Zakład Żywienia Klinicznego, Instytut Żywności i Żywienia w Warszawie, Warszawa
Bibliografia
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17. Illingworth D. R., Corbin D. K., Kemp E. D., et al.: Hormone changes during the menstmal cycle in abetalipoproteinemia. Reduced luteal phase progeserone in a patient with homozygous hypobetalipoproteinemia. Proc. Natl. Acad. Sci. USA 1982, 79, 6685.
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20. Innerarity T. L., Weisgraber U. H., Arnold K. S., et al.: Familial defective apolipoprotein B-100: low density lipoprotein with abnormal receptor binding. Proc. Natl. Acad. Sci. USA 1987, 84, 6919.
21. Kane J. D., Hardman D. A., Paulus H. E.: Heterogenity of apolipoprotein B. Isolation of a new species from human chylomicron. Proc. Natl. Acad. Sci. USA 1980, 77, 2465.
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26. Krempler F., Kostner G. M., Roscher A., et al.: Studies on the role of specific cell surface receptors in the removal of lipoprotein(a) in man. J. Clin. Invest., 1983, 72, 1431.
27. Kwiterovich P.O., White S., Forte T., et al.: Hyperapobetalipoproteinemia in a kindered with familial combined hyperlipidemia and familial hypercholesterolemia. Arteriosclerosis 1987, 7, 211.
28. Law S. W., Lachner R.J., Hospattanker A.V., et al.: Human apolipoprotein B-100: cloning, analysis of liver mRNA, and assigument of the gene to chromosome 2. Proc. Natl. Acad. Sci. USA 1985, 82, 8340.
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30. Malloy M. J., Kane J. P., Hardman D. A., et al.: Normotriglyceridemic abetalipoproteinemia. Absence of the B-100 apolipoprotein. J. Clin. Invest., 1981, 67, 1441.
31. Nowicka G.: Lp(a) jako czynnik sprzyjający rozwojowi choroby wieńcowej serca. Żyw. Człow. Metab., 1990, 17, 70.
32. Rose H. G., Krauz P., Weinstock M., et al.: Inheritance of combined hyperlipoproteinemia. Evidence for a new lipoprotein phenotype. Amer. J. Med., 1973, 54, 148.
33. Roy C. C„ Levy E., Green P. H. R., et al.: Malabsorption, hypocholesterolemia, fat-filled enterocytes with increased intenstinal apoprotein B: Chylomicron retention disease. Gastroenterology 1987, 92, 390.
34. Scanu A. M.: Lipoprotein(a): genetically determined lipoprotein containing a glycoprotein of the plasminogen family. Seminars in Thrombosis and Hemostasis 1988, 14, 266.
35. Scanu A. M., Aggebeck L. P., Kruski A. W., et al.: A study of the abnormal lipoproteins in abetalipoproteinemia. J. Clin. Invest., 1974, 53, 440.
36. Soria L. F., Ludwig E. H., Clarke H. R. G., et al.: Association between a specific apolipoprotein B mutation and familial defective apolipoprotein B-100. Proc. Natl. Acad. Sci. USA 1989, 86, 587.
37. Talmud P. J., Lloyd J.U., Muller D. P. R., et al.: Genetic evidence from two families that the apolipoprotein B gene is not involved in abetalipoproteinemia. J. Clin. Invest., 1988, 82, 1803.
38. Teng B., Thompson G. R., Sniderman A. D., et al.: Composition and distritution of low density lipoprotein fractions in hyperapobetalipoproteinemia, normolipidemia and hypercholesterolemia. Proc. Natl. Acad. Sci. USA 1983, 80, 6662.
39. Uterman G., Menzel H.J., Kraft H.G., et al.: Lp(a) glycoprotein phenotypes. J. Clin. Invest., 1987, 80, 458.
40. Vessby G., Kostner G. H., Idhell H., et al.: Diverging effects of cholestyramine an apolipoprotein B and lipoprotein Lp(a). Atherosclerosis 1982, 44, 61.
41. Young S. G., Bertics S. J., Curtiss L. K., et al.: Characterization of an abnormal species of apolipoprotein B, apolipoprotein B-37, associated with familial hypobetalipoproteinemia. J. Clin. Invest. 1987, 79, 1831.
42. Young S.G., Bertics S. J., Curtiss L. K., et al.: Genetic analysis of a kindred with familial hypobetalipoproteinemia. Evidence for two separate gene defects: one associated with an abnormal apolipoprotein B species, apolipoprotein B-37, and a second associated with low plasma concentrations of apolipoprotein B-100. J. Clin. Invest., 1987, 79, 1842.
43. Zenker G., Rocltringer P., Bone G.: Lipoprotein Lp(a) as a strong indicator for cerebrovascular disease. Stroke 1986, 17, 942.