductive Biology

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2006

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tom 06

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nr Suppl.2

11

Adenylate cyclase - properties, activity and methodology

86%

Jaworska-Adamu J., Cybulska R.

Folia Morphologica

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1991

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tom 50

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nr 1-2

12

Immunofluorescence of bulk isolated rat astrocytes with anti-GFAP and anti-GS sera

86%

Weinrauder H., Albrecht J.

Bulletin of the Polish Academy of Sciences. Biological Sciences

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1989

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tom 37

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nr 10-12

13

Recent findings on brain opioid-opioid pathways

86%

Kim E.-M.

Acta Neurobiologiae Experimentalis

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2005

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tom 65

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nr 5

14

Influence of selected dietary components on the functioning of the human nervous system

86%

Wendolowicz A., Stefanska E., Ostrowska L.

Roczniki Państwowego Zakładu Higieny

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2018

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tom 69

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nr 1

The diet is directly connected not only with the physical status but also with the functioning of the brain and the mental status. The potentially beneficial nutrients with a protective effect on the nervous system function include amino acids (tryptophan, phenylalanine, tyrosine, taurine), glucose and vitamins C, E, D and beta-carotene, B group vitamins (vitamin B12, vitamin B6, vitamin B4, vitamin B1) and minerals (selenium, zinc, magnesium, sodium, iron, copper, manganese, iodine). The presence of antioxidants in the diet protects against oxidative damage to nervous system cells. Biochemical data indicate that polyunsaturated fatty acids such as arachidonic acid (AA), docosahexaenoic acid (DHA), eicosapentaenoic acid (EPA) and gamma-linolenic acid (GLA) as structural components of the nervous system play a key role in its function. The nutrition of the entire body also influences the production of neurotransmitters in the brain. A diet without an appropriate supply of protein, mineral nutrients or vitamins may result in a failure to form appropriately balanced numbers of neurotransmitters, which, as a result, may lead to neurotransmission dysfunction. This is the reason why proper nutrition is based on vegetables, fruits, whole-grain cereal products supplemented with products providing full-value protein (dairy products, fish, lean meat) and high-quality fat products (vegetable oils, fish fats).

15

Gut microbiota in neurological disorders

86%

Grochawska M., Laskus T., Radkowski M.

Archivum Immunologiae et Therapiae Experimentalis

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2019

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tom 67

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nr 6

16

Glucagon-like peptides in the CNS of man: localization and possible functional importance

86%

Bernstein H. G.

Folia Histochemica et Cytobiologica

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1984

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tom 22

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nr 3-4

17

Investigation of the 22q11.2 candidate region in patients with midline facial defects with hypertelorism

86%

Simioni M., Lopes F. E., Paiva V. T., Lopes-Cendes I., Gil-da-Silva-Lopes V. L.

Journal of Applied Genetics

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2010

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tom 51

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nr 2

219-221

Midline facial defects with hypertelorism (MFDH) are mainly characterized by ocular hypertelorism and bifid nose. They are often associated with structural and functional anomalies of the central nervous system similar to those found in 22q11.2 deletion syndromes. In addition, there are some isolated reports of MFDH and 22q11.2 deletion. These findings suggest that MFDH may be part of the spectrum of 22q11.2 deletion syndromes. To test this hypothesis, 10 individuals with MFDH were analyzed by fluorescent in situ hybridization (FISH), but no 22q11.2 deletion was detected. In view of this result, the TBX1 gene located within the 22q11.2 candidate region was screened. A new sequence variant (1132GA) was identified in one patient. This variant was not found in 110 control individuals genotyped. Considering the rarity of this condition and results of this study, the involvement of the 22q11.2 chromosomal region in the pathogenesis of MFDH could not be excluded.

18

Cystatin C and neuropeptid Y levels in brain tissues after experimental subarachnoid hemorrhage

86%

Acikgoz S., Can M., Guven B., Edebali N., Barut F., Buyukuysal C., Tekin I. O., Acikgoz B.

Acta Biochimica Polonica

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2014

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tom 61

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nr 4

19

Epigenetic mechanisms of gene expression regulation in neurological diseases

86%

Gos M., Rzonca S., Szopa D., Abramowicz A., Bal J.

Acta Neurobiologiae Experimentalis

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2013

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tom 73

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nr 1

Neurological diseases, including intellectual disability (ID), can be caused by disturbances in epigenetic regulation of specific genes that encode proteins necessary for appropriate central nervous system functioning. The “epigenetically caused” diseases can be due to the imprinting defects formed during germinal cells development or gained throughout life as a somatic changes. They can also result from abnormal functioning of transcriptional machinery caused by mutations in genes coding for specific proteins. Two most classical examples of disease caused by imprinting defect in germinal cells are Prader-Willi and Angelman syndromes, both characterized by ID and developmental delay. Both these diseases are caused by altered epigenetic regulation of genes localized on chromosome 15 (region q11–q13) that can be due to chromosome deletion or uniparental disomy. The other neurological disease that is related to abnormal epigenetic regulation is Fragile X syndrome characterized by ID and specific behavior. Almost all disease cases are due to the expansion of CGG repeat (>200) in the 5’UTR of FMR1 gene that leads to promoter methylation and lack of FMRP protein that is indispensable for neuron development and signaling. The example of neurological “epigenetic diseases” caused by altered transcriptional regulation is Rett syndrome caused by the mutation presence in MECP2 gene or its variant – Rett-like syndrome caused by the mutation in CDKL5 gene. Both these diseases are characterized by ID and childhood epilepsy. Herein, we present our experience from the research and diagnosis of above mentioned disorders in the context of neurological pathways altered by improper epigenetic regulation.

20

Morphological and molecular organization of the brain postsynaptic densities

86%

Rysak M., Konecki J., Helewski K.

Folia Morphologica

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1988

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tom 47

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nr 1-4

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