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  1. 13 Acta Neurobiologiae Experimentalis

  2. 10 Journal of Physiology and Pharmacology. Supplement

  3. 8 Acta Parasitologica

  4. 8 Folia Histochemica et Cytobiologica

  5. 8 Wiadomości Parazytologiczne. Suplement

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  1. 56 Walski M.

  2. 14 Frontczak-Baniewicz M.

  3. 11 Grytner-Ziecina B.

  4. 9 Dabrowska J.

  5. 8 Chomicz L.

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  1. 1 2013

  2. 1 2011

  3. 3 2009

  4. 1 2008

  5. 5 2007

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1

Fine structure of the surfactant system in amphibian lungs

100%
Walski M.
Folia Histochemica et Cytobiologica
|
1984
|
tom 22
|
nr 2
2

The ultrastructure of the "air-blood barrier" in birds

100%
Walski M.
Folia Histochemica et Cytobiologica
|
1984
|
tom 22
|
nr 2
3

Morphological differentiation of tegument and the visualization of carbohydrate groups on the surface of Diorchis elisae

63%
Walski M., Hassa Z.
Folia Histochemica et Cytobiologica
|
1984
|
tom 22
|
nr 2
4
Content available remote

Ultrastructural features of the neurovascular unit in Alzheimer's neurodegeneration

63%
Frontczak-Baniewicz M., Walski M.
Journal of Physiology and Pharmacology. Supplement
|
2006
|
tom 57
|
nr 4
91-96
Recent studies suggest that capillaries, neurons, and astrocytes form a functional unit that serves to maintain cerebral homeostasis. Physiological interactions between all these components of the neurovascular unit control cerebral microcirculation, while abnormal regulatory mechanisms lead to cerebral dysfunction and disease states, such as Alzheimer’s disease (AD). Using electron microscopy, we studied a fragment of the frontotemporal cortex obtained intraoperatively from a patient with established AD. The objective of our study was to assess the ultrastructure of the components of the neurovascular unit. Such ultrastructural studies allow analyzing the structural process of new blood vessels formation and also the appearance of neurons and astrocytes contributing to the neurovascular unit. We suggest that dysfunction of particular components of the neurovascular unit underlies AD and ultimately leads to neurodegeneration.
5
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Content available

Interakcja Pneumocystis spp. z komorkami nablonka pecherzykowego pluc w badaniach ultrastrukturalnych

63%
Walski M., Golab E.
Wiadomości Parazytologiczne. Suplement
|
2007
|
tom 53
59
6
Artykuł dostępny w postaci pełnego tekstu - kliknij by otworzyć plik
Content available

Pluca jako siedlisko pasozytow. Badania ultrastrukturalne bariery oddechowej pluc

63%
Walski M., Grytner-Ziecina B.
Wiadomości Parazytologiczne. Suplement
|
2007
|
tom 53
60
7

Mozgowe naczynia kapilarne. Ultrastrukturalne wykladniki procesow angiogenezy

63%
Frontczak-Baniewicz M., Walski M.
Postępy Biologii Komórki
|
2004
|
tom 31
|
nr 3
563-573
8

A model of intraalveolar conversion of lamellar structures to tubular myelin in the rat lung

63%
Groniowski J., Walski M.
Folia Histochemica et Cytobiologica
|
1984
|
tom 22
|
nr 2
9
Content available remote

Effects of 3-aminobenzamide on ultrastructure of hippocampal CA1 layer after global ischemia in gerbils

63%
Strosznajder R. P., Walski M.
Journal of Physiology and Pharmacology. Supplement
|
2004
|
tom 55
|
nr 3
127-133
Poly(ADP-ribose) polymerase (PARP EC 2.4.2.30) is a key enzyme in the DNA repair machinery, but its excessive stimulation during reperfusion after ischemia could play a critical role in cell death. Our previous study indicated that the PARP inhibitor 3-aminobenzamide (3-AB) significantly protected neuronal cells against death after a short ischemic insult. In this study we investigated the effect of 3-AB on the ischemia-evoked alterations in intracellular organelles. Gerbils were submitted to 3 min of transient forebrain ischemia followed by reinstitution of recirculation for 1-7 days. Electron microscopy showed only the signs of necrotic cell death after ischemia-reperfusion. The examination of specimens revealed a pronounced protective effect of 3-AB on the swelling of astrocytes and neurons 1 day after the ischemic insult. 3-AB also decreased the swelling of pericytes, but it had no effect on the accumulation of osmiophilic inclusions and fibril formation in astrocytes. 3-AB decreased the ischemia-induced swelling of mitochondria. The protective effects of 3-AB on cellular ultrastructure were also observed 7 days after reperfusion. These findings indicate that the inhibition of PARP may have a protective effect on cell swelling and on the state of intracellular organelles after a short-term ischemic episode.
10
Content available remote

