Wyniki wyszukiwania

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Seeing and doing: sensorimotor simulation and neuroaesthetics

100%

Haggard P.

Acta Neurobiologiae Experimentalis

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2005

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tom 65

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nr 5

2

Anatomic heterogeneity of the rat amygdaloid complex

86%

Pitkanen A., Jolkkonen E., Kemppainen S.

Folia Morphologica

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2000

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tom 59

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nr 1

The amygdala is a nuclear complex composed of 13 nuclei and cortical areas and their subdivisions. Tract-tracing studies performed over the past 20 years demonstrate that each nucleus is uniquely connected with other brain areas. Consistent with anatomic heterogeneity, the functions of the amygdala vary from attention to memory to formation of emotional responses to sensory stimuli. Here, we briefly review the principles of amygdaloid neuronal wiring that underlie the computations necessary to perform such complex behavioural functions.

3

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Different receptor subtypes are involved in the serotonin-induced modulation of epileptiform activity in rat frontal cortex in vitro

86%

Bobula B., Zahorodna A., Bijak M.

Journal of Physiology and Pharmacology

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2001

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tom 52

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nr 2

4

Musicians as a model for early and going neuroplasticity

72%

Jancke L.

Acta Neurobiologiae Experimentalis

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2005

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tom 65

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nr 5

5

Carboxyl terminal fragment of beta-amyloid precursor protein: electrophysiological effects and cellular toxicity

72%

Fraser S. P., Bryan A. A., Diss J. K. J., Kim J. H., Kim S. H., Suh Y. H., Djamgoz M. B. A.

Cellular and Molecular Biology Letters

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1997

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tom 02

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nr 2

Alzheimer's disease (AD) is characterized by deposition of β-amyloid (Aβ) in areas of the brain. Aβ is a metabolic fragment of the β-amyloid precursor protein (βAPP). Genetic evidence has linked βAPP to AD, and there is increasing evidence that fragments from βAPP are neurotoxic. Aβ, the main research focus, has been shown to induce depolarizing ion channel activity. Involvement of other cleaved products from βAPP are less clear. We have investigated the 105 amino acid C-terminal peptide (CT105) (containing the full sequence Aβ), an alternative fragment linked with cellular toxicity. CT105 induced non-selective ionic currents in Xenopus oocytes (a model cell used in cell signalling studies) and was toxic to oocytes and mammalian cortical neurones. These results suggest possible involvement of CT105 in inducing the neural toxicity characteristic of AD.

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Fluoro-Jade and TUNEL staining as useful tools to identify ischemic brain damage following moderate extradural compression of sensorimotor cortex

72%

Kundrotiene J., Wagner A., Liljequist S.

Acta Neurobiologiae Experimentalis

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2004

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tom 64

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nr 2

7

Orexin a integration of feeding, wakefulness and activity: implications for energy balance

58%

Kotz C. M.

Acta Neurobiologiae Experimentalis

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2005

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tom 65

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nr 5

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Dynein c1h1, dynactin and syntaphilin expression in brain areas related to neurodegenerative diseases following exposure to rotenone

58%

Chaves R. S., Melo T. Q., D'Unhao A. M., Farizatto K. L. G., Ferrari M. F. R.

Acta Neurobiologiae Experimentalis

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2013

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tom 73

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nr 4

Neurodegeneration is often accompanied by protein inclusions which may interfere with cell physiology. On the other hand, alteration in intracellular trafficking may precede impairment of neurotransmission and therefore trigger cell death. In view of this, it is hypothesized that changes in mitochondrial traffic may occur before neurodegeneration triggered by rotenone exposure and could favor this process. The effects of low concentrations of rotenone on the expression of dynein clhl, dynactin and syntaphilin, which are proteins related to mitochondria transport and anchoring, were evaluated in cell cultures of substantia nigra, locus coeruleus and hippocampus as well as in these same brain areas in Lewis aged rats. The results indicate that low concentrations of rotenone decrease dynein clhl protein levels in cell cultures and brain areas of aged rats. Dynactin is decreased after exposure to 0.1 and 0.3 nM of rotenone, and increased after exposure to 0.5 nM of rotenone in cell cultures. Aged rats present increased dynactin expression. Syntaphilin expression decreased in vitro and increased in vivo after rotenone exposure. These findings suggest that changes in protein expression related to mitochondrial retrograde transport and anchoring occur before neurodegeneration induced by rotenone exposure, which may be a primary factor to trigger neurodegenerative mechanisms.

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Neuropeptide Y [NPY] and its mRNA in discrete brain areas after subchronic administration of neuroleptics

58%

Herman Z. S.

Acta Neurobiologiae Experimentalis

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1996

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tom 56

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nr 1

Ograniczanie wyników

Seeing and doing: sensorimotor simulation and neuroaesthetics

Anatomic heterogeneity of the rat amygdaloid complex

Different receptor subtypes are involved in the serotonin-induced modulation of epileptiform activity in rat frontal cortex in vitro

Musicians as a model for early and going neuroplasticity

Carboxyl terminal fragment of beta-amyloid precursor protein: electrophysiological effects and cellular toxicity

Fluoro-Jade and TUNEL staining as useful tools to identify ischemic brain damage following moderate extradural compression of sensorimotor cortex

Orexin a integration of feeding, wakefulness and activity: implications for energy balance

Dynein c1h1, dynactin and syntaphilin expression in brain areas related to neurodegenerative diseases following exposure to rotenone

Neuropeptide Y [NPY] and its mRNA in discrete brain areas after subchronic administration of neuroleptics