Glial scar instability after brain injury

63%
Frontczak-Baniewicz M., Walski M.
Journal of Physiology and Pharmacology. Supplement
|
2006
|
tom 57
|
nr 4
97-102
Glial scar is formed following surgical damage to the cerebral cortex. In the present study we examined the ultrastructural status of the cerebral cortex 14 to 180 days following surgical damage to cerebral parenchyma. The results showed a contribution of astrocytes, but also mesodermal cells, to the process of scar formation. Furthermore, our study showed that the process initiated by trauma did not terminate with the formation of a glial scar. Late phases of repair following tissue damage were associated with lytic processes and a disassembly of the cerebral parenchyma. These findings indicate a changing and unstable nature of the glial scar and its components.
11

NADPH-diaphorase in the cat carotid body

63%
Walski M., Pokorski M.
Acta Neurobiologiae Experimentalis
|
2000
|
tom 60
|
nr 1
12

New vessel formation after surgical brain injury in the rat's cerebral cortex. I. Formation of the blood vessels proximally to the surgical injury

63%
Frontczak-Baniewicz M., Walski M.
Acta Neurobiologiae Experimentalis
|
2003
|
tom 63
|
nr 2
13

New vessel formation after surgical brain injury in the rat's cerebral cortex. II. Formation of the blood vessels distal to the surgical injury

63%
Walski M., Frontczak-Baniewicz M.
Acta Neurobiologiae Experimentalis
|
2003
|
tom 63
|
nr 2
14
Artykuł dostępny w postaci pełnego tekstu - kliknij by otworzyć plik
Content available

Ultrastructure of oncospheral envelopes at the final stage of infective - egg formation in some species of Cyclophyllidea

63%
Chomicz L., Walski M.
Wiadomości Parazytologiczne
|
1998
|
tom 44
|
nr 3
15

Hypoxia-induced subcellular migration of calcium in the carotid body: an ultrastructural assessment by ion-capture cytochemistry

63%
Pokorski M., Walski M.
Cellular and Molecular Biology Letters
|
2002
|
tom 07
|
nr Suppl.
16

Iatrogenic Chlamydia infection-associated damage in the basilar arterial wall of the rat

63%
Chrapusta S. J., Walski M.
Folia Histochemica et Cytobiologica
|
2002
|
tom 40
|
nr 4
17

Myelin glycoproteins as a target of Ph in the rodent model of prolonged exposure

51%
Dabrowska-Bouta B., Walski M., Rafalowska U.
Acta Neurobiologiae Experimentalis
|
2006
|
tom 66
|
nr 4
18

Influence of 0.02 M EDTA and 3 M KCl on surface of Hymenolepis diminuta and composition of isolated proteins

51%
Machnicka B., Krawczuk S., Walski M.
Folia Histochemica et Cytobiologica
|
1986
|
tom 24
|
nr 1
19

An attempt to exhibit the point of absorption and the passage for transporting proteine substances in tapeworms

51%
Walski M., Hassa Z., Cielecka D.
Folia Histochemica et Cytobiologica
|
1986
|
tom 24
|
nr 4
20
Content available remote

The aging corotid body

51%
Pokorski M., Walski M., Dymecka A., Marczak M.
Journal of Physiology and Pharmacology. Supplement
|
2004
|
tom 55
|
nr 3
107-113
The respiratory system is subject to the aging process, which could limit its responsiveness to hyperventilatory stimuli. Attenuation of the ventilatory response to hypoxia in old age is, as yet, an unresolved issue. Such attenuation may be germane for the pathogenesis of respiratory disorders developing more often in elderly subjects. The aim of this study was to determine the potential adverse effects of age on the morphology and function of carotid bodies. Morphology was studied at the level of electron microscopy on carotid bodies dissected from adult young (3 months) and old (>2 years) rats and function by comparing the hypoxic ventilatory response in populations of young (mean age 24 years) and old (mean age 71 years) female subjects. The human protocol consisted of a progressive hypoxia test, based on a rebreathing technique in a closed system. The hypoxic ventilatory response was evaluated from the slopes of minute ventilation on arterial oxygen saturation. The results of the morphological study showed degenerative changes developing with age in the ultrastructure of carotid bodies. On the other side, respiratory responses to hypoxia in old women were well preserved and were no less than those in young women. Therefore, a discrepancy appeared between the morphological and functional aspects. These findings suggest development of compensatory mechanisms in brain respiratory areas which maintain primary defensive reflexes, such as the hyperventilation of hypoxia.
